Lithium-mediated protection against ethanol neurotoxicity - Wikidata - wikimedia - TriplyDB

Lithium-mediated protection against ethanol neurotoxicity

Lithium-mediated protection against ethanol neurotoxicity

http://www.wikidata.org/entity/Q28397186

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Therapeutic potential of mood stabilizers lithium and valproic acid: beyond bipolar disorder Cyanidin-3-glucoside ameliorates ethanol neurotoxicity in the developing brain Fetal Alcohol Spectrum Disorder (FASD) Associated Neural Defects: Complex Mechanisms and Potential Therapeutic Targets Could selenium administration alleviate the disturbances of blood parameters caused by lithium administration in rats?Motor Function Deficits Following Chronic Prenatal Ethanol Exposure are Linked to Impairments in Insulin/IGF, Notch and Wnt Signaling in the Cerebellum.Dose-dependent protective effect of lithium chloride on retinal ganglion cells is interrelated with an upregulated intraretinal BDNF after optic nerve transection in adult rats.Cdc42-dependent activation of NADPH oxidase is involved in ethanol-induced neuronal oxidative stress.A Case of Treatment Resistant Depression and Alcohol Abuse in a Person with Mental Retardation: Response to Aripiprazole and Fluvoxamine Therapy upon Consideration of a Bipolar Diathesis after Repetitive Failure to Respond to Multiple Antidepressant Lithium prevents long-term neural and behavioral pathology induced by early alcohol exposure Autophagy and ethanol neurotoxicity Developmental neurotoxicity screening using human embryonic stem cells.Compartment-dependent mitochondrial alterations in experimental ALS, the effects of mitophagy and mitochondriogenesis Long-lasting neural circuit dysfunction following developmental ethanol exposure Lithium protects against oxidative stress-mediated cell death in α-synuclein-overexpressing in vitro and in vivo models of Parkinson's disease.Involvement of sphingolipids in ethanol neurotoxicity in the developing brain Sustained Impairments in Brain Insulin/IGF Signaling in Adolescent Rats Subjected to Binge Alcohol Exposures during Development Isoflurane Is More Deleterious to Developing Brain Than Desflurane: The Role of the Akt/GSK3β Signaling Pathway.CaMKII activation is a novel effector of alcohol's neurotoxicity in neural crest stem/progenitor cells.Minocycline protects developing brain against ethanol-induced damage.Developmental Ethanol-Induced Sleep Fragmentation, Behavioral Hyperactivity, Cognitive Impairment and Parvalbumin Cell Loss are Prevented by Lithium Co-treatment.Neurotrophins in the Brain: Interaction With Alcohol Exposure During Development.Time course of acute neuroprotective effects of lithium carbonate evaluated by brain impedanciometry in the global ischemia model.Binge Alcohol Exposure Causes Neurobehavioral Deficits and GSK3β Activation in the Hippocampus of Adolescent Rats.

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P2860

Lithium-mediated protection against ethanol neurotoxicity

http://www.wikidata.org/entity/Q28397186

description

2010 nî lūn-bûn

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2010 թուականին հրատարակուած գիտական յօդուած

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2010 թվականին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Lithium-mediated protection against ethanol neurotoxicity

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Lithium-mediated protection against ethanol neurotoxicity

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Lithium-mediated protection against ethanol neurotoxicity

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Lithium-mediated protection against ethanol neurotoxicity

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Lithium-mediated protection against ethanol neurotoxicity

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Lithium-mediated protection against ethanol neurotoxicity

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Lithium-mediated protection against ethanol neurotoxicity

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Lithium-mediated protection against ethanol neurotoxicity

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Lithium-mediated protection against ethanol neurotoxicity

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FNINS.2010.00041

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Frontiers in Neuroscience

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Lithium-mediated protection against ethanol neurotoxicity

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Jia Luo

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P2860

Cyanidin-3-glucoside reverses ethanol-induced inhibition of neurite outgrowth: role of glycogen synthase kinase 3 beta

GSK-3: tricks of the trade for a multi-tasking kinase

The role of lithium in the treatment of bipolar disorder: convergent evidence for neurotrophic effects as a unifying hypothesis

No benefit from chronic lithium dosing in a sibling-matched, gender balanced, investigator-blinded trial using a standard mouse model of familial ALS

GSK3beta in ethanol neurotoxicity

The multifaceted roles of glycogen synthase kinase 3beta in cellular signaling

Development and resolution of brain lesions caused by pyrithiamine- and dietary-induced thiamine deficiency and alcohol exposure in the alcohol-preferring rat: a longitudinal magnetic resonance imaging and spectroscopy study.

Selective small molecule inhibitors of glycogen synthase kinase-3 modulate glycogen metabolism and gene transcription.

Tau phosphorylation and cleavage in ethanol-induced neurodegeneration in the developing mouse brain.

Estimating the prevalence of fetal alcohol syndrome. A summary.

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