YY1 protects cardiac myocytes from pathologic hypertrophy by interacting with HDAC5
about
Histone deacetylase signaling in cardioprotectionPost-translational modifications regulate class IIa histone deacetylase (HDAC) function in health and diseaseCharacterization of human septic sera induced gene expression modulation in human myocytes.AFos inhibits phenylephrine-mediated contractile dysfunction by altering phospholamban phosphorylation.Roles and targets of class I and IIa histone deacetylases in cardiac hypertrophy.Co-occupancy by multiple cardiac transcription factors identifies transcriptional enhancers active in heart.β-Adrenergic receptor stimulation and activation of protein kinase A protect against α1-adrenergic-mediated phosphorylation of protein kinase D and histone deacetylase 5Sex differences in cardiomyocyte connexin43 expression.Temporal expression of miRNAs and mRNAs in a mouse model of myocardial infarction.Temporal analysis of mRNA and miRNA expression in transgenic mice overexpressing Arg- and Gly389 polymorphic variants of the β1-adrenergic receptor.Transgenic over-expression of YY1 induces pathologic cardiac hypertrophy in a sex-specific mannerInhibition of class I histone deacetylase activity represses matrix metalloproteinase-2 and -9 expression and preserves LV function postmyocardial infarction.Evidence for a non-canonical role of HDAC5 in regulation of the cardiac Ncx1 and Bnp genes.β-Adrenergic receptor antagonism in mice: a model for pediatric heart disease.Beta-adrenergic adaptation in paediatric idiopathic dilated cardiomyopathy.Targeting histone deacetylases for heart failure.Class IIa HDACs: from important roles in differentiation to possible implications in tumourigenesis.Function of YY1 in Long-Distance DNA Interactions.Inflammatory cytokines epigenetically regulate rheumatoid arthritis fibroblast-like synoviocyte activation by suppressing HDAC5 expressionTranscription factors in heart: promising therapeutic targets in cardiac hypertrophy.Chromatin modifications remodel cardiac gene expression.Keeping up the balance: role of HDACs in cardiac proteostasis and therapeutic implications for atrial fibrillation.Yin-Yang 1, a new player in early heart developmentYY1 Expression Is Sufficient for the Maintenance of Cardiac Progenitor Cell State.Distinct patterns of histone modifications at cardiac-specific gene promoters between cardiac stem cells and mesenchymal stem cells.Exosomes from pediatric dilated cardiomyopathy patients modulate a pathological response in cardiomyocytes.Molecular Changes in Children with Heart Failure Undergoing Left Ventricular Assist Device Therapy.Critical role of YY1 in cardiac morphogenesis.The effect of melatonin on cardio fibrosis in juvenile rats with pressure overload and deregulation of HDACs
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P2860
YY1 protects cardiac myocytes from pathologic hypertrophy by interacting with HDAC5
description
2008 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2008
@ast
im Oktober 2008 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2008/10/01)
@sk
vědecký článek publikovaný v roce 2008
@cs
wetenschappelijk artikel (gepubliceerd op 2008/10/01)
@nl
наукова стаття, опублікована в жовтні 2008
@uk
مقالة علمية (نشرت في أكتوبر 2008)
@ar
name
YY1 protects cardiac myocytes from pathologic hypertrophy by interacting with HDAC5
@ast
YY1 protects cardiac myocytes from pathologic hypertrophy by interacting with HDAC5
@en
YY1 protects cardiac myocytes from pathologic hypertrophy by interacting with HDAC5
@nl
type
label
YY1 protects cardiac myocytes from pathologic hypertrophy by interacting with HDAC5
@ast
YY1 protects cardiac myocytes from pathologic hypertrophy by interacting with HDAC5
@en
YY1 protects cardiac myocytes from pathologic hypertrophy by interacting with HDAC5
@nl
prefLabel
YY1 protects cardiac myocytes from pathologic hypertrophy by interacting with HDAC5
@ast
YY1 protects cardiac myocytes from pathologic hypertrophy by interacting with HDAC5
@en
YY1 protects cardiac myocytes from pathologic hypertrophy by interacting with HDAC5
@nl
P2093
P2860
P356
P1476
YY1 protects cardiac myocytes from pathologic hypertrophy by interacting with HDAC5
@en
P2093
Carmen C. Sucharov
Karen Dockstader
Timothy A. McKinsey
P2860
P304
P356
10.1091/MBC.E07-12-1217
P577
2008-10-01T00:00:00Z