Enhanced hippocampal CA1 LTP but normal spatial learning in inositol 1,4,5-trisphosphate 3-kinase(A)-deficient mice
about
Inositol 1,4,5-trisphosphate 3-kinase a functions as a scaffold for synaptic Rac signalingRegulation of immune cell development through soluble inositol-1,3,4,5-tetrakisphosphateThe Role of Astrocytic Aquaporin-4 in Synaptic Plasticity and Learning and MemoryAquaporin-4 water channels and synaptic plasticity in the hippocampusGenes and signaling pathways involved in memory enhancement in mutant miceThe role of inositol 1,4,5-trisphosphate 3-kinase A in regulating emotional behavior and amygdala function.The regulation and function of inositol 1,4,5-trisphosphate 3-kinasesInositol (1,4,5) trisphosphate 3 kinase B controls positive selection of T cells and modulates Erk activityAblation of P/Q-type Ca(2+) channel currents, altered synaptic transmission, and progressive ataxia in mice lacking the alpha(1A)-subunitIP3 3-kinase opposes NGF driven neurite outgrowthNeuronal IP3 3-kinase is an F-actin-bundling protein: role in dendritic targeting and regulation of spine morphology.Lipid signaling in T-cell development and functionThe molecular and cellular biology of enhanced cognition.Intrahippocampal wortmannin infusion enhances long-term spatial and contextual memories.A systematic investigation of the protein kinases involved in NMDA receptor-dependent LTD: evidence for a role of GSK-3 but not other serine/threonine kinasesLearning-induced glutamate receptor phosphorylation resembles that induced by long term potentiation.Issues about the physiological functions of prolyl oligopeptidase based on its discordant spatial association with substrates and inconsistencies among mRNA, protein levels, and enzymatic activityInositol-1,4,5-trisphosphate 3-kinase-A (ITPKA) is frequently over-expressed and functions as an oncogene in several tumor types.Inositol 1,3,4,5-tetrakisphosphate negatively regulates phosphatidylinositol-3,4,5- trisphosphate signaling in neutrophils.Inositol 1,4,5-trisphosphate 3-kinase-A is a new cell motility-promoting protein that increases the metastatic potential of tumor cells by two functional activities.Impairment of select forms of spatial memory and neurotrophin-dependent synaptic plasticity by deletion of glial aquaporin-4.Inositol 1,4,5-trisphosphate 3-kinase A overexpressed in mouse forebrain modulates synaptic transmission and mGluR-LTD of CA1 pyramidal neurons.Regulation of Hematopoietic Cell Development and Function Through Phosphoinositides.
P2860
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P2860
Enhanced hippocampal CA1 LTP but normal spatial learning in inositol 1,4,5-trisphosphate 3-kinase(A)-deficient mice
description
1998 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 1998
@ast
scientific journal article
@en
vedecký článok (publikovaný 1998/09/01)
@sk
vědecký článek publikovaný v roce 1998
@cs
wetenschappelijk artikel (gepubliceerd op 1998/09/01)
@nl
наукова стаття, опублікована у вересні 1998
@uk
مقالة علمية (نشرت في سبتمبر 1998)
@ar
name
Enhanced hippocampal CA1 LTP b ...... ate 3-kinase(A)-deficient mice
@ast
Enhanced hippocampal CA1 LTP b ...... ate 3-kinase(A)-deficient mice
@en
Enhanced hippocampal CA1 LTP b ...... ate 3-kinase(A)-deficient mice
@nl
type
label
Enhanced hippocampal CA1 LTP b ...... ate 3-kinase(A)-deficient mice
@ast
Enhanced hippocampal CA1 LTP b ...... ate 3-kinase(A)-deficient mice
@en
Enhanced hippocampal CA1 LTP b ...... ate 3-kinase(A)-deficient mice
@nl
prefLabel
Enhanced hippocampal CA1 LTP b ...... ate 3-kinase(A)-deficient mice
@ast
Enhanced hippocampal CA1 LTP b ...... ate 3-kinase(A)-deficient mice
@en
Enhanced hippocampal CA1 LTP b ...... ate 3-kinase(A)-deficient mice
@nl
P2093
P2860
P3181
P1433
P1476
Enhanced hippocampal CA1 LTP b ...... ate 3-kinase(A)-deficient mice
@en
P2093
P2860
P304
P3181
P577
1998-09-01T00:00:00Z