Crosstalk from non-cancerous mitochondria can inhibit tumor properties of metastatic cells by suppressing oncogenic pathways
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Homeostasis of redox status derived from glucose metabolic pathway could be the key to understanding the Warburg effect.Cancer as a mitochondrial metabolic disease.Mitochondrial DNA variants can mediate methylation status of inflammation, angiogenesis and signaling genes.Enhanced tumorigenicity by mitochondrial DNA mild mutationsFatty Acid Oxidation-Driven Src Links Mitochondrial Energy Reprogramming and Oncogenic Properties in Triple-Negative Breast Cancer.Mitochondrial retrograde signaling at the crossroads of tumor bioenergetics, genetics and epigenetics.Origin of Cancer: An Information, Energy, and Matter Disease.Mitochondrial DNA mutations and breast tumorigenesis.Cancer as a metabolic disease: implications for novel therapeutics.Mitochondria in lung diseases.The emergence of the mitochondrial genome as a partial regulator of nuclear function is providing new insights into the genetic mechanisms underlying age-related complex disease.From radioresistance to radiosensitivity: In vitro evolution of L5178Y lymphoma.Anticancer properties of lamellarins.Mitochondrial-epigenetic crosstalk in environmental toxicology.Metabolic changes associated with tumor metastasis, part 2: Mitochondria, lipid and amino acid metabolism.Mitochondrial remodeling: Rearranging, recycling, and reprogramming.Individual-specific variation in the respiratory activities of HMECs and their bioenergetic response to IGF1 and TNFα.Mitochondrial DNA variants in colorectal carcinogenesis: Drivers or passengers?Mitochondrial determinants of cancer health disparities.Press-pulse: a novel therapeutic strategy for the metabolic management of cancer.Aging, Metabolism, and Cancer Development: from Peto's Paradox to the Warburg Effect.Homeostasis of redox status derived from glucose metabolic pathway could be the key to understanding the Warburg effect.The Landscape of mtDNA Modifications in Cancer: A Tale of Two Cities.Epigenetic modification of miR-663 controls mitochondria-to-nucleus retrograde signaling and tumor progression.The synergistic antitumor effect of cinobufagin and cisplatin in human osteosarcoma cell line in vitro and in vivo.Elucidating the Metabolic Plasticity of Cancer: Mitochondrial Reprogramming and Hybrid Metabolic States.Mitochondrial genome instability in colorectal adenoma and adenocarcinoma.Mitochondrial variants in MT-CO2 and D-loop instability are involved in MUTYH-associated polyposis.
P2860
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P2860
Crosstalk from non-cancerous mitochondria can inhibit tumor properties of metastatic cells by suppressing oncogenic pathways
description
2013 nî lūn-bûn
@nan
2013 թուականին հրատարակուած գիտական յօդուած
@hyw
2013 թվականին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Crosstalk from non-cancerous m ...... suppressing oncogenic pathways
@ast
Crosstalk from non-cancerous m ...... suppressing oncogenic pathways
@en
Crosstalk from non-cancerous m ...... suppressing oncogenic pathways
@nl
type
label
Crosstalk from non-cancerous m ...... suppressing oncogenic pathways
@ast
Crosstalk from non-cancerous m ...... suppressing oncogenic pathways
@en
Crosstalk from non-cancerous m ...... suppressing oncogenic pathways
@nl
prefLabel
Crosstalk from non-cancerous m ...... suppressing oncogenic pathways
@ast
Crosstalk from non-cancerous m ...... suppressing oncogenic pathways
@en
Crosstalk from non-cancerous m ...... suppressing oncogenic pathways
@nl
P2093
P2860
P50
P1433
P1476
Crosstalk from non-cancerous m ...... suppressing oncogenic pathways
@en
P2093
Chad J Creighton
Jun Hyoung Park
Lee-Jun C Wong
Patricia Yotnda
Yiqun Zhang
P2860
P304
P356
10.1371/JOURNAL.PONE.0061747
P407
P577
2013-05-09T00:00:00Z