Hypoxia inducible factor signaling modulates susceptibility to mycobacterial infection via a nitric oxide dependent mechanism
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The Zebrafish Breathes New Life into the Study of TuberculosisBiology of Bony Fish MacrophagesExploring the HIFs, buts and maybes of hypoxia signalling in disease: lessons from zebrafish modelsFit for consumption: zebrafish as a model for tuberculosisImmunometabolism in TuberculosisSpotlights on immunological effects of reactive nitrogen species: When inflammation says nitric oxideProtection and pathology in TB: learning from the zebrafish modelMycobacteria counteract a TLR-mediated nitrosative defense mechanism in a zebrafish infection modelHypoxia and tissue destruction in pulmonary TBInterception of host angiogenic signalling limits mycobacterial growth.Hif-1α regulates macrophage-endothelial interactions during blood vessel development in zebrafish.Real-time imaging and genetic dissection of host-microbe interactions in zebrafish.Macrophage-pathogen interactions in infectious diseases: new therapeutic insights from the zebrafish host modelAnimal models of tuberculosis: zebrafishRole of Hypoxia Inducible Factor-1α (HIF-1α) in Innate Defense against Uropathogenic Escherichia coli Infection.Neutrophils in host defense: new insights from zebrafish.Inverse Correlation between IL-10 and HIF-1α in Macrophages Infected with Histoplasma capsulatumEstablishment of Infection Models in Zebrafish Larvae (Danio rerio) to Study the Pathogenesis of Aeromonas hydrophila.The regulation of pulmonary inflammation by the hypoxia-inducible factor-hydroxylase oxygen-sensing pathway.The impact of hypoxia on bacterial infection.Involvement of ATF3 in the negative regulation of iNOS expression and NO production in activated macrophages.Hypoxia-dependent regulation of inflammatory pathways in immune cellsSiderophores in Iron Metabolism: From Mechanism to Therapy Potential.IRAK-M alters the polarity of macrophages to facilitate the survival of Mycobacterium tuberculosis.Modulation of host cell SUMOylation facilitates efficient development of Plasmodium berghei and Toxoplasma gondii.HIF-1α Is an Essential Mediator of IFN-γ-Dependent Immunity to Mycobacterium tuberculosis.Hypoxia promotes Mycobacterium tuberculosis-specific up-regulation of granulysin in human T cells.The CXCR3-CXCL11 signaling axis mediates macrophage recruitment and dissemination of mycobacterial infection.Lipid droplet formation in Mycobacterium tuberculosis infected macrophages requires IFN-γ/HIF-1α signaling and supports host defense.Priming of innate antimycobacterial immunity by heat-killed Listeria monocytogenes induces sterilizing response in the adult zebrafish tuberculosis model.Mechanisms of Fish Macrophage Antimicrobial Immunity.
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P2860
Hypoxia inducible factor signaling modulates susceptibility to mycobacterial infection via a nitric oxide dependent mechanism
description
2013 nî lūn-bûn
@nan
2013 թուականին հրատարակուած գիտական յօդուած
@hyw
2013 թվականին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Hypoxia inducible factor signa ...... tric oxide dependent mechanism
@ast
Hypoxia inducible factor signa ...... tric oxide dependent mechanism
@en
Hypoxia inducible factor signa ...... tric oxide dependent mechanism
@nl
type
label
Hypoxia inducible factor signa ...... tric oxide dependent mechanism
@ast
Hypoxia inducible factor signa ...... tric oxide dependent mechanism
@en
Hypoxia inducible factor signa ...... tric oxide dependent mechanism
@nl
prefLabel
Hypoxia inducible factor signa ...... tric oxide dependent mechanism
@ast
Hypoxia inducible factor signa ...... tric oxide dependent mechanism
@en
Hypoxia inducible factor signa ...... tric oxide dependent mechanism
@nl
P2860
P50
P3181
P1433
P1476
Hypoxia inducible factor signa ...... tric oxide dependent mechanism
@en
P2093
Fredericus J van Eeden
Sabrina Brizee
P2860
P304
P3181
P356
10.1371/JOURNAL.PPAT.1003789
P407
P577
2013-12-19T00:00:00Z