Duox, Flotillin-2, and Src42A are required to activate or delimit the spread of the transcriptional response to epidermal wounds in Drosophila - Wikidata - wikimedia - TriplyDB

Duox, Flotillin-2, and Src42A are required to activate or delimit the spread of the transcriptional response to epidermal wounds in Drosophila

Duox, Flotillin-2, and Src42A are required to activate or delimit the spread of the transcriptional response to epidermal wounds in Drosophila

http://www.wikidata.org/entity/Q28478651

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DrosophilaEmbryos as a Model for Wound-Induced Transcriptional Dynamics: Genetic Strategies to Achieve a Localized Wound Response Epidermal Wound Healing in the Nematode Caenorhabditis elegans Role of DUOX in gut inflammation: lessons from Drosophila model of gut-microbiota interactions Mechanisms of epithelial wound detection Identification and functional analysis of healing regulators in Drosophila Tetraspanin is required for generation of reactive oxygen species by the dual oxidase system in Caenorhabditis elegans Communicating Science through a Novel Type of Journal.Evolution of the ferric reductase domain (FRD) superfamily: modularity, functional diversification, and signature motifs Transcriptional regulation of flotillins by the extracellularly regulated kinases and retinoid X receptor complexes.ATP-mediated transactivation of the epidermal growth factor receptor in airway epithelial cells involves DUOX1-dependent oxidation of Src and ADAM17.Selective sweep in the Flotillin-2 region of European Drosophila melanogaster.Serine proteolytic pathway activation reveals an expanded ensemble of wound response genes in Drosophila.Gut homeostasis in a microbial world: insights from Drosophila melanogaster.Microinjection wound assay and in vivo localization of epidermal wound response reporters in Drosophila embryos.Hydrogen peroxide as a damage signal in tissue injury and inflammation: murderer, mediator, or messenger?Draper/CED-1 mediates an ancient damage response to control inflammatory blood cell migration in vivo.Early redox, Src family kinase, and calcium signaling integrate wound responses and tissue regeneration in zebrafish.Evolutionary origin and function of NOX4-art, an arthropod specific NADPH oxidase.Extracellular Reactive Oxygen Species Drive Apoptosis-Induced Proliferation via Drosophila Macrophages.Dual oxidase-1 is required for airway epithelial cell migration and bronchiolar reepithelialization after injury.Insertion mutants in Drosophila melanogaster Hsc20 halt larval growth and lead to reduced iron-sulfur cluster enzyme activities and impaired iron homeostasis.Dual oxidases control release of hydrogen peroxide by the gastric epithelium to prevent Helicobacter felis infection and inflammation in mice.Identification of DUOX1-dependent redox signaling through protein S-glutathionylation in airway epithelial cells.Conventional and non-conventional Drosophila Toll signaling.Drosophila blood cell chemotaxis Will the wound-healing field earn its wings?The immediate wound-induced oxidative burst of Saccharina latissima depends on light via photosynthetic electron transport.Inducible defenses stay up late: temporal patterns of immune gene expression in Tenebrio molitor Toll pathway is required for wound-induced expression of barrier repair genes in the Drosophila epidermis Rapid clearance of epigenetic protein reporters from wound edge cells in Drosophila larvae does not depend on the JNK or PDGFR/VEGFR signaling pathways.The evolving role of the orphan nuclear receptor ftz-f1, a pair-rule segmentation gene.Hippo, TGF-β, and Src-MAPK pathways regulate transcription of the upd3 cytokine in Drosophila enterocytes upon bacterial infection.DUAL OXIDASE: A NOVEL THERAPEUTIC TARGET IN ALLERGIC DISEASE.Automated multidimensional image analysis reveals a role for Abl in embryonic wound repair.Why mammalian wound-healing researchers may wish to turn to Drosophila as a model.DUOX1 Silencing in Mammary Cell Alters the Response to Genotoxic Stress.How Does a Fruit Fly Say “Ouch”?Translating Research as an Approach to Enhance Science Engagement

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Duox, Flotillin-2, and Src42A are required to activate or delimit the spread of the transcriptional response to epidermal wounds in Drosophila

http://www.wikidata.org/entity/Q28478651

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2011 nî lūn-bûn

@nan

2011 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Duox, Flotillin-2, and Src42A ...... epidermal wounds in Drosophila

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Duox, Flotillin-2, and Src42A ...... epidermal wounds in Drosophila

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Duox, Flotillin-2, and Src42A ...... epidermal wounds in Drosophila

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type

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Duox, Flotillin-2, and Src42A ...... epidermal wounds in Drosophila

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Duox, Flotillin-2, and Src42A ...... epidermal wounds in Drosophila

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Duox, Flotillin-2, and Src42A ...... epidermal wounds in Drosophila

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prefLabel

Duox, Flotillin-2, and Src42A ...... epidermal wounds in Drosophila

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Duox, Flotillin-2, and Src42A ...... epidermal wounds in Drosophila

@en

Duox, Flotillin-2, and Src42A ...... epidermal wounds in Drosophila

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JOURNAL.PGEN.1002424

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Duox, Flotillin-2, and Src42A ...... epidermal wounds in Drosophila

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Efren Sandoval-Guillen

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Rachel A Patterson

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William McGinnis

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P2860

Cellular and genetic analysis of wound healing in Drosophila larvae

Flotillin and epidermal surface antigen define a new family of caveolae-associated integral membrane proteins

SU6656, a selective src family kinase inhibitor, used to probe growth factor signaling

Glycosylphosphatidyl inositol-anchored proteins and fyn kinase assemble in noncaveolar plasma membrane microdomains defined by reggie-1 and -2

Flotillin microdomains interact with the cortical cytoskeleton to control uropod formation and neutrophil recruitment

A tissue-scale gradient of hydrogen peroxide mediates rapid wound detection in zebrafish.

A Drosophila full-length cDNA resource

Targeted gene expression as a means of altering cell fates and generating dominant phenotypes

The reggie/flotillin connection to growth

A direct role for dual oxidase in Drosophila gut immunity

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e1002424

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scholarly article

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12

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Michelle T Juarez

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221744683

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22242003

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