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Arginine deprivation and immune suppression in a mouse model of Alzheimer's disease

Arginine deprivation and immune suppression in a mouse model of Alzheimer's disease

http://www.wikidata.org/entity/Q28391826

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Targeting innate immunity for neurodegenerative disorders of the central nervous system Getting to NO Alzheimer's Disease: Neuroprotection versus Neurotoxicity Mediated by Nitric Oxide Accumulation of ubiquitin and sequestosome-1 implicate protein damage in diacetyl-induced cytotoxicity Getting into the brain: liposome-based strategies for effective drug delivery across the blood-brain barrier Eliminating microglia in Alzheimer's mice prevents neuronal loss without modulating amyloid-β pathology.Expanding the phenotype in argininosuccinic aciduria: need for new therapies.Old Maids: Aging and Its Impact on Microglia Function.Bioinformatics methods in drug repurposing for Alzheimer's disease.TLR4-activated microglia require IFN-γ to induce severe neuronal dysfunction and death in situ.Gene expression profiling in stroke: relevance of blood-brain interaction.Fate mapping reveals that microglia and recruited monocyte-derived macrophages are definitively distinguishable by phenotype in the retina.Microglial immunophenotype in dementia with Alzheimer's pathology.The impact of glutamine supplementation on the symptoms of ataxia-telangiectasia: a preclinical assessment.Arginase 2 promotes neurovascular degeneration during ischemia/reperfusion injury.Association of Serum Manganese Levels with Alzheimer's Disease and Mild Cognitive Impairment: A Systematic Review and Meta-Analysis Dissecting the Contribution of Vascular Alterations and Aging to Alzheimer's Disease.Multifaceted roles of neuroinflammation: the need to consider both sides of the coin.Difluoromethylornithine in cancer: new advances.New Insights into Epigenetic and Pharmacological Regulation of Amyloid-Degrading Enzymes.Inflammation in Alzheimer's disease: Lessons learned from microglia-depletion models.A polarizing question: do M1 and M2 microglia exist?Modulation of neuroinflammation and pathology in the 5XFAD mouse model of Alzheimer's disease using a biased and selective beta-1 adrenergic receptor partial agonist.Amino Acid Catabolism in Alzheimer's Disease Brain: Friend or Foe?Effect of Neuroinflammation on Synaptic Organization and Function in the Developing Brain: Implications for Neurodevelopmental and Neurodegenerative Disorders.The human-specific CASP4 gene product contributes to Alzheimer-related synaptic and behavioural deficits.The roles of inflammation and immune mechanisms in Alzheimer's disease.The fornix provides multiple biomarkers to characterize circuit disruption in a mouse model of Alzheimer's disease.Application of Metabolomics in Alzheimer's Disease.Neuroinflammation: Microglia and T Cells Get Ready to Tango.Synthesis, Molecular Modelling and Biological Evaluation of Novel Heterodimeric, Multiple Ligands Targeting Cholinesterases and Amyloid Beta.Use of Eflornithine (DFMO) in the Treatment of Early Alzheimer's Disease: A Compassionate Use, Single-Case Study.Identification of seven novel loci associated with amino acid levels using single-variant and gene-based tests in 8545 Finnish men from the METSIM study.Arginase-1 expression in myeloid cells regulates S. aureus planktonic but not biofilm infection.Intracerebroventricular Administration of L-arginine Improves Spatial Memory Acquisition in Triple Transgenic Mice Via Reduction of Oxidative Stress and Apoptosis.Altered plasma arginine metabolome precedes behavioural and brain arginine metabolomic profile changes in the APPswe/PS1ΔE9 mouse model of Alzheimer's disease.Systemic infection modifies the neuroinflammatory response in late stage Alzheimer's disease

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P2860

Arginine deprivation and immune suppression in a mouse model of Alzheimer's disease

http://www.wikidata.org/entity/Q28391826

description

2015 nî lūn-bûn

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2015 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2015 թվականի ապրիլին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

Arginine deprivation and immune suppression in a mouse model of Alzheimer's disease

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Arginine deprivation and immune suppression in a mouse model of Alzheimer's disease

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Arginine deprivation and immune suppression in a mouse model of Alzheimer's disease

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Arginine deprivation and immune suppression in a mouse model of Alzheimer's disease

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Arginine deprivation and immune suppression in a mouse model of Alzheimer's disease

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Arginine deprivation and immune suppression in a mouse model of Alzheimer's disease

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Arginine deprivation and immune suppression in a mouse model of Alzheimer's disease

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Arginine deprivation and immune suppression in a mouse model of Alzheimer's disease

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JNEUROSCI.4668-14.2015

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Journal of Neuroscience

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Arginine deprivation and immune suppression in a mouse model of Alzheimer's disease

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Angela L Everhart

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Joan G Wilson

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Marilyn Jansen

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P2860

Neuroinflammation – An Early Event in Both the History and Pathogenesis of Alzheimer’s Disease

Arginase activities and global arginine bioavailability in wild-type and ApoE-deficient mice: responses to high fat and high cholesterol diets

Molecular identification of a novel family of human Ig superfamily members that possess immunoreceptor tyrosine-based inhibition motifs and homology to the mouse gp49B1 inhibitory receptor

A scan of chromosome 10 identifies a novel locus showing strong association with late-onset Alzheimer disease.

miRNA-939 regulates human inducible nitric oxide synthase posttranscriptional gene expression in human hepatocytes

CNS immune privilege: hiding in plain sight.

Quantitative PCR for detection of the OT-1 transgene.

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Myeloid-derived suppressor cells as regulators of the immune system

Dispensable and indispensable amino acids for humans

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5969-5982

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1096097

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10.1523/JNEUROSCI.4668-14.2015

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Michael Dee Gunn

Candice M Brown

Michael P Vitek

Carol A. Colton

Andrew N Hoofnagle

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2015-04-01T00:00:00Z

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275052312

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Alzheimer's disease

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4397598

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