Naturally occurring lipid A mutants in neisseria meningitidis from patients with invasive meningococcal disease are associated with reduced coagulopathy
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Infections of People with Complement Deficiencies and Patients Who Have Undergone SplenectomyReview of meningococcal group B vaccines.Cellular and molecular biology of Neisseria meningitidis colonization and invasive diseaseThe transcriptional repressor FarR is not involved in meningococcal fatty acid resistance mediated by the FarAB efflux pump and dependent on lipopolysaccharide structure.The structure of Neisseria meningitidis lipid A determines outcome in experimental meningococcal disease.Does M. tuberculosis genomic diversity explain disease diversity?The sweet tooth of bacteria: common themes in bacterial glycoconjugates.LOS oligosaccharide modification enhances dendritic cell responses to meningococcal native outer membrane vesicles expressing a non-toxic lipid AMeningococcal disease and the complement system.Humanized TLR4/MD-2 mice reveal LPS recognition differentially impacts susceptibility to Yersinia pestis and Salmonella entericaMeningococcal factor H binding protein fHbpd184 polymorphism influences clinical course of meningococcal meningitis.Prevalence and clinical course in invasive infections with meningococcal endotoxin variants.Bordetella pertussis naturally occurring isolates with altered lipooligosaccharide structure fail to fully mature human dendritic cellsFrom agonist to antagonist: structure and dynamics of innate immune glycoprotein MD-2 upon recognition of variably acylated bacterial endotoxinsGenome-Based Characterization of Emergent Invasive Neisseria meningitidis Serogroup Y Isolates in Sweden from 1995 to 2012.Invasive meningococcal capsular group Y disease, England and Wales, 2007-2009.Lipooligosaccharide Structures of Invasive and Carrier Isolates of Neisseria meningitidis Are Correlated with Pathogenicity and CarriageHexa-acylated lipid A is required for host inflammatory response to Neisseria gonorrhoeae in experimental gonorrheaLipopolysaccharide engineering in Neisseria meningitidis: structural analysis of different pentaacyl lipid A mutants and comparison of their modified agonist properties.Large scale genomic analysis shows no evidence for pathogen adaptation between the blood and cerebrospinal fluid niches during bacterial meningitis.Recognition of lipid A variants by the TLR4-MD-2 receptor complex.Pathogenesis of meningococcemia.Biosynthetically engineered lipopolysaccharide as vaccine adjuvant.Antimicrobial peptide resistance in Neisseria meningitidis.Fighting Neisseria meningitidis: past and current vaccination strategies.The Neisseria meningitidis lpxL1 mutant induces less tissue factor expression and activity in primary human monocytes and monocyte-derived microvesicles than the wild type meningococcus.Innate immune recognition and inflammation in Neisseria meningitidis infection.Different cytokine production and Toll-like receptor expression induced by heat-killed invasive and carrier strains of Neisseria meningitidis.Innate immune response to lipooligosaccharide: pivotal regulator of the pathobiology of invasive Neisseria meningitidis infections.Evasion of human innate immunity without antagonizing TLR4 by mutant Salmonella enterica serovar Typhimurium having penta-acylated lipid A.Persistent occurrence of serogroup Y/sequence type (ST)-23 complex invasive meningococcal disease among patients aged five to 14 years, Italy, 2007 to 2013.Structural Modifications of Bacterial Lipopolysaccharide that Facilitate Gram-Negative Bacteria Evasion of Host Innate Immunity.Lack of lipid A pyrophosphorylation and functional lptA reduces inflammation by Neisseria commensalsWhole-blood incubation with the Neisseria meningitidis lpxL1 mutant induces less pro-inflammatory cytokines than the wild type, and IL-10 reduces the MyD88-dependent cytokines.
P2860
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P2860
Naturally occurring lipid A mutants in neisseria meningitidis from patients with invasive meningococcal disease are associated with reduced coagulopathy
description
2009 nî lūn-bûn
@nan
2009 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Naturally occurring lipid A mu ...... ated with reduced coagulopathy
@ast
Naturally occurring lipid A mu ...... ated with reduced coagulopathy
@en
Naturally occurring lipid A mu ...... ated with reduced coagulopathy
@nl
type
label
Naturally occurring lipid A mu ...... ated with reduced coagulopathy
@ast
Naturally occurring lipid A mu ...... ated with reduced coagulopathy
@en
Naturally occurring lipid A mu ...... ated with reduced coagulopathy
@nl
prefLabel
Naturally occurring lipid A mu ...... ated with reduced coagulopathy
@ast
Naturally occurring lipid A mu ...... ated with reduced coagulopathy
@en
Naturally occurring lipid A mu ...... ated with reduced coagulopathy
@nl
P2093
P2860
P50
P1433
P1476
Naturally occurring lipid A mu ...... ated with reduced coagulopathy
@en
P2093
Claire J P Boog
Floris Fransen
Hendrik Jan Hamstra
Jos P M van Putten
Moniek Feller
Peter van der Ley
Sebastiaan G B Heckenberg
P2860
P304
P356
10.1371/JOURNAL.PPAT.1000396
P407
P577
2009-04-01T00:00:00Z