Decreased expression of voltage- and Ca(2+)-activated K(+) channels in coronary smooth muscle during aging
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Maxi K+ channel mediates regulatory volume decrease response in a human bronchial epithelial cell lineVascular endothelial dysfunction and pharmacological treatmentThe vascular conducted response in cerebral blood flow regulationFunction and regulation of large conductance Ca(2+)-activated K+ channel in vascular smooth muscle cellsVascular aging and hemodynamic stability in the intraoperative periodTestosterone and vascular function in agingEndothelial effects of emission source particles: acute toxic response gene expression profilesAging increases capacitance and spontaneous transient outward current amplitude of smooth muscle cells from murine superior epigastric arteries.Altered L-type Ca2+ channel activity contributes to exacerbated hypoperfusion and mortality in smooth muscle cell BK channel-deficient septic mice.Functional insights into modulation of BKCa channel activity to alter myometrial contractility.Membrane trafficking of large conductance calcium-activated potassium channels is regulated by alternative splicing of a transplantable, acidic trafficking motif in the RCK1-RCK2 linker.BK channels regulate sinoatrial node firing rate and cardiac pacing in vivoLarge conductance, Ca2+-activated K+ channels (BKCa) and arteriolar myogenic signalingGenomic variation associated with mortality among adults of European and African ancestry with heart failure: the cohorts for heart and aging research in genomic epidemiology consortium.Vascular BK channel deficiency exacerbates organ damage and mortality in endotoxemic mice.Distinct transcriptional regulation of human large conductance voltage- and calcium-activated K+ channel gene (hSlo1) by activated estrogen receptor alpha and c-Src tyrosine kinasePotassium channels and membrane potential in the modulation of intracellular calcium in vascular endothelial cells.An endoplasmic reticulum trafficking signal prevents surface expression of a voltage- and Ca2+-activated K+ channel splice variant.(+/-)-Naringenin as large conductance Ca(2+)-activated K+ (BKCa) channel opener in vascular smooth muscle cellsMaxiK channel partners: physiological impact.Ca2+-activated K+ channels in murine endothelial cells: block by intracellular calcium and magnesium.Ca2+ sparks as a plastic signal for skeletal muscle health, aging, and dystrophy.BK Channels in Cardiovascular Diseases and AgingEndothelial function and cardiovascular disease: effects of quercetin and wine polyphenols.Hypercontractility and impaired sildenafil relaxations in the BKCa channel deletion model of erectile dysfunctionCutaneous blood flow during intradermal NO administration in young and older adults: roles for calcium-activated potassium channels and cyclooxygenase?Calcium-activated potassium channels and endothelial dysfunction: therapeutic options?Hypertension of Kcnmb1-/- is linked to deficient K secretion and aldosteronismAging and muscle fiber type alter K+ channel contributions to the myogenic response in skeletal muscle arteriolesRole of potassium channels in coronary vasodilation.Vasodilator compounds derived from plants and their mechanisms of action.Differential distribution and functional impact of BK channel beta1 subunits across mesenteric, coronary, and different cerebral arteries of the rat.Potassium Channels in Regulation of Vascular Smooth Muscle Contraction and Growth.Smooth Muscle Ion Channels and Regulation of Vascular Tone in Resistance Arteries and Arterioles.Palmitoylation of the S0-S1 linker regulates cell surface expression of voltage- and calcium-activated potassium (BK) channels.Coexpression and characterization of the human large-conductance Ca(2+)-activated K (+) channel alpha + beta1 subunits in HEK293 cells.Recent Progress in Vascular Aging: Mechanisms and Its Role in Age-related Diseases.Ageing causes cytoplasmic retention of MaxiK channels in rat corporal smooth muscle cells.Aortic vasoreactivity during a postnatal critical window of the pancreas in rats.Estrogen replacement reduces PGHS-2-dependent vasoconstriction in the aged rat.
P2860
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P2860
Decreased expression of voltage- and Ca(2+)-activated K(+) channels in coronary smooth muscle during aging
description
2001 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2001
@ast
im Februar 2001 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2001/02/02)
@sk
vědecký článek publikovaný v roce 2001
@cs
wetenschappelijk artikel (gepubliceerd op 2001/02/02)
@nl
наукова стаття, опублікована в лютому 2001
@uk
مقالة علمية (نشرت في 2-2-2001)
@ar
name
Decreased expression of voltag ...... ary smooth muscle during aging
@ast
Decreased expression of voltag ...... ary smooth muscle during aging
@en
Decreased expression of voltag ...... ary smooth muscle during aging
@nl
type
label
Decreased expression of voltag ...... ary smooth muscle during aging
@ast
Decreased expression of voltag ...... ary smooth muscle during aging
@en
Decreased expression of voltag ...... ary smooth muscle during aging
@nl
prefLabel
Decreased expression of voltag ...... ary smooth muscle during aging
@ast
Decreased expression of voltag ...... ary smooth muscle during aging
@en
Decreased expression of voltag ...... ary smooth muscle during aging
@nl
P2093
P356
P1433
P1476
Decreased expression of voltag ...... ary smooth muscle during aging
@en
P2093
P304
P356
10.1161/01.RES.88.2.210
P577
2001-02-02T00:00:00Z