Arrhythmogenic adverse effects of cardiac glycosides are mediated by redox modification of ryanodine receptors
about
Calcium and ROS: A mutual interplayIbandronate and ventricular arrhythmia risk.Dantrolene suppresses spontaneous Ca2+ release without altering excitation-contraction coupling in cardiomyocytes of aged mice.Modulation of intracellular calcium waves and triggered activities by mitochondrial ca flux in mouse cardiomyocytes.Chasing cardiac physiology and pathology down the CaMKII cascadePrevention of postoperative atrial fibrillation: novel and safe strategy based on the modulation of the antioxidant systemTXNIP/TRX/NF-κB and MAPK/NF-κB pathways involved in the cardiotoxicity induced by Venenum Bufonis in rats.Electrochemical Na+ and Ca2+ gradients drive coupled-clock regulation of automaticity of isolated rabbit sinoatrial nodal pacemaker cells.Ryanodine receptor phosphorylation by oxidized CaMKII contributes to the cardiotoxic effects of cardiac glycosides.Redox control of cardiac excitability.Regulation of the cardiac sodium/bicarbonate cotransporter by angiotensin II: potential Contribution to structural, ionic and electrophysiological myocardial remodelling.Neuronal sodium channels: emerging components of the nano-machinery of cardiac calcium cycling.Cardiomyocyte Ca2+ homeostasis as a therapeutic target in heart failure with reduced and preserved ejection fraction.Oxidation of RyR2 Has a Biphasic Effect on the Threshold for Store Overload-Induced Calcium Release.DIDS and the Janus-faced Na⁺-K⁺-activated ATPase. Focus on "DIDS inhibits Na-K-ATPase activity in porcine nonpigmented ciliary epithelial cells by a Src family kinase-dependent mechanism".Muscarinic Stimulation Facilitates Sarcoplasmic Reticulum Ca Release by Modulating Ryanodine Receptor 2 Phosphorylation Through Protein Kinase G and Ca/Calmodulin-Dependent Protein Kinase II.Reactive oxygen species contribute to the development of arrhythmogenic Ca²⁺ waves during β-adrenergic receptor stimulation in rabbit cardiomyocytes.Na+/K+ ATPase activity promotes invasion of endocrine resistant breast cancer cells.
P2860
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P2860
Arrhythmogenic adverse effects of cardiac glycosides are mediated by redox modification of ryanodine receptors
description
2011 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2011
@ast
im Oktober 2011 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2011/10/01)
@sk
vědecký článek publikovaný v roce 2011
@cs
wetenschappelijk artikel (gepubliceerd op 2011/10/01)
@nl
наукова стаття, опублікована в жовтні 2011
@uk
مقالة علمية (نشرت في أكتوبر 2011)
@ar
name
Arrhythmogenic adverse effects ...... ication of ryanodine receptors
@ast
Arrhythmogenic adverse effects ...... ication of ryanodine receptors
@en
Arrhythmogenic adverse effects ...... ication of ryanodine receptors
@nl
type
label
Arrhythmogenic adverse effects ...... ication of ryanodine receptors
@ast
Arrhythmogenic adverse effects ...... ication of ryanodine receptors
@en
Arrhythmogenic adverse effects ...... ication of ryanodine receptors
@nl
prefLabel
Arrhythmogenic adverse effects ...... ication of ryanodine receptors
@ast
Arrhythmogenic adverse effects ...... ication of ryanodine receptors
@en
Arrhythmogenic adverse effects ...... ication of ryanodine receptors
@nl
P2093
P2860
P1476
Arrhythmogenic adverse effects ...... ication of ryanodine receptors
@en
P2093
Cynthia A. Carnes
Dmitry Terentyev
Hsiang-Ting Ho
Radmila Terentyeva
Sandor Györke
Sarah C. W. Stevens
P2860
P304
P356
10.1113/JPHYSIOL.2011.210005
P407
P577
2011-10-01T00:00:00Z