Therapeutic effect of topical administration of SN50, an inhibitor of nuclear factor-kappaB, in treatment of corneal alkali burns in mice - Wikidata - wikimedia - TriplyDB

Therapeutic effect of topical administration of SN50, an inhibitor of nuclear factor-kappaB, in treatment of corneal alkali burns in mice

Therapeutic effect of topical administration of SN50, an inhibitor of nuclear factor-kappaB, in treatment of corneal alkali burns in mice

http://www.wikidata.org/entity/Q28391341

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Involvement of NADPH oxidases in alkali burn-induced corneal injury Induction of FGF-2 synthesis by IL-1beta in aqueous humor through P13-kinase and p38 in rabbit corneal endothelium.Multi-gene targeted antiangiogenic therapies for experimental corneal neovascularization.Tetrandrine suppresses activation of human subconjunctival fibroblasts in vitro.Therapeutic potential of trichostatin A to control inflammatory and fibrogenic disorders of the ocular surface.Bovine lactoferrin promotes corneal wound healing and suppresses IL-1 expression in alkali wounded mouse cornea.Full-Thickness Thermal Injury Delays Wound Closure in a Murine Model TRPV1 involvement in inflammatory tissue fibrosis in mice SN50 enhances the effects of LY294002 on cell death induction in gastric cancer cell line SGC7901.Expression of Smad7 in mouse eyes accelerates healing of corneal tissue after exposure to alkali Loss of tumor necrosis factor alpha potentiates transforming growth factor beta-mediated pathogenic tissue response during wound healing.Therapeutic effects of topical netrin-4 in a corneal acute inflammatory model.Tim-4 Inhibits NO Generation by Murine Macrophages.Corneal antifibrotic switch identified in genetic and pharmacological deficiency of vimentin Fasudil hydrochloride, a potent ROCK inhibitor, inhibits corneal neovascularization after alkali burns in mice Inhibition of VEGF expression and corneal neovascularization by shRNA targeting HIF-1α in a mouse model of closed eye contact lens wear.Disruption of eyelid and cornea morphogenesis by epithelial β-catenin gain-of-function.Dexamethasone inhibits interleukin-1beta-induced corneal neovascularization: role of nuclear factor-kappaB-activated stromal cells in inflammatory angiogenesis.Suppression of In Vivo Neovascularization by the Loss of TRPV1 in Mouse Cornea.Loss of TRPV4 Function Suppresses Inflammatory Fibrosis Induced by Alkali-Burning Mouse Corneas Thymosin beta 4 suppression of corneal NFkappaB: a potential anti-inflammatory pathway Dual roles of NF-kappaB in cell survival and implications of NF-kappaB inhibitors in neuroprotective therapy.Lornoxicam suppresses recurrent herpetic stromal keratitis through down-regulation of nuclear factor-kappaB: an experimental study in mice.The molecular mechanisms of action of PPAR-γ agonists in the treatment of corneal alkali burns (Review)Therapeutic peptides for cancer therapy. Part I - peptide inhibitors of signal transduction cascades.Mucosal immune tolerance at the ocular surface in health and disease.Inhibitory effects of polysaccharide extract from Spirulina platensis on corneal neovascularization.LIM Homeobox Domain 2 Is Required for Corneal Epithelial Homeostasis.Impaired Healing of a Cutaneous Wound in an Inducible Nitric Oxide Synthase-Knockout Mouse.Inhibitory effect of blocking TGF-beta/Smad signal on injury-induced fibrosis of corneal endothelium.Hydrocortisone suppression of the expression of VEGF may relate to toll-like receptor (TLR) 2 and 4.Ocular surface alkali injury damages meibomian glands in mice.Effect of different biomedical membranes on alkali-burned cornea.Peroxisome proliferator-activated receptor alpha agonist suppresses neovascularization by reducing both vascular endothelial growth factor and angiopoietin-2 in corneal alkali burn.Antiangiogenic effects of catalpol on rat corneal neovascularization.Loss of tenascin X gene function impairs injury-induced stromal angiogenesis in mouse corneas.Comprehensive Modeling of Corneal Alkali Injury in the Rat Eye.Inhibition of HUVEC tube formation via suppression of NFκB suggests an anti-angiogenic role for SLURP1 in the transparent cornea.Therapeutic effect of SN50, an inhibitor of nuclear factor-κB, in treatment of TBI in mice.TRPA1 is required for TGF-β signaling and its loss blocks inflammatory fibrosis in mouse corneal stroma.

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Therapeutic effect of topical administration of SN50, an inhibitor of nuclear factor-kappaB, in treatment of corneal alkali burns in mice

http://www.wikidata.org/entity/Q28391341

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2005 nî lūn-bûn

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The American Journal of Pathology

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Kathleen C Flanders

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Kazuo Ikeda

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Osamu Yamanaka

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Yasuteru Muragaki

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Opposite effect of NF-kB and c-Jun N-terminal kinase on p53-independent GADD45 induction by arsenite

Therapeutic potential of inhibition of the NF-kappaB pathway in the treatment of inflammation and cancer

New insights into the role of nuclear factor-kB in cell growth regulation

Myofibroblasts and mechano-regulation of connective tissue remodelling

TGFbeta2 in corneal morphogenesis during mouse embryonic development

Sonic hedgehog expression and role in healing corneal epithelium

Costimulation of fibroblast collagen and transforming growth factor beta1 gene expression by monocyte chemoattractant protein-1 via specific receptors

Immune and inflammatory responses in TNF alpha-deficient mice: a critical requirement for TNF alpha in the formation of primary B cell follicles, follicular dendritic cell networks and germinal centers, and in the maturation of the humoral immune re

Shaping the nuclear action of NF-kappaB

NF-kappaB: a key role in inflammatory diseases

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