Episomal viral cDNAs identify a reservoir that fuels viral rebound after treatment interruption and that contributes to treatment failure - Wikidata - wikimedia - TriplyDB

Episomal viral cDNAs identify a reservoir that fuels viral rebound after treatment interruption and that contributes to treatment failure

Episomal viral cDNAs identify a reservoir that fuels viral rebound after treatment interruption and that contributes to treatment failure

http://www.wikidata.org/entity/Q28477257

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Post-transcriptional gene silencing, transcriptional gene silencing and human immunodeficiency virus Quantification of viral infection dynamics in animal experiments Phosphorylation of CDK9 at Ser175 enhances HIV transcription and is a marker of activated P-TEFb in CD4(+) T lymphocytes Advances in biosensing strategies for HIV-1 detection, diagnosis, and therapeutic monitoring.Highly divergent patterns of genetic diversity and evolution in proviral quasispecies from HIV controllers.High-throughput Characterization of HIV-1 Reservoir Reactivation Using a Single-Cell-in-Droplet PCR Assay.Rapid turnover of 2-LTR HIV-1 DNA during early stage of highly active antiretroviral therapy.Long-term antiretroviral treatment initiated at primary HIV-1 infection affects the size, composition, and decay kinetics of the reservoir of HIV-1-infected CD4 T cells Incorporating alternative splicing and mRNA editing into the genetic analysis of complex traits.HIV controllers with different viral load cutoff levels have distinct virologic and immunologic profiles.The role of unintegrated DNA in HIV infection Integrated and Total HIV-1 DNA Predict Ex Vivo Viral Outgrowth Th1/17 Polarization of CD4 T Cells Supports HIV-1 Persistence during Antiretroviral Therapy.Impact of a decade of successful antiretroviral therapy initiated at HIV-1 seroconversion on blood and rectal reservoirs.Concurrent measures of total and integrated HIV DNA monitor reservoirs and ongoing replication in eradication trials The role of HIV integration in viral persistence: no more whistling past the proviral graveyard.Peripheral T Follicular Helper Cells Are the Major HIV Reservoir within Central Memory CD4 T Cells in Peripheral Blood from Chronically HIV-Infected Individuals on Combination Antiretroviral Therapy.Long-term reduction in peripheral blood HIV type 1 reservoirs following reduced-intensity conditioning allogeneic stem cell transplantation Cell-associated HIV RNA: a dynamic biomarker of viral persistence.Modelling HIV-1 2-LTR dynamics following raltegravir intensification.Short Intracellular HIV-1 Transcripts as Biomarkers of Residual Immune Activation in Patients on Antiretroviral Therapy Lack of concordance between residual viremia and viral variants driving de novo infection of CD4(+) T cells on ART.How to best measure HIV reservoirs?Effects of raltegravir on 2-long terminal repeat circle junctions in HIV type 1 viremic and aviremic patients An HIV-1 replication pathway utilizing reverse transcription products that fail to integrate.Spatial modeling of HIV cryptic viremia and 2-LTR formation during raltegravir intensification.HIV-1 persistence in CD4+ T cells with stem cell-like properties.Potential strategies for an HIV infection cure.HIV reservoirs and strategies for eradication.Eliminating the HIV reservoir.Targeting HIV latency: resting memory T cells, hematopoietic progenitor cells and future directions.Analysis of multiple cell reservoirs expressing unspliced HIV-1 gag-pol mRNA in patients on antiretroviral therapy Accurate quantification of episomal HIV-1 two-long terminal repeat circles by use of optimized DNA isolation and droplet digital PCR.The Use of Ultra-Sensitive Molecular Assays in HIV Cure-Related Research.Next-generation sequencing analyses of the emergence and maintenance of mutations in CTL epitopes in HIV controllers with differential viremia control How to escape treatment

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Episomal viral cDNAs identify a reservoir that fuels viral rebound after treatment interruption and that contributes to treatment failure

http://www.wikidata.org/entity/Q28477257

description

2011 nî lūn-bûn

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2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2011 թվականի փետրվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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Episomal viral cDNAs identify ...... ntributes to treatment failure

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Episomal viral cDNAs identify ...... ntributes to treatment failure

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Episomal viral cDNAs identify ...... ntributes to treatment failure

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Episomal viral cDNAs identify ...... ntributes to treatment failure

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Episomal viral cDNAs identify ...... ntributes to treatment failure

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Episomal viral cDNAs identify ...... ntributes to treatment failure

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Episomal viral cDNAs identify ...... ntributes to treatment failure

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JOURNAL.PPAT.1001303

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Episomal viral cDNAs identify ...... ntributes to treatment failure

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Daniel R Kuritzkes

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Dunja Z Babic

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Mario Stevenson

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Mark Sharkey

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Roy Gulick

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Thomas Greenough

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P2860

Discovery and characterization of vicriviroc (SCH 417690), a CCR5 antagonist with potent activity against human immunodeficiency virus type 1

Residual human immunodeficiency virus type 1 viremia in some patients on antiretroviral therapy is dominated by a small number of invariant clones rarely found in circulating CD4+ T cells

MEGA4: Molecular Evolutionary Genetics Analysis (MEGA) Software Version 4.0

Identification of a reservoir for HIV-1 in patients on highly active antiretroviral therapy

The effect of raltegravir intensification on low-level residual viremia in HIV-infected patients on antiretroviral therapy: a randomized controlled trial

Latent infection of CD4+ T cells provides a mechanism for lifelong persistence of HIV-1, even in patients on effective combination therapy

Presence of an inducible HIV-1 latent reservoir during highly active antiretroviral therapy

Recovery of replication-competent HIV despite prolonged suppression of plasma viremia

HIV reservoir size and persistence are driven by T cell survival and homeostatic proliferation

In vivo evidence for instability of episomal human immunodeficiency virus type 1 cDNA.

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10.1371/JOURNAL.PPAT.1001303

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21383975

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3044693

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