Regulation of lymphoid versus myeloid fate 'choice' by the transcription factor Mef2c
about
Dual roles for MEF2A and MEF2D during human macrophage terminal differentiation and c-Jun expressionGATA-3 is required for early T lineage progenitor developmentT-cells null for the MED23 subunit of mediator express decreased levels of KLF2 and inefficiently populate the peripheral lymphoid organsMEF2C and EBF1 Co-regulate B Cell-Specific Transcription.Logical modeling of lymphoid and myeloid cell specification and transdifferentiation.Repetitive busulfan administration after hematopoietic stem cell gene therapy associated with a dominant HDAC7 clone in a nonhuman primate.Dnmt3a is essential for hematopoietic stem cell differentiation.The transcription factors IRF8 and PU.1 negatively regulate plasma cell differentiation.Assessment of benzene-induced hematotoxicity using a human-like hematopoietic lineage in NOD/Shi-scid/IL-2Rγnull miceHDAC7 is a repressor of myeloid genes whose downregulation is required for transdifferentiation of pre-B cells into macrophages.1,25-Dihydroxyvitamin D3 induces monocytic differentiation of human myeloid leukemia cells by regulating C/EBPβ expression through MEF2CMyocyte enhancer factor (MEF)-2 plays essential roles in T-cell transformation associated with HTLV-1 infection by stabilizing complex between Tax and CREBA filter microplate assay for quantitative analysis of DNA binding proteins using fluorescent DNA.BCL2 Inhibitor (ABT-737): A Restorer of Prednisolone Sensitivity in Early T-Cell Precursor-Acute Lymphoblastic Leukemia with High MEF2C Expression?Intraclonal complexity in chronic lymphocytic leukemia: fractions enriched in recently born/divided and older/quiescent cells.High expression of myocyte enhancer factor 2C (MEF2C) is associated with adverse-risk features and poor outcome in pediatric acute myeloid leukemia: a report from the Children's Oncology GroupHhex is Required at Multiple Stages of Adult Hematopoietic Stem and Progenitor Cell Differentiation.Bcl11a is essential for lymphoid development and negatively regulates p53.Bone marrow-induced Mef2c deficiency delays B-cell development and alters the expression of key B-cell regulatory proteins.MicroRNA-126-mediated control of cell fate in B-cell myeloid progenitors as a potential alternative to transcriptional factors.MEF2C protects bone marrow B-lymphoid progenitors during stress haematopoiesis.In vivo conditional deletion of HDAC7 reveals its requirement to establish proper B lymphocyte identity and development.Alu repeats as transcriptional regulatory platforms in macrophage responses to M. tuberculosis infection.Myocyte enhancer factor 2C in hematopoiesis and leukemia.MAP Kinase Cascades in Antigen Receptor Signaling and Physiology.The miR-23a~27a~24-2 microRNA cluster buffers transcription and signaling pathways during hematopoiesis.The transcriptional repressor HDAC7 promotes apoptosis and c-Myc downregulation in particular types of leukemia and lymphoma.Ikaros represses and activates PU.1 cell-type-specifically through the multifunctional Sfpi1 URE and a myeloid specific enhancer.The MEF2C-Related and 5q14.3q15 Microdeletion Syndrome.A recurrent network involving the transcription factors PU.1 and Gfi1 orchestrates innate and adaptive immune cell fatesFernando Camargo: no limits to learning about stem cells. Interview by Caitlin Sedwick.Reprogramming immune responses via microRNA modulation.Suppression of SRCAP chromatin remodelling complex and restriction of lymphoid lineage commitment by Pcid2.Ligament versus bone cell identity in the zebrafish hyoid skeleton is regulated by mef2ca.Recurrent SPI1 (PU.1) fusions in high-risk pediatric T cell acute lymphoblastic leukemia.MEF2C phosphorylation is required for chemotherapy resistance in acute myeloid leukemia.Transcriptional deregulation of oncogenic myocyte enhancer factor 2C in T-cell acute lymphoblastic leukemia.BCR-ABL mediated repression of miR-223 results in the activation of MEF2C and PTBP2 in chronic myeloid leukemia.PU.1 Is Required for the Developmental Progression of Multipotent Progenitors to Common Lymphoid Progenitors.
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P2860
Regulation of lymphoid versus myeloid fate 'choice' by the transcription factor Mef2c
description
2009 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի մարտին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2009
@ast
im März 2009 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2009/03/01)
@sk
vědecký článek publikovaný v roce 2009
@cs
wetenschappelijk artikel (gepubliceerd op 2009/03/01)
@nl
наукова стаття, опублікована в березні 2009
@uk
مقالة علمية (نشرت في مارس 2009)
@ar
name
Regulation of lymphoid versus myeloid fate 'choice' by the transcription factor Mef2c
@ast
Regulation of lymphoid versus myeloid fate 'choice' by the transcription factor Mef2c
@en
Regulation of lymphoid versus myeloid fate 'choice' by the transcription factor Mef2c
@nl
type
label
Regulation of lymphoid versus myeloid fate 'choice' by the transcription factor Mef2c
@ast
Regulation of lymphoid versus myeloid fate 'choice' by the transcription factor Mef2c
@en
Regulation of lymphoid versus myeloid fate 'choice' by the transcription factor Mef2c
@nl
prefLabel
Regulation of lymphoid versus myeloid fate 'choice' by the transcription factor Mef2c
@ast
Regulation of lymphoid versus myeloid fate 'choice' by the transcription factor Mef2c
@en
Regulation of lymphoid versus myeloid fate 'choice' by the transcription factor Mef2c
@nl
P2093
P2860
P3181
P356
P1433
P1476
Regulation of lymphoid versus myeloid fate 'choice' by the transcription factor Mef2c
@en
P2093
Fernando D. Camargo
Jessica Dade
Rodney P. DeKoter
Sandra Stehling-Sun
Stephen L. Nutt
P2860
P2888
P304
P3181
P356
10.1038/NI.1694
P407
P577
2009-03-01T00:00:00Z
P5875
P6179
1002557636