BCL-2 protects human and mouse Th17 cells from glucocorticoid-induced apoptosis - Wikidata - wikimedia - TriplyDB

BCL-2 protects human and mouse Th17 cells from glucocorticoid-induced apoptosis

BCL-2 protects human and mouse Th17 cells from glucocorticoid-induced apoptosis

http://www.wikidata.org/entity/Q28392989

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Granulocyte colony-stimulating factor blockade enables dexamethasone to inhibit lipopolysaccharide-induced murine lung neutrophils.Systems analysis uncovers inflammatory Th/Tc17-driven modules during acute GVHD in monkey and human T cells.Immunopathology alters Th17 cell glucocorticoid sensitivity.A gradient of glucocorticoid sensitivity among helper T cell cytokines.Prednisone may induce immunologic tolerance by activating the functions of decidual immune cells in early pregnancy.Diabetes Downregulates Allergen-Induced Airway Inflammation in Mice.Loss of Perp in T Cells Promotes Resistance to Apoptosis of T Helper 17 Cells and Exacerbates the Development of Experimental Autoimmune Encephalomyelitis in Mice.Regulatory and Mechanistic Actions of Glucocorticoids on T and Inflammatory Cells.Microbiota Composition and the Integration of Exogenous and Endogenous Signals in Reactive Nasal Inflammation.1,25(OH)D and dexamethasone additively suppress synovial fibroblast activation by CCR6 T helper memory cells and enhance the effect of tumor necrosis factor alpha blockade

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BCL-2 protects human and mouse Th17 cells from glucocorticoid-induced apoptosis

http://www.wikidata.org/entity/Q28392989

description

2016 nî lūn-bûn

@nan

2016 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2016 թվականի մայիսին հրատարակված գիտական հոդված

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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name

BCL-2 protects human and mouse Th17 cells from glucocorticoid-induced apoptosis

@ast

BCL-2 protects human and mouse Th17 cells from glucocorticoid-induced apoptosis

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BCL-2 protects human and mouse Th17 cells from glucocorticoid-induced apoptosis

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type

label

BCL-2 protects human and mouse Th17 cells from glucocorticoid-induced apoptosis

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BCL-2 protects human and mouse Th17 cells from glucocorticoid-induced apoptosis

@en

BCL-2 protects human and mouse Th17 cells from glucocorticoid-induced apoptosis

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prefLabel

BCL-2 protects human and mouse Th17 cells from glucocorticoid-induced apoptosis

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BCL-2 protects human and mouse Th17 cells from glucocorticoid-induced apoptosis

@en

BCL-2 protects human and mouse Th17 cells from glucocorticoid-induced apoptosis

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BCL-2 protects human and mouse Th17 cells from glucocorticoid-induced apoptosis

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B S Bochner

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C Dong

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G R S Budinger

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J Banuelos

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J Xin

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L Morales-Nebreda

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L Zhou

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M W Lingen

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N Z Lu

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R P Schleimer

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P2860

Adjuvant effect of zymosan after pulmonary treatment in a mouse ovalbumin allergy model

TGF-beta-induced Foxp3 inhibits T(H)17 cell differentiation by antagonizing RORgammat function

miR-15 and miR-16 induce apoptosis by targeting BCL2

Endotoxin Exposure during Sensitization to Blomia tropicalis Allergens Shifts TH2 Immunity Towards a TH17-Mediated Airway Neutrophilic Inflammation: Role of TLR4 and TLR2

International Union of Pharmacology. LXV. The pharmacology and classification of the nuclear receptor superfamily: glucocorticoid, mineralocorticoid, progesterone, and androgen receptors

Negative cross-talk between RelA and the glucocorticoid receptor: a possible mechanism for the antiinflammatory action of glucocorticoids

Innate IL-17-producing cells: the sentinels of the immune system.

Widespread negative response elements mediate direct repression by agonist-liganded glucocorticoid receptor.

Glucocorticoids sensitize the innate immune system through regulation of the NLRP3 inflammasome.

The Stat3/GR interaction code: predictive value of direct/indirect DNA recruitment for transcription outcome.

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10.1111/ALL.12840

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