bcl-x is expressed in embryonic and postnatal neural tissues and functions to prevent neuronal cell death
about
Diva, a Bcl-2 homologue that binds directly to Apaf-1 and induces BH3-independent cell deathOverexpression of the nucleoporin CAN/NUP214 induces growth arrest, nucleocytoplasmic transport defects, and apoptosisA1 demonstrates restricted tissue distribution during embryonic development and functions to protect against cell deathThe roles of Bcl-xL in modulating apoptosis during development of Xenopus laevisDefining the role of the Bcl-2 family proteins in Huntington's diseaseAnti-apoptotic BCL-2 family proteins in acute neural injuryMtd, a novel Bcl-2 family member activates apoptosis in the absence of heterodimerization with Bcl-2 and Bcl-XLTranslocation of apoptosis-inducing factor in vulnerable neurons after transient cerebral ischemia and in neuronal cultures after oxygen-glucose deprivationSusceptibility of cerebellar granule neurons derived from Bcl-2-deficient and transgenic mice to cell deathMyocyte enhancer factor 2C as a neurogenic and antiapoptotic transcription factor in murine embryonic stem cellsAntisense inhibition of BCL-2 expression induces retinoic acid-mediated cell death during differentiation of human NT2N neurons.In vitro and in vivo enhanced generation of human A9 dopamine neurons from neural stem cells by Bcl-XLApoptosis and the nervous system.Variations in the rheostat model of apoptosis: what studies of retinal ganglion cell death tell us about the functions of the Bcl2 family proteins.Akt, a target of phosphatidylinositol 3-kinase, inhibits apoptosis in a differentiating neuronal cell lineThe BCL2L1 and PGAM5 axis defines hypoxia-induced receptor-mediated mitophagy.Valproic acid inhibits neural progenitor cell death by activation of NF-κB signaling pathway and up-regulation of Bcl-XL.Muller cell expression of glutamate cycle related proteins and anti-apoptotic proteins in early human retinal development.microRNA Expression Profiling of Propofol-Treated Developing Rat Hippocampal AstrocytesBcl-xL protects adult septal cholinergic neurons from axotomized cell death.GDNF-deprived sympathetic neurons die via a novel nonmitochondrial pathway.bcl-x exhibits regulated expression during B cell development and activation and modulates lymphocyte survival in transgenic mice.Activation of nuclear factor kappaB and Bcl-x survival gene expression by nerve growth factor requires tyrosine phosphorylation of IkappaBalpha.Zinc-dependent multi-conductance channel activity in mitochondria isolated from ischemic brainA G-tract element in apoptotic agents-induced alternative splicing.An additional form of rat Bcl-x, Bcl-xbeta, generated by an unspliced RNA, promotes apoptosis in promyeloid cells.Bcl-xL Is Essential for the Survival and Function of Differentiated Neurons in the Cortex That Control Complex Behaviors.Temporal alterations in cellular Bax:Bcl-2 ratio following traumatic brain injury in the ratBcl-2, Bcl-xL, and p-AKT are involved in neuroprotective effects of transcription factor Brn3b in an ocular hypertension rat model of glaucomaA high soy diet reduces programmed cell death and enhances bcl-xL expression in experimental stroke.Proapoptotic N-truncated BCL-xL protein activates endogenous mitochondrial channels in living synaptic terminals.A novel Bcl-x isoform connected to the T cell receptor regulates apoptosis in T cells.Axotomized neonatal motoneurons overexpressing the bcl2 proto-oncogene retain functional electrophysiological properties.ATM-dependent phosphorylation of MEF2D promotes neuronal survival after DNA damage.Expression of cellular oncogene Bcl-xL prevents coronavirus-induced cell death and converts acute infection to persistent infection in progenitor rat oligodendrocytesThe nuclear events guiding successful nerve regeneration.Increased expression of the anti-apoptotic protein Bcl-xL in the brain is associated with resilience to stress-induced depression-like behavior.Driving apoptosis-relevant proteins toward neural differentiation.Differential evolution of signal-responsive RNA elements and upstream factors that control alternative splicing.Deletion mutational analysis of BMRP, a pro-apoptotic protein that binds to Bcl-2.
P2860
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P2860
bcl-x is expressed in embryonic and postnatal neural tissues and functions to prevent neuronal cell death
description
1995 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի մայիսին հրատարակված գիտական հոդված
@hy
article publié dans les Procee ...... f the United States of America
@fr
artículu científicu espublizáu en 1995
@ast
im Mai 1995 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 1995/05/09)
@sk
vědecký článek publikovaný v roce 1995
@cs
wetenschappelijk artikel (gepubliceerd op 1995/05/09)
@nl
наукова стаття, опублікована в травні 1995
@uk
name
bcl-x is expressed in embryoni ...... to prevent neuronal cell death
@ast
bcl-x is expressed in embryoni ...... to prevent neuronal cell death
@en
bcl-x is expressed in embryoni ...... to prevent neuronal cell death
@nl
type
label
bcl-x is expressed in embryoni ...... to prevent neuronal cell death
@ast
bcl-x is expressed in embryoni ...... to prevent neuronal cell death
@en
bcl-x is expressed in embryoni ...... to prevent neuronal cell death
@nl
prefLabel
bcl-x is expressed in embryoni ...... to prevent neuronal cell death
@ast
bcl-x is expressed in embryoni ...... to prevent neuronal cell death
@en
bcl-x is expressed in embryoni ...... to prevent neuronal cell death
@nl
P2093
P2860
P356
P1476
bcl-x is expressed in embryoni ...... to prevent neuronal cell death
@en
P2093
C. B. Thompson
L. H. Boise
M. González-García
P2860
P304
P356
10.1073/PNAS.92.10.4304
P407
P577
1995-05-09T00:00:00Z