Cell death control: the interplay of apoptosis and autophagy in the pathogenicity of Sclerotinia sclerotiorum
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Emerging Trends in Molecular Interactions between Plants and the Broad Host Range Fungal Pathogens Botrytis cinerea and Sclerotinia sclerotiorumTrehalose Accumulation Triggers Autophagy during Plant DesiccationManipulation of the Xanthophyll Cycle Increases Plant Susceptibility to Sclerotinia sclerotiorumFungi infecting plants and animals: killers, non-killers, and cell death.Impression cytology implicates cell autophagy in aqueous deficiency dry eye.The biocontrol agent Pseudomonas chlororaphis PA23 primes Brassica napus defenses through distinct gene networks.Autophagy and its link to type II diabetes mellitusTomato SlMKK2 and SlMKK4 contribute to disease resistance against Botrytis cinerea.Hijacking of an autophagy-like process is critical for the life cycle of a DNA virus infecting oceanic algal blooms.Tripping on acid: trans-kingdom perspectives on biological acids in immunity and pathogenesis.Death be not proud--cell death control in plant fungal interactions.Analysis of genes that are differentially expressed during the Sclerotinia sclerotiorum-Phaseolus vulgaris interaction.Candidate effector proteins of the necrotrophic apple canker pathogen Valsa mali can suppress BAX-induced PCD.A Small Secreted Virulence-Related Protein Is Essential for the Necrotrophic Interactions of Sclerotinia sclerotiorum with Its Host Plants.Cellular Tracking and Gene Profiling of Fusarium graminearum during Maize Stalk Rot Disease Development Elucidates Its Strategies in Confronting Phosphorus Limitation in the Host Apoplast.Live-cell fluorescence imaging to investigate the dynamics of plant cell death during infection by the rice blast fungus Magnaporthe oryzae.A "footprint" of plant carbon fixation cycle functions during the development of a heterotrophic fungus.Transcriptome analysis of the Brassica napus-Leptosphaeria maculans pathosystem identifies receptor, signaling and structural genes underlying plant resistance.Changes in the Sclerotinia sclerotiorum transcriptome during infection of Brassica napus.A Phytophthora sojae effector suppresses endoplasmic reticulum stress-mediated immunity by stabilizing plant Binding immunoglobulin ProteinsTranscriptomic comparison between Brassica oleracea and rice (Oryza sativa) reveals diverse modulations on cell death in response to Sclerotinia sclerotiorum.Cell death paradigms in the pathogenesis of Mycobacterium tuberculosis infection.Autophagy functions as an antiviral mechanism against geminiviruses in plants.The perplexing role of autophagy in plant innate immune responses.Botrytis species: relentless necrotrophic thugs or endophytes gone rogue?When supply does not meet demand-ER stress and plant programmed cell deathNew advances in autophagy in plants: Regulation, selectivity and function.Arabidopsis B-cell lymphoma2 (Bcl-2)-associated athanogene 7 (BAG7)-mediated heat tolerance requires translocation, sumoylation and binding to WRKY29.The pathogenic development of Sclerotinia sclerotiorum in soybean requires specific host NADPH oxidases.BcXYG1, a Secreted Xyloglucanase from Botrytis cinerea, Triggers Both Cell Death and Plant Immune Responses.Plant Bax Inhibitor-1 interacts with ATG6 to regulate autophagy and programmed cell death.The six Tomato yellow leaf curl virus genes expressed individually in tomato induce different levels of plant stress response attenuation.Ss-Rhs1, a secretory Rhs repeat-containing protein, is required for the virulence of Sclerotinia sclerotiorum.An atypical forkhead-containing transcription factor SsFKH1 is involved in sclerotial formation and is essential for pathogenicity in Sclerotinia sclerotiorum.Spore Density Determines Infection Strategy by the Plant Pathogenic Fungus Plectosphaerella cucumerina.Tomato plant cell death induced by inhibition of HSP90 is alleviated by Tomato yellow leaf curl virus infection.Fungal oxalate decarboxylase activity contributes to Sclerotinia sclerotiorum early infection by affecting both compound appressoria development and function.Primary metabolism plays a central role in moulding silicon-inducible brown spot resistance in rice.Oxaloacetate acetylhydrolase gene mutants of Sclerotinia sclerotiorum do not accumulate oxalic acid, but do produce limited lesions on host plants.BcCFEM1, a CFEM Domain-Containing Protein with Putative GPI-Anchored Site, Is Involved in Pathogenicity, Conidial Production, and Stress Tolerance in Botrytis cinerea.
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P2860
Cell death control: the interplay of apoptosis and autophagy in the pathogenicity of Sclerotinia sclerotiorum
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2013 nî lūn-bûn
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2013 թուականին հրատարակուած գիտական յօդուած
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2013 թվականին հրատարակված գիտական հոդված
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年论文
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name
Cell death control: the interp ...... ty of Sclerotinia sclerotiorum
@ast
Cell death control: the interp ...... ty of Sclerotinia sclerotiorum
@en
Cell death control: the interp ...... ty of Sclerotinia sclerotiorum
@nl
type
label
Cell death control: the interp ...... ty of Sclerotinia sclerotiorum
@ast
Cell death control: the interp ...... ty of Sclerotinia sclerotiorum
@en
Cell death control: the interp ...... ty of Sclerotinia sclerotiorum
@nl
prefLabel
Cell death control: the interp ...... ty of Sclerotinia sclerotiorum
@ast
Cell death control: the interp ...... ty of Sclerotinia sclerotiorum
@en
Cell death control: the interp ...... ty of Sclerotinia sclerotiorum
@nl
P2860
P3181
P1433
P1476
Cell death control: the interp ...... ty of Sclerotinia sclerotiorum
@en
P2093
Martin B Dickman
Mehdi Kabbage
P2860
P304
P3181
P356
10.1371/JOURNAL.PPAT.1003287
P407
P577
2013-04-11T00:00:00Z