Embryonic fibroblasts from mice lacking Tgif were defective in cell cycling
about
Identification of PCTA, a TGIF antagonist that promotes PML function in TGF-beta signallingTgif1 and Tgif2 regulate Nodal signaling and are required for gastrulation.Functional analysis of mutations in TGIF associated with holoprosencephaly.The TGF-β/Smad repressor TG-interacting factor 1 (TGIF1) plays a role in radiation-induced intestinal injury independently of a Smad signaling pathwayDAZAP1, an hnRNP protein, is required for normal growth and spermatogenesis in micePremature senescence and increased TGFβ signaling in the absence of Tgif1.Tgif1 and Tgif2 Repress Expression of the RabGAP Evi5l.Genetic and Molecular Analyses indicate independent effects of TGIFs on Nodal and Gli3 in neural tube patterning.Tgif1 and Tgif2 Regulate Axial Patterning in Mouse.Loss of Tgif function causes holoprosencephaly by disrupting the SHH signaling pathway.Maternal Tgif is required for vascularization of the embryonic placentaRole of 5'TG3'-interacting factors (TGIFs) in Vorinostat (HDAC inhibitor)-mediated Corneal Fibrosis Inhibition.Stepwise reprogramming of liver cells to a pancreas progenitor state by the transcriptional regulator Tgif2.Glimpse into Hox and tale regulation of cell differentiation and reprogramming.Silencing of TGIF attenuates the tumorigenicity of A549 cells in vitro and in vivo.Transforming growth-interacting factor (TGIF) regulates proliferation and differentiation of human myeloid leukemia cells.The TG-interacting factor TGIF1 regulates stress-induced proinflammatory phenotype of endothelial cellsZebrafish model of holoprosencephaly demonstrates a key role for TGIF in regulating retinoic acid metabolism.Involvement of TG-interacting factor in microglial activation during experimental traumatic brain injury.Functions of TGIF homeodomain proteins and their roles in normal brain development and holoprosencephaly
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P2860
Embryonic fibroblasts from mice lacking Tgif were defective in cell cycling
description
2006 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հունիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2006
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im Juni 2006 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
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vedecký článok (publikovaný 2006/06/01)
@sk
vědecký článek publikovaný v roce 2006
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wetenschappelijk artikel (gepubliceerd op 2006/06/01)
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наукова стаття, опублікована в червні 2006
@uk
مقالة علمية (نشرت في يونيو 2006)
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name
Embryonic fibroblasts from mice lacking Tgif were defective in cell cycling
@ast
Embryonic fibroblasts from mice lacking Tgif were defective in cell cycling
@en
Embryonic fibroblasts from mice lacking Tgif were defective in cell cycling
@nl
type
label
Embryonic fibroblasts from mice lacking Tgif were defective in cell cycling
@ast
Embryonic fibroblasts from mice lacking Tgif were defective in cell cycling
@en
Embryonic fibroblasts from mice lacking Tgif were defective in cell cycling
@nl
prefLabel
Embryonic fibroblasts from mice lacking Tgif were defective in cell cycling
@ast
Embryonic fibroblasts from mice lacking Tgif were defective in cell cycling
@en
Embryonic fibroblasts from mice lacking Tgif were defective in cell cycling
@nl
P2860
P3181
P356
P1476
Embryonic fibroblasts from mice lacking Tgif were defective in cell cycling
@en
P2093
Pamela A. Hoodless
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P304
P3181
P356
10.1128/MCB.02156-05
P407
P577
2006-06-01T00:00:00Z