Postnatal loss of methyl-CpG binding protein 2 in the forebrain is sufficient to mediate behavioral aspects of Rett syndrome in mice
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Anaplerotic triheptanoin diet enhances mitochondrial substrate use to remodel the metabolome and improve lifespan, motor function, and sociability in MeCP2-null miceEpigenetics in the mature mammalian brain: effects on behavior and synaptic transmissionAstrocytes conspire with neurons during progression of neurological diseaseThe impact of MeCP2 loss- or gain-of-function on synaptic plasticityThe molecular basis of cognitive deficits in pervasive developmental disordersSynaptic maturation at cortical projections to the lateral amygdala in a mouse model of Rett syndromeMultiple organ system defects and transcriptional dysregulation in the Nipbl(+/-) mouse, a model of Cornelia de Lange SyndromeRett syndrome - biological pathways leading from MECP2 to disorder phenotypesAutistic-like behaviours and hyperactivity in mice lacking ProSAP1/Shank2MEF2C, a transcription factor that facilitates learning and memory by negative regulation of synapse numbers and functionComprehensive Analysis of the 16p11.2 Deletion and Null Cntnap2 Mouse Models of Autism Spectrum Disorder.Enhanced anxiety and stress-induced corticosterone release are associated with increased Crh expression in a mouse model of Rett syndromeMeCP2 deficiency results in robust Rett-like behavioural and motor deficits in male and female ratsMeCP2 controls BDNF expression and cocaine intake through homeostatic interactions with microRNA-212.Dendritic spine pathologies in hippocampal pyramidal neurons from Rett syndrome brain and after expression of Rett-associated MECP2 mutationsDysfunction in GABA signalling mediates autism-like stereotypies and Rett syndrome phenotypesBehavioural phenotyping assays for mouse models of autismMouse behavioral tasks relevant to autism: phenotypes of 10 inbred strainsMechanisms of Functional Hypoconnectivity in the Medial Prefrontal Cortex of Mecp2 Null Mice.Loss of MeCP2 in Parvalbumin-and Somatostatin-Expressing Neurons in Mice Leads to Distinct Rett Syndrome-like PhenotypesManipulations of MeCP2 in glutamatergic neurons highlight their contributions to Rett and other neurological disordersBeyond Widespread Mecp2 Deletions to Model Rett Syndrome: Conditional Spatio-Temporal Knockout, Single-Point Mutations and Transgenic Rescue MiceAnnual Research Review: Transgenic mouse models of childhood-onset psychiatric disorders.The loss of methyl-CpG binding protein 1 leads to autism-like behavioral deficits.MeCP2 and histone deacetylases 1 and 2 in dorsal striatum collectively suppress repetitive behaviorsHigh levels of MeCP2 depress MHC class I expression in neuronal cells.Synaptic circuit abnormalities of motor-frontal layer 2/3 pyramidal neurons in an RNA interference model of methyl-CpG-binding protein 2 deficiency.TLR-4-dependent and -independent mechanisms of fetal brain injury in the setting of preterm birthMagnesium sulfate reduces inflammation-associated brain injury in fetal mice.The neural circuit basis of Rett syndromeA mouse model for MeCP2 duplication syndrome: MeCP2 overexpression impairs learning and memory and synaptic transmissionRett syndrome and the impact of MeCP2 associated transcriptional mechanisms on neurotransmissionMeCP2-mediated transcription repression in the basolateral amygdala may underlie heightened anxiety in a mouse model of Rett syndromeNovel MeCP2 isoform-specific antibody reveals the endogenous MeCP2E1 expression in murine brain, primary neurons and astrocytes.Experimental models of Rett syndrome based on Mecp2 dysfunction.The role of MeCP2 in CNS development and function.Epigenetic regulation of neuronal dendrite and dendritic spine developmentComplexities of Rett syndrome and MeCP2Non-cell autonomous influence of MeCP2-deficient glia on neuronal dendritic morphologyBrain region-specific expression of MeCP2 isoforms correlates with DNA methylation within Mecp2 regulatory elements.
P2860
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P2860
Postnatal loss of methyl-CpG binding protein 2 in the forebrain is sufficient to mediate behavioral aspects of Rett syndrome in mice
description
2006 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի մարտին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2006
@ast
im März 2006 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2006/03/01)
@sk
vědecký článek publikovaný v roce 2006
@cs
wetenschappelijk artikel (gepubliceerd op 2006/03/01)
@nl
наукова стаття, опублікована в березні 2006
@uk
مقالة علمية نشرت في مارس 2006 حول موضوع: دماغ
@ar
name
Postnatal loss of methyl-CpG b ...... pects of Rett syndrome in mice
@ast
Postnatal loss of methyl-CpG b ...... pects of Rett syndrome in mice
@en
Postnatal loss of methyl-CpG b ...... pects of Rett syndrome in mice
@nl
type
label
Postnatal loss of methyl-CpG b ...... pects of Rett syndrome in mice
@ast
Postnatal loss of methyl-CpG b ...... pects of Rett syndrome in mice
@en
Postnatal loss of methyl-CpG b ...... pects of Rett syndrome in mice
@nl
prefLabel
Postnatal loss of methyl-CpG b ...... pects of Rett syndrome in mice
@ast
Postnatal loss of methyl-CpG b ...... pects of Rett syndrome in mice
@en
Postnatal loss of methyl-CpG b ...... pects of Rett syndrome in mice
@nl
P2093
P3181
P1476
Postnatal loss of methyl-CpG b ...... pects of Rett syndrome in mice
@en
P2093
Erika D. Nelson
Linda I. Perrotti
Olivier Berton
Terry Gemelli
P304
P3181
P356
10.1016/J.BIOPSYCH.2005.07.025
P407
P577
2006-03-01T00:00:00Z