Defects in pulmonary vasculature and perinatal lung hemorrhage in mice heterozygous null for the Forkhead Box f1 transcription factor
about
Genomic and genic deletions of the FOX gene cluster on 16q24.1 and inactivating mutations of FOXF1 cause alveolar capillary dysplasia and other malformationsUnique aspects of the developing lung circulation: structural development and regulation of vasomotor toneForkhead transcription factor FoxF1 interacts with Fanconi anemia protein complexes to promote DNA damage responsePulmonary mastocytosis and enhanced lung inflammation in mice heterozygous null for the Foxf1 geneRespiratory distress and neonatal lethality in mice lacking Golgi alpha1,2-mannosidase IB involved in N-glycan maturationForkhead box F1 is essential for migration of mesenchymal cells and directly induces integrin-beta3 expressionBasement membrane distortions impair lung lobation and capillary organization in the mouse model for fraser syndromeThe forkhead box m1 transcription factor is essential for embryonic development of pulmonary vasculatureThe molecular era of surfactant biology.Diseases of pulmonary surfactant homeostasis.Chronic obstructive pulmonary disease * 3: Experimental animal models of pulmonary emphysema.Multiple faces of FoxM1 transcription factor: lessons from transgenic mouse models.Resident tissue-specific mesenchymal progenitor cells contribute to fibrogenesis in human lung allograftsComparative analyses of lung transcriptomes in patients with alveolar capillary dysplasia with misalignment of pulmonary veins and in foxf1 heterozygous knockout miceForkhead box F2 regulation of platelet-derived growth factor and myocardin/serum response factor signaling is essential for intestinal developmentAlveolar capillary dysplasiaMesenchymal stromal cells from neonatal tracheal aspirates demonstrate a pattern of lung-specific gene expression.Molecular and clinical analyses of 16q24.1 duplications involving FOXF1 identify an evolutionarily unstable large minisatelliteGenetically determined heterogeneity of lung disease in a mouse model of airway mucus obstruction.Lung organogenesisThe molecular basis for abnormal human lung development.Downregulation of Forkhead box F1 gene expression in the pulmonary vasculature of nitrofen-induced congenital diaphragmatic hernia.Use of transgenic mice to study lung morphogenesis and function.Small noncoding differentially methylated copy-number variants, including lncRNA genes, cause a lethal lung developmental disorder.In control of biology: of mice, men and Foxesβ-catenin and Kras/Foxm1 signaling pathway are critical to restrict Sox9 in basal cells during pulmonary branching morphogenesisNovel FOXF1 mutations in sporadic and familial cases of alveolar capillary dysplasia with misaligned pulmonary veins imply a role for its DNA binding domain.Forkhead Box m1 transcription factor is required for perinatal lung function.Forkhead transcription factors and cardiovascular biologyGestational exposure of mice to secondhand cigarette smoke causes bronchopulmonary dysplasia blocked by the nicotinic receptor antagonist mecamylamineResident cellular components of the lung: developmental aspects.Integrated proteomic and transcriptomic profiling of mouse lung development and Nmyc target genesForkhead box M1 transcriptional factor is required for smooth muscle cells during embryonic development of blood vessels and esophagus.Lethal lung hypoplasia and vascular defects in mice with conditional Foxf1 overexpression.Genetic factors in esophageal atresia, tracheo-esophageal fistula and the VACTERL association: roles for FOXF1 and the 16q24.1 FOX transcription factor gene cluster, and review of the literatureRapid hepatocyte nuclear translocation of the Forkhead Box M1B (FoxM1B) transcription factor caused a transient increase in size of regenerating transgenic hepatocytes.Lung endoderm morphogenesis: gasping for form and function.Lung Circulation.FOXF1 inhibits hematopoietic lineage commitment during early mesoderm specification.FOXF1 transcription factor is required for formation of embryonic vasculature by regulating VEGF signaling in endothelial cells.
P2860
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P2860
Defects in pulmonary vasculature and perinatal lung hemorrhage in mice heterozygous null for the Forkhead Box f1 transcription factor
description
2001 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2001
@ast
im Juli 2001 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2001/07/15)
@sk
vědecký článek publikovaný v roce 2001
@cs
wetenschappelijk artikel (gepubliceerd op 2001/07/15)
@nl
наукова стаття, опублікована в липні 2001
@uk
مقالة علمية (نشرت في 15-7-2001)
@ar
name
Defects in pulmonary vasculatu ...... ad Box f1 transcription factor
@ast
Defects in pulmonary vasculatu ...... ad Box f1 transcription factor
@en
Defects in pulmonary vasculatu ...... ad Box f1 transcription factor
@nl
type
label
Defects in pulmonary vasculatu ...... ad Box f1 transcription factor
@ast
Defects in pulmonary vasculatu ...... ad Box f1 transcription factor
@en
Defects in pulmonary vasculatu ...... ad Box f1 transcription factor
@nl
prefLabel
Defects in pulmonary vasculatu ...... ad Box f1 transcription factor
@ast
Defects in pulmonary vasculatu ...... ad Box f1 transcription factor
@en
Defects in pulmonary vasculatu ...... ad Box f1 transcription factor
@nl
P2093
P3181
P356
P1476
Defects in pulmonary vasculatu ...... ad Box f1 transcription factor
@en
P2093
D. B. Stolz
F. M. Rausa
J. A. Whitsett
R. H. Costa
S. C. Watkins
V. V. Kalinichenko
P304
P3181
P356
10.1006/DBIO.2001.0322
P407
P577
2001-07-15T00:00:00Z