Rtp801, a suppressor of mTOR signaling, is an essential mediator of cigarette smoke-induced pulmonary injury and emphysema - Wikidata - wikimedia - TriplyDB

Rtp801, a suppressor of mTOR signaling, is an essential mediator of cigarette smoke-induced pulmonary injury and emphysema

Rtp801, a suppressor of mTOR signaling, is an essential mediator of cigarette smoke-induced pulmonary injury and emphysema

http://www.wikidata.org/entity/Q28397712

about

Paradoxical effects of rapamycin on experimental house dust mite-induced asthma Regulation of the mTOR complex 1 pathway by nutrients, growth factors, and stress Regulation of Rela/p65 and endothelial cell inflammation by proline-rich tyrosine kinase 2.Quantitative Lys-ϵ-Gly-Gly (diGly) proteomics coupled with inducible RNAi reveals ubiquitin-mediated proteolysis of DNA damage-inducible transcript 4 (DDIT4) by the E3 ligase HUWE1.Novosphingobium and its potential role in chronic obstructive pulmonary diseases: insights from microbiome studies.The stress-response gene redd1 regulates dorsoventral patterning by antagonizing Wnt/β-catenin activity in zebrafish Oxidative DNA damage in lung tissue from patients with COPD is clustered in functionally significant sequences.REDD1 functions at the crossroads between the therapeutic and adverse effects of topical glucocorticoids Fatty acid synthase inhibition engages a novel caspase-2 regulatory mechanism to induce ovarian cancer cell death.Rhodiola rosea extracts and salidroside decrease the growth of bladder cancer cell lines via inhibition of the mTOR pathway and induction of autophagy.Cigarette smoke induces Akt protein degradation by the ubiquitin-proteasome system Mechanisms of lung endothelial barrier disruption induced by cigarette smoke: role of oxidative stress and ceramides The role of heparanase in pulmonary cell recruitment in response to an allergic but not non-allergic stimulus.REDD1 Is Essential for Optimal T Cell Proliferation and Survival.Glucocorticoid genes and the developmental origins of asthma susceptibility and treatment response Stress responses affecting homeostasis of the alveolar capillary unit.Blocking NF-κB: an inflammatory issue.SIRT1 protects against emphysema via FOXO3-mediated reduction of premature senescence in mice.Pathogenesis of chronic obstructive pulmonary disease Perspectives on translational and therapeutic aspects of SIRT1 in inflammaging and senescence.The emerging importance of autophagy in pulmonary diseases.Differential IKK/NF-κB Activity Is Mediated by TSC2 through mTORC1 in PTEN-Null Prostate Cancer and Tuberous Sclerosis Complex Tumor Cells.Resolvin D1 Reduces Emphysema and Chronic Inflammation.RTP801 is required for ceramide-induced cell-specific death in the murine lung Mitochondrial iron chelation ameliorates cigarette smoke-induced bronchitis and emphysema in mice.Rapamycin attenuates acute lung injury induced by LPS through inhibition of Th17 cell proliferation in mice Second hand smoke and COPD: lessons from animal studies.Correlation of Cigarette Smoke-Induced Pulmonary Inflammation and Emphysema in C3H and C57Bl/6 Mice.Tobacco smoke induced COPD/emphysema in the animal model-are we all on the same page?Chronic NF-κB activation links COPD and lung cancer through generation of an immunosuppressive microenvironment in the lungs.The pulmonary endothelial glycocalyx regulates neutrophil adhesion and lung injury during experimental sepsis.Role of IL-18 in second-hand smoke-induced emphysema α₁-Antitrypsin modulates lung endothelial cell inflammatory responses to TNF-α Ascorbate attenuates pulmonary emphysema by inhibiting tobacco smoke and Rtp801-triggered lung protein modification and proteolysis.Sestrin-2, a repressor of PDGFRβ signalling, promotes cigarette-smoke-induced pulmonary emphysema in mice and is upregulated in individuals with COPD.Cigarette smoke reversibly activates hypoxia-inducible factor 1 in a reactive oxygen species-dependent manner.Formaldehyde impairs transepithelial sodium transport.Heparanase mediates renal dysfunction during early sepsis in mice.DNA Damage-Inducible Transcript 4 Is an Innate Surveillant of Hair Follicular Stress in Vitamin D Receptor Knockout Mice and a Regulator of Wound Re-Epithelialization.The Role of Mitochondria and Oxidative/Antioxidative Imbalance in Pathobiology of Chronic Obstructive Pulmonary Disease

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Rtp801, a suppressor of mTOR signaling, is an essential mediator of cigarette smoke-induced pulmonary injury and emphysema

http://www.wikidata.org/entity/Q28397712

description

2010 nî lūn-bûn

@nan

2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Rtp801, a suppressor of mTOR s ...... pulmonary injury and emphysema

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Rtp801, a suppressor of mTOR s ...... pulmonary injury and emphysema

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Rtp801, a suppressor of mTOR s ...... pulmonary injury and emphysema

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type

label

Rtp801, a suppressor of mTOR s ...... pulmonary injury and emphysema

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Rtp801, a suppressor of mTOR s ...... pulmonary injury and emphysema

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Rtp801, a suppressor of mTOR s ...... pulmonary injury and emphysema

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prefLabel

Rtp801, a suppressor of mTOR s ...... pulmonary injury and emphysema

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Rtp801, a suppressor of mTOR s ...... pulmonary injury and emphysema

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Rtp801, a suppressor of mTOR s ...... pulmonary injury and emphysema

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Nature Medicine

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Rtp801, a suppressor of mTOR s ...... pulmonary injury and emphysema

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Amy Richter

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Aneta Gandjeva

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Anil K Bhunia

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Anna Gelfand

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Elena Feinstein

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Emile Brown

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Gregory Cosgrove

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Hagit Ashush

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Igor Mett

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Irina Petrache

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P2860

Arsenite induces a cell stress-response gene, RTP801, through reactive oxygen species and transcription factors Elk-1 and CCAAT/enhancer-binding protein

REDD1, a developmentally regulated transcriptional target of p63 and p53, links p63 to regulation of reactive oxygen species

IKK beta suppression of TSC1 links inflammation and tumor angiogenesis via the mTOR pathway

Signaling by target of rapamycin proteins in cell growth control

Regulation of mTOR and cell growth in response to energy stress by REDD1.

Identification of a novel hypoxia-inducible factor 1-responsive gene, RTP801, involved in apoptosis.

Regulation of hypoxia-inducible factor 1alpha expression and function by the mammalian target of rapamycin.

Regulation of mTOR function in response to hypoxia by REDD1 and the TSC1/TSC2 tumor suppressor complex

Hypoxia regulates TSC1/2-mTOR signaling and tumor suppression through REDD1-mediated 14-3-3 shuttling

Current development of mTOR inhibitors as anticancer agents

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767-73

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scholarly article

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2892062

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10.1038/NM.2157

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7

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16

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2010-07-01T00:00:00Z

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44602485

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1033917160

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20473305

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3956129

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