Particle-induced pulmonary acute phase response correlates with neutrophil influx linking inhaled particles and cardiovascular risk
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Pulmonary effects of carbon nanomaterialsTime-dependent subcellular distribution and effects of carbon nanotubes in lungs of miceInflammation and Vascular Effects after Repeated Intratracheal Instillations of Carbon Black and LipopolysaccharideNanomaterials vs Ambient Ultrafine Particles: an Opportunity to Exchange Toxicology KnowledgeEpoxy composite dusts with and without carbon nanotubes cause similar pulmonary responses, but differences in liver histology in mice following pulmonary depositionOccupational exposure levels of bioaerosol components are associated with serum levels of the acute phase protein Serum Amyloid A in greenhouse workersCarbon black and titanium dioxide nanoparticles induce distinct molecular mechanisms of toxicityParticle-induced pulmonary acute phase response may be the causal link between particle inhalation and cardiovascular diseaseSingle-walled carbon nanotubes increase pandemic influenza A H1N1 virus infectivity of lung epithelial cellsMulti-walled carbon nanotube physicochemical properties predict pulmonary inflammation and genotoxicityTranscriptomic analysis reveals novel mechanistic insight into murine biological responses to multi-walled carbon nanotubes in lungs and cultured lung epithelial cellsUltrafine carbon particle mediated cardiovascular impairment of aged spontaneously hypertensive ratsComparative hazard identification by a single dose lung exposure of zinc oxide and silver nanomaterials in miceSurface modification does not influence the genotoxic and inflammatory effects of TiO2 nanoparticles after pulmonary exposure by instillation in miceAirway exposure to multi-walled carbon nanotubes disrupts the female reproductive cycle without affecting pregnancy outcomes in mice.Differences in inflammation and acute phase response but similar genotoxicity in mice following pulmonary exposure to graphene oxide and reduced graphene oxide.The Toxicological Mechanisms of Environmental Soot (Black Carbon) and Carbon Black: Focus on Oxidative Stress and Inflammatory PathwaysDNA damage following pulmonary exposure by instillation to low doses of carbon black (Printex 90) nanoparticles in mice.Multi-walled carbon nanotube-physicochemical properties predict the systemic acute phase response following pulmonary exposure in mice.Nanomaterial translocation--the biokinetics, tissue accumulation, toxicity and fate of materials in secondary organs--a review.Reproductive and developmental toxicity of carbon-based nanomaterials: A literature review.Mechanism of TH2/TH17-predominant and neutrophilic TH2/TH17-low subtypes of asthma.Hepatic hazard assessment of silver nanoparticle exposure in healthy and chronically alcohol fed mice.Characterization of genotoxic response to 15 multiwalled carbon nanotubes with variable physicochemical properties including surface functionalizations in the FE1-Muta(TM) mouse lung epithelial cell line.Worker Exposure and High Time-Resolution Analyses of Process-Related Submicrometre Particle Concentrations at Mixing Stations in Two Paint Factories.Transcriptional profiling identifies physicochemical properties of nanomaterials that are determinants of the in vivo pulmonary response.Primary genotoxicity in the liver following pulmonary exposure to carbon black nanoparticles in mice.Editorial: dose-dependent ZnO particle-induced acute phase response in humans warrants re-evaluation of occupational exposure limits for metal oxides.Modifications of carbon black nanoparticle surfaces modulate type II pneumocyte homoeostasis.Nano zerovalent iron particles induce pulmonary and cardiovascular toxicity in an in vitro human co-culture model.DNA strand breaks, acute phase response and inflammation following pulmonary exposure by instillation to the diesel exhaust particle NIST1650b in mice.Cerium dioxide nanoparticles exacerbate house dust mite induced type II airway inflammation.Maternal inhalation of carbon black nanoparticles induces neurodevelopmental changes in mouse offspring
P2860
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P2860
Particle-induced pulmonary acute phase response correlates with neutrophil influx linking inhaled particles and cardiovascular risk
description
2013 nî lūn-bûn
@nan
2013 թուականին հրատարակուած գիտական յօդուած
@hyw
2013 թվականին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Particle-induced pulmonary acu ...... ticles and cardiovascular risk
@ast
Particle-induced pulmonary acu ...... ticles and cardiovascular risk
@en
Particle-induced pulmonary acu ...... ticles and cardiovascular risk
@nl
type
label
Particle-induced pulmonary acu ...... ticles and cardiovascular risk
@ast
Particle-induced pulmonary acu ...... ticles and cardiovascular risk
@en
Particle-induced pulmonary acu ...... ticles and cardiovascular risk
@nl
prefLabel
Particle-induced pulmonary acu ...... ticles and cardiovascular risk
@ast
Particle-induced pulmonary acu ...... ticles and cardiovascular risk
@en
Particle-induced pulmonary acu ...... ticles and cardiovascular risk
@nl
P2093
P2860
P50
P3181
P1433
P1476
Particle-induced pulmonary acu ...... ticles and cardiovascular risk
@en
P2093
Allen Njimeri Nyendi
Anne Thoustrup Saber
Håkan Wallin
Jacob Stuart Lamson
Petra Jackson
Pia Wahlberg
P2860
P304
P3181
P356
10.1371/JOURNAL.PONE.0069020
P407
P577
2013-07-24T00:00:00Z