Physiological and glycomic characterization of N-acetylglucosaminyltransferase-IVa and -IVb double deficient mice
about
Enzymes for N-Glycan Branching and Their Genetic and Nongenetic Regulation in CancerThe absence of core fucose up-regulates GnT-III and Wnt target genes: a possible mechanism for an adaptive response in terms of glycan functionGolgi self-correction generates bioequivalent glycans to preserve cellular homeostasis.MS-based glycomic strategies for probing the structural details of polylactosaminoglycan chain on N-glycans and glycoproteomic identification of its protein carriers.Suppression of cancer progression by MGAT1 shRNA knockdown.Systemic blockade of sialylation in mice with a global inhibitor of sialyltransferases.Golgi N-glycan branching N-acetylglucosaminyltransferases I, V and VI promote nutrient uptake and metabolism.Analysis of carbohydrates and glycoconjugates by matrix-assisted laser desorption/ionization mass spectrometry: an update for 2009-2010.Synthesis and secretion of gonadotropins including structure-function correlates.UDP-galactose (SLC35A2) and UDP-N-acetylglucosamine (SLC35A3) Transporters Form Glycosylation-related Complexes with Mannoside Acetylglucosaminyltransferases (Mgats).Specific N-glycans of Hepatocellular Carcinoma Cell Surface and the Abnormal Increase of Core-α-1, 6-fucosylated Triantennary Glycan via N-acetylglucosaminyltransferases-IVa Regulation.MicroRNA-424 Predicts a Role for β-1,4 Branched Glycosylation in Cell Cycle Progression.Mass isotopomer analysis of metabolically labeled nucleotide sugars and N- and O-glycans for tracing nucleotide sugar metabolisms.Enzymatic glycosylation of multivalent scaffolds.Modulation of voltage-gated ion channels by sialylation.DNA hypomethylation upregulates expression of the MGAT3 gene in HepG2 cells and leads to changes in N-glycosylation of secreted glycoproteins.What Have We Learned from Glycosyltransferase Knockouts in Mice?Density-dependent lectin-glycan interactions as a paradigm for conditional regulation by posttranslational modifications.Serum protein N-glycan alterations of diethylnitrosamine-induced hepatocellular carcinoma mice and their evolution after inhibition of the placental growth factor.Giantin-knockout models reveal a feedback loop between Golgi function and glycosyltransferase expression.
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P2860
Physiological and glycomic characterization of N-acetylglucosaminyltransferase-IVa and -IVb double deficient mice
description
2009 nî lūn-bûn
@nan
2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Physiological and glycomic cha ...... and -IVb double deficient mice
@ast
Physiological and glycomic cha ...... and -IVb double deficient mice
@en
Physiological and glycomic cha ...... and -IVb double deficient mice
@nl
type
label
Physiological and glycomic cha ...... and -IVb double deficient mice
@ast
Physiological and glycomic cha ...... and -IVb double deficient mice
@en
Physiological and glycomic cha ...... and -IVb double deficient mice
@nl
prefLabel
Physiological and glycomic cha ...... and -IVb double deficient mice
@ast
Physiological and glycomic cha ...... and -IVb double deficient mice
@en
Physiological and glycomic cha ...... and -IVb double deficient mice
@nl
P2093
P2860
P3181
P356
P1433
P1476
Physiological and glycomic cha ...... and -IVb double deficient mice
@en
P2093
Aristotelis Antonopoulos
David Ditto
Dzung T Le
Howard R Morris
Jamey D Marth
Kazuaki Ohtsubo
Naoyuki Taniguchi
Shinji Takamatsu
Stuart M Haslam
P2860
P304
P3181
P356
10.1093/GLYCOB/CWP200
P577
2009-12-16T00:00:00Z