Nuclear localization of the C1 factor (host cell factor) in sensory neurons correlates with reactivation of herpes simplex virus from latency
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Potential role for luman, the cellular homologue of herpes simplex virus VP16 (alpha gene trans-inducing factor), in herpesvirus latencyThe novel coactivator C1 (HCF) coordinates multiprotein enhancer formation and mediates transcription activation by GABPZhangfei: a second cellular protein interacts with herpes simplex virus accessory factor HCF in a manner similar to Luman and VP16.HCF-1 functions as a coactivator for the zinc finger protein Krox20Association of C-terminal ubiquitin hydrolase BRCA1-associated protein 1 with cell cycle regulator host cell factor 1An activation domain in the C-terminal subunit of HCF-1 is important for transactivation by VP16 and LZIP.Luman, the cellular counterpart of herpes simplex virus VP16, is processed by regulated intramembrane proteolysisA novel function for human factor C1 (HCF-1), a host protein required for herpes simplex virus infection, in pre-mRNA splicingA novel 50-kilodalton fragment of host cell factor 1 (C1) in G(0) cellsA comparison of herpes simplex virus type 1 and varicella-zoster virus latency and reactivationThe molecular basis of herpes simplex virus latencyInteraction of HCF-1 with a cellular nuclear export factorPDCD2 is a negative regulator of HCF-1 (C1)Transient reversal of episome silencing precedes VP16-dependent transcription during reactivation of latent HSV-1 in neuronsGenital Herpes: Insights into Sexually Transmitted Infectious DiseaseRole of chromatin during herpesvirus infections.Transcriptional coactivator HCF-1 couples the histone chaperone Asf1b to HSV-1 DNA replication componentsTowards an understanding of the herpes simplex virus type 1 latency-reactivation cycleControl of alpha-herpesvirus IE gene expression by HCF-1 coupled chromatin modification activities.Analysis of HCF, the cellular cofactor of VP16, in herpes simplex virus-infected cells.Gene array analysis reveals changes in peripheral nervous system gene expression following stimuli that result in reactivation of latent herpes simplex virus type 1: induction of transcription factor Bcl-3.The neuronal host cell factor-binding protein Zhangfei inhibits herpes simplex virus replication.Tegument protein control of latent herpesvirus establishment and animation.A 2.5-kilobase deletion containing a cluster of nine microRNAs in the latency-associated-transcript locus of the pseudorabies virus affects the host response of porcine trigeminal ganglia during established latency.Herpes simplex virus type 1 and bovine herpesvirus 1 latency.The checkpoints of viral gene expression in productive and latent infection: the role of the HDAC/CoREST/LSD1/REST repressor complexAutocatalytic proteolysis of the transcription factor-coactivator C1 (HCF): a potential role for proteolytic regulation of coactivator function.Dynamic modulation of HSV chromatin drives initiation of infection and provides targets for epigenetic therapies.Nuclear sensing of viral DNA, epigenetic regulation of herpes simplex virus infection, and innate immunityRoles of the nuclear lamina in stable nuclear association and assembly of a herpesviral transactivator complex on viral immediate-early genes.Latent herpes simplex virus infection of sensory neurons alters neuronal gene expression.The coactivator host cell factor-1 mediates Set1 and MLL1 H3K4 trimethylation at herpesvirus immediate early promoters for initiation of infection.The HSV-1 Latency-Associated Transcript Functions to Repress Latent Phase Lytic Gene Expression and Suppress Virus Reactivation from Latently Infected NeuronsHerpes simplex virus infections are arrested in Oct-1-deficient cellsHerpes Simplex Virus 1 Infection of Tree Shrews Differs from That of Mice in the Severity of Acute Infection and Viral Transcription in the Peripheral Nervous SystemKinetics of facultative heterochromatin and polycomb group protein association with the herpes simplex viral genome during establishment of latent infection.C-terminal trans-activation sub-region of VP16 is uniquely required for forskolin-induced herpes simplex virus type 1 reactivation from quiescently infected-PC12 cells but not for replication in neuronally differentiated-PC12 cells.Herpes simplex virus 1 microRNAs expressed abundantly during latent infection are not essential for latency in mouse trigeminal gangliaTargeting the JMJD2 histone demethylases to epigenetically control herpesvirus infection and reactivation from latencyAssociation of the cellular coactivator HCF-1 with the Golgi apparatus in sensory neurons.
P2860
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P2860
Nuclear localization of the C1 factor (host cell factor) in sensory neurons correlates with reactivation of herpes simplex virus from latency
description
1999 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
article publié dans les Procee ...... f the United States of America
@fr
artículu científicu espublizáu en 1999
@ast
im Februar 1999 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 1999/02/16)
@sk
vědecký článek publikovaný v roce 1999
@cs
wetenschappelijk artikel (gepubliceerd op 1999/02/16)
@nl
наукова стаття, опублікована в лютому 1999
@uk
name
Nuclear localization of the C1 ...... pes simplex virus from latency
@ast
Nuclear localization of the C1 ...... pes simplex virus from latency
@en
Nuclear localization of the C1 ...... pes simplex virus from latency
@nl
type
label
Nuclear localization of the C1 ...... pes simplex virus from latency
@ast
Nuclear localization of the C1 ...... pes simplex virus from latency
@en
Nuclear localization of the C1 ...... pes simplex virus from latency
@nl
prefLabel
Nuclear localization of the C1 ...... pes simplex virus from latency
@ast
Nuclear localization of the C1 ...... pes simplex virus from latency
@en
Nuclear localization of the C1 ...... pes simplex virus from latency
@nl
P2093
P2860
P3181
P356
P1476
Nuclear localization of the C1 ...... pes simplex virus from latency
@en
P2093
A. E. Sears
J. L. Vogel
T. M. Kristie
P2860
P304
P3181
P356
10.1073/PNAS.96.4.1229
P407
P577
1999-02-16T00:00:00Z