c-Jun NH2-terminal kinase is crucially involved in renal tubulo-interstitial inflammation
about
TGF-β signaling via TAK1 pathway: role in kidney fibrosisAcute kidney injury: a springboard for progression in chronic kidney diseaseEnteral arginine modulates inhibition of AP-1/c-Jun by SP600125 in the postischemic gut.PTEN loss defines a TGF-β-induced tubule phenotype of failed differentiation and JNK signaling during renal fibrosis.Simultaneous deletion of Bax and Bak is required to prevent apoptosis and interstitial fibrosis in obstructive nephropathyInvolvement of the NLRC4-Inflammasome in Diabetic Nephropathy.Targeted proximal tubule injury triggers interstitial fibrosis and glomerulosclerosisTGF-β-activated kinase-1: New insights into the mechanism of TGF-β signaling and kidney disease.Proximal tubule-specific overexpression of netrin-1 suppresses acute kidney injury-induced interstitial fibrosis and glomerulosclerosis through suppression of IL-6/STAT3 signalingP53 inhibitor pifithrin-α prevents the renal tubular epithelial cells against injury.Inflammation in diabetic nephropathy.Signaling pathways of PAX2 and its role in renal interstitial fibrosis and glomerulosclerosis.Mechanisms of maladaptive repair after AKI leading to accelerated kidney ageing and CKD.ASK1: a new therapeutic target for kidney disease.Dendrimer-based macromolecular conjugate for the kidney-directed delivery of a multitargeted sunitinib analogue.Baicalein attenuates renal fibrosis by inhibiting inflammation via down-regulating NF-κB and MAPK signal pathways.ASK1/p38 signaling in renal tubular epithelial cells promotes renal fibrosis in the mouse obstructed kidney.The JNK Signaling Pathway in Renal Fibrosis.Supplementing diet with Manitoba lingonberry juice reduces kidney ischemia-reperfusion injury.TLR4-mediated inflammation is a key pathogenic event leading to kidney damage and fibrosis in cyclosporine nephrotoxicity.Fibroblast growth factor 1 ameliorates diabetic nephropathy by an anti-inflammatory mechanism.Evaluation of JNK blockade as an early intervention treatment for type 1 diabetic nephropathy in hypertensive rats.TGF-β1-activated kinase-1 regulates inflammation and fibrosis in the obstructed kidney.
P2860
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P2860
c-Jun NH2-terminal kinase is crucially involved in renal tubulo-interstitial inflammation
description
2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2009
@ast
im Dezember 2009 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2009/12/01)
@sk
vědecký článek publikovaný v roce 2009
@cs
wetenschappelijk artikel (gepubliceerd op 2009/12/01)
@nl
наукова стаття, опублікована в грудні 2009
@uk
مقالة علمية (نشرت في ديسمبر 2009)
@ar
name
c-Jun NH2-terminal kinase is crucially involved in renal tubulo-interstitial inflammation
@ast
c-Jun NH2-terminal kinase is crucially involved in renal tubulo-interstitial inflammation
@en
c-Jun NH2-terminal kinase is crucially involved in renal tubulo-interstitial inflammation
@nl
type
label
c-Jun NH2-terminal kinase is crucially involved in renal tubulo-interstitial inflammation
@ast
c-Jun NH2-terminal kinase is crucially involved in renal tubulo-interstitial inflammation
@en
c-Jun NH2-terminal kinase is crucially involved in renal tubulo-interstitial inflammation
@nl
prefLabel
c-Jun NH2-terminal kinase is crucially involved in renal tubulo-interstitial inflammation
@ast
c-Jun NH2-terminal kinase is crucially involved in renal tubulo-interstitial inflammation
@en
c-Jun NH2-terminal kinase is crucially involved in renal tubulo-interstitial inflammation
@nl
P2093
P3181
P356
P1476
c-Jun NH2-terminal kinase is crucially involved in renal tubulo-interstitial inflammation
@en
P2093
Inge Hamming
Jai Prakash
Maria Sandovici
Martin H. de Borst
Pieter A. Klok
Robbert Jan Kok
P304
P3181
P356
10.1124/JPET.109.154179
P407
P577
2009-12-01T00:00:00Z