A sequential treatment regimen with melatonin and all-trans retinoic acid induces apoptosis in MCF-7 tumour cells
about
Rotating night shifts and risk of breast cancer in women participating in the nurses' health studyConnective tissue growth factor induces apoptosis in human breast cancer cell line MCF-7.Ectopic expression of cyclin D1 amplifies a retinoic acid-induced mitochondrial death pathway in breast cancer cells.Evidence for a biphasic apoptotic pathway induced by melatonin in MCF-7 breast cancer cells.Growth arrest and cell death in the breast tumor cell in response to ionizing radiation and chemotherapeutic agents which induce DNA damage.Melatonin in the treatment of cancer: a systematic review of randomized controlled trials and meta-analysis.TRAIL/Apo-2L: mechanisms and clinical applications in cancer.Melatonin: an inhibitor of breast cancer.Melatonin decreases breast cancer metastasis by modulating Rho-associated kinase protein-1 expression.Principles of psychoneuroendocrinoimmunotherapy of cancer.Molecular mechanisms of melatonin's inhibitory actions on breast cancers.Molecular mechanisms of the pro-apoptotic actions of melatonin in cancer: a review.Differential regulation of estrogen receptor alpha, glucocorticoid receptor and retinoic acid receptor alpha transcriptional activity by melatonin is mediated via different G proteins.The isomerase PIN1 controls numerous cancer-driving pathways and is a unique drug target.In vitro and in vivo antitumor activity of melatonin receptor agonists.Melatonin induces apoptosis in human neuroblastoma cancer cells.Conversion of Fas-resistant to Fas-sensitive MCF-7 breast cancer cells by the synergistic interaction of interferon-gamma and all-trans retinoic acid.Prooxidant activity of melatonin promotes fas-induced cell death in human leukemic Jurkat cells.Pathways through which a regimen of melatonin and retinoic acid induces apoptosis in MCF-7 human breast cancer cells.Effect of Melatonin on the Expression of Apoptotic Genes in Vitrified-thawed Spermatogonia Stem Cells Type A of 6-Day-Old MiceModulation of intracellular calcium and calmodulin by melatonin in MCF-7 human breast cancer cells.Tumor prevention by 9-cis-retinoic acid in the N-nitroso-N-methylurea model of mammary carcinogenesis is potentiated by the pineal hormone melatonin.Melatonin provokes cell death in human B-lymphoma cells by mitochondrial-dependent apoptotic pathway activation.Inhibitory effects of melatonin on the growth of pituitary prolactin-secreting tumor in rats.A novel L-fucose-binding lectin from Fenneropenaeus indicus induced cytotoxicity in breast cancer cells.all-trans-retinoic acid improves immunocompetence in a murine model of lipopolysaccharide-induced immunosuppression.Melatonin: An Anti-Tumor Agent in Hormone-Dependent Cancers
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P2860
A sequential treatment regimen with melatonin and all-trans retinoic acid induces apoptosis in MCF-7 tumour cells
description
1998 nî lūn-bûn
@nan
1998 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
A sequential treatment regimen ...... poptosis in MCF-7 tumour cells
@ast
A sequential treatment regimen ...... poptosis in MCF-7 tumour cells
@en
A sequential treatment regimen ...... poptosis in MCF-7 tumour cells
@nl
type
label
A sequential treatment regimen ...... poptosis in MCF-7 tumour cells
@ast
A sequential treatment regimen ...... poptosis in MCF-7 tumour cells
@en
A sequential treatment regimen ...... poptosis in MCF-7 tumour cells
@nl
prefLabel
A sequential treatment regimen ...... poptosis in MCF-7 tumour cells
@ast
A sequential treatment regimen ...... poptosis in MCF-7 tumour cells
@en
A sequential treatment regimen ...... poptosis in MCF-7 tumour cells
@nl
P2093
P2860
P356
P1476
A sequential treatment regimen ...... poptosis in MCF-7 tumour cells
@en
P2093
P2860
P2888
P304
P356
10.1038/BJC.1998.357
P407
P577
1998-06-01T00:00:00Z
P5875
P6179
1013312603