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Fibroblast growth factor signaling in skeletal development and diseaseFibroblast Growth Factor 23: A New Dimension to Diseases of Calcium-Phosphorus MetabolismExcessive Osteocytic Fgf23 Secretion Contributes to Pyrophosphate Accumulation and Mineralization Defect in Hyp MiceCharacterization of FN1-FGFR1 and novel FN1-FGF1 fusion genes in a large series of phosphaturic mesenchymal tumors.Membrane and integrative nuclear fibroblastic growth factor receptor (FGFR) regulation of FGF-23Conditional Deletion of Fgfr1 in the Proximal and Distal Tubule Identifies Distinct Roles in Phosphate and Calcium Transport.Sustained Brown Fat Stimulation and Insulin Sensitization by a Humanized Bispecific Antibody Agonist for Fibroblast Growth Factor Receptor 1/βKlotho Complex.Downregulation of PHEX in multibacillary leprosy patients: observational cross-sectional study.Deletion of Mbtps1 (Pcsk8, S1p, Ski-1) Gene in Osteocytes Stimulates Soleus Muscle Regeneration and Increased Size and Contractile Force with Age.Systemic Control of Bone Homeostasis by FGF23 Signaling.1,25-Dihydroxyvitamin D Alone Improves Skeletal Growth, Microarchitecture, and Strength in a Murine Model of XLH, Despite Enhanced FGF23 Expression.FGF signaling in the osteoprogenitor lineage non-autonomously regulates postnatal chondrocyte proliferation and skeletal growth.Searching for additional endocrine functions of the skeleton: genetic approaches and implications for therapeutics.Multiple faces of fibroblast growth factor-23Mitochondrial superoxide in osteocytes perturbs canalicular networks in the setting of age-related osteoporosis.Targeting of Mesenchymal Stromal Cells by Cre-Recombinase Transgenes Commonly Used to Target Osteoblast Lineage CellsEffects of klotho deletion from bone during chronic kidney disease.Chronic Hyperphosphatemia and Vascular Calcification Are Reduced by Stable Delivery of Soluble Klotho.Conditional Deletion of Murine Fgf23: Interruption of the Normal Skeletal Responses to Phosphate Challenge and Rescue of Genetic Hypophosphatemia.Cardiovascular Effects of Renal Distal Tubule Deletion of the FGF Receptor 1 Gene.Acute blood loss stimulates fibroblast growth factor 23 production.A computationally identified compound antagonizes excess FGF-23 signaling in renal tubules and a mouse model of hypophosphatemia.Hartsfield syndrome associated with a novel heterozygous missense mutation in FGFR1 and incorporating tumoral calcinosis.Pleiotropic Actions of FGF23.Phosphate-dependent FGF23 secretion is modulated by PiT2/Slc20a2.Physiological Actions of Fibroblast Growth Factor-23.
P2860
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P2860
description
2014 nî lūn-bûn
@nan
2014 թուականին հրատարակուած գիտական յօդուած
@hyw
2014 թվականին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
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name
Osteocyte-specific deletion of Fgfr1 suppresses FGF23
@ast
Osteocyte-specific deletion of Fgfr1 suppresses FGF23
@en
Osteocyte-specific deletion of Fgfr1 suppresses FGF23
@nl
type
label
Osteocyte-specific deletion of Fgfr1 suppresses FGF23
@ast
Osteocyte-specific deletion of Fgfr1 suppresses FGF23
@en
Osteocyte-specific deletion of Fgfr1 suppresses FGF23
@nl
prefLabel
Osteocyte-specific deletion of Fgfr1 suppresses FGF23
@ast
Osteocyte-specific deletion of Fgfr1 suppresses FGF23
@en
Osteocyte-specific deletion of Fgfr1 suppresses FGF23
@nl
P2093
P2860
P3181
P1433
P1476
Osteocyte-specific deletion of Fgfr1 suppresses FGF23
@en
P2093
Jinsong Huang
Xiaobin Han
Yingjuan Liang
P2860
P304
P3181
P356
10.1371/JOURNAL.PONE.0104154
P407
P577
2014-08-04T00:00:00Z