Mice lacking JunB are osteopenic due to cell-autonomous osteoblast and osteoclast defects
about
Distinct roles for mitogen-activated protein kinase phosphatase-1 (MKP-1) and ERK-MAPK in PTH1R signaling during osteoblast proliferation and differentiationMenin suppresses osteoblast differentiation by antagonizing the AP-1 factor, JunDDoubly truncated FosB isoform (Delta2DeltaFosB) induces osteosclerosis in transgenic mice and modulates expression and phosphorylation of Smads in osteoblasts independent of intrinsic AP-1 activityDeletion of Jun proteins in adult oligodendrocytes does not perturb cell survival, or myelin maintenance in vivoTargeting WW domains linker of HECT-type ubiquitin ligase Smurf1 for activation by CKIP-1The estrogen receptor-alpha in osteoclasts mediates the protective effects of estrogens on cancellous but not cortical boneJunb regulates arterial contraction capacity, cellular contractility, and motility via its target Myl9 in miceUbiquitin ligase Smurf1 controls osteoblast activity and bone homeostasis by targeting MEKK2 for degradationMouse genome-wide association and systems genetics identify Asxl2 as a regulator of bone mineral density and osteoclastogenesisNFATc1 in mice represses osteoprotegerin during osteoclastogenesis and dissociates systemic osteopenia from inflammation in cherubism.Gene identification and analysis of transcripts differentially regulated in fracture healing by EST sequencing in the domestic sheep.NIK stabilization in osteoclasts results in osteoporosis and enhanced inflammatory osteolysis.Down-regulation of Irf8 by Lyz2-cre/loxP accelerates osteoclast differentiation in vitro.Maf promotes osteoblast differentiation in mice by mediating the age-related switch in mesenchymal cell differentiation.Psoriasis-like skin disease and arthritis caused by inducible epidermal deletion of Jun proteins.[Viennese Clinical Weekly Review].Fra-2/AP-1 controls bone formation by regulating osteoblast differentiation and collagen production.Adipose triglyceride lipase and hormone-sensitive lipase are involved in fat loss in JunB-deficient mice.Smurf1 inhibits mesenchymal stem cell proliferation and differentiation into osteoblasts through JunB degradation.Loss of JUNB/AP-1 promotes invasive prostate cancerGsα enhances commitment of mesenchymal progenitors to the osteoblast lineage but restrains osteoblast differentiation in mice.Reliable quantification of protein expression and cellular localization in histological sections.Positive regulators of osteoclastogenesis and bone resorption in rheumatoid arthritisMolecular genetic studies of gene identification for osteoporosis: a 2004 update.Extracellular Matrix Stiffness Regulates Osteogenic Differentiation through MAPK Activation.RNA therapeutics targeting osteoclast-mediated excessive bone resorption.Trophoblast differentiation during embryo implantation and formation of the maternal-fetal interface.Genetic mouse models for bone studies--strengths and limitationsmiR-34s inhibit osteoblast proliferation and differentiation in the mouse by targeting SATB2.Biochemical and cellular mechanisms of mammalian CDK inhibitors: a few unresolved issues.JunB is required for endothelial cell morphogenesis by regulating core-binding factor beta.Deletion of Orai1 alters expression of multiple genes during osteoclast and osteoblast maturation.Loss of Gsα in the Postnatal Skeleton Leads to Low Bone Mass and a Blunted Response to Anabolic Parathyroid Hormone TherapySignaling and transcriptional regulation in osteoblast commitment and differentiationDevelopment of the endochondral skeletonActivator protein 1 (Fos/Jun) functions in inflammatory bone and skin diseaseA RANKL-PKCβ-TFEB signaling cascade is necessary for lysosomal biogenesis in osteoclasts.Osteoblastic regulation of B lymphopoiesis is mediated by Gs{alpha}-dependent signaling pathwaysUbiquitin E3 ligase Wwp1 negatively regulates osteoblast function by inhibiting osteoblast differentiation and migrationCancer-Osteoblast Interaction Reduces Sost Expression in Osteoblasts and Up-Regulates lncRNA MALAT1 in Prostate Cancer.
P2860
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P2860
Mice lacking JunB are osteopenic due to cell-autonomous osteoblast and osteoclast defects
description
2004 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2004
@ast
im Februar 2004 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2004/02/16)
@sk
vědecký článek publikovaný v roce 2004
@cs
wetenschappelijk artikel (gepubliceerd op 2004/02/16)
@nl
наукова стаття, опублікована в лютому 2004
@uk
مقالة علمية (نشرت في 16-2-2004)
@ar
name
Mice lacking JunB are osteopenic due to cell-autonomous osteoblast and osteoclast defects
@ast
Mice lacking JunB are osteopenic due to cell-autonomous osteoblast and osteoclast defects
@en
Mice lacking JunB are osteopenic due to cell-autonomous osteoblast and osteoclast defects
@nl
type
label
Mice lacking JunB are osteopenic due to cell-autonomous osteoblast and osteoclast defects
@ast
Mice lacking JunB are osteopenic due to cell-autonomous osteoblast and osteoclast defects
@en
Mice lacking JunB are osteopenic due to cell-autonomous osteoblast and osteoclast defects
@nl
prefLabel
Mice lacking JunB are osteopenic due to cell-autonomous osteoblast and osteoclast defects
@ast
Mice lacking JunB are osteopenic due to cell-autonomous osteoblast and osteoclast defects
@en
Mice lacking JunB are osteopenic due to cell-autonomous osteoblast and osteoclast defects
@nl
P2093
P2860
P50
P356
P1476
Mice lacking JunB are osteopenic due to cell-autonomous osteoblast and osteoclast defects
@en
P2093
Agnieszka Szremska
Astrid Hoebertz
F. Timo Beil
Florian Karreth
Harald Scheuch
Marina Schorpp-Kistner
Michael Amling
Niamh Keon
Peter Angel
P2860
P304
P356
10.1083/JCB.200308155
P407
P577
2004-02-16T00:00:00Z