Pharmacological interactions between calcium/calmodulin-dependent kinase II alpha and TRPV1 receptors in rat trigeminal sensory neurons
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Expanding Spectrum of Sodium Potassium Chloride Co-transporters in the Pathophysiology of DiseasesAKAP150-mediated TRPV1 sensitization is disrupted by calcium/calmodulin.CaMKIIα may modulate fentanyl-induced hyperalgesia via a CeLC-PAG-RVM-spinal cord descending facilitative pain pathway in ratsActivation of CaMKII and ERK1/2 contributes to the time-dependent potentiation of Ca2+ response elicited by repeated application of capsaicin in rat DRG neuronsComprehensive RNA-Seq expression analysis of sensory ganglia with a focus on ion channels and GPCRs in Trigeminal gangliaThe case for peripheral CB₁ receptor blockade in the treatment of visceral obesity and its cardiometabolic complications.Mice lacking functional TRPV1 are protected from pressure overload cardiac hypertrophySpinal NKCC1 blockade inhibits TRPV1-dependent referred allodyniaAn HSV-based library screen identifies PP1α as a negative TRPV1 regulator with analgesic activity in models of painNeurobiological mechanisms of pain in sickle cell disease.Intracellular signaling in primary sensory neurons and persistent pain.Acute inhibition of Ca2+/calmodulin-dependent protein kinase II reverses experimental neuropathic pain in mice.TRPV1: a stress response protein in the central nervous system.Endocannabinoids, related compounds and their metabolic routes.Inhibition of CaMKIIα in the Central Nucleus of Amygdala Attenuates Fentanyl-Induced Hyperalgesia in Rats.Neurokinin 2 receptor-mediated activation of protein kinase C modulates capsaicin responses in DRG neurons from adult rats.Combined genetic and pharmacological inhibition of TRPV1 and P2X3 attenuates colorectal hypersensitivity and afferent sensitization.CaMKIIα underlies spontaneous and evoked pain behaviors in Berkeley sickle cell transgenic mice.Presynaptic inhibition of transient receptor potential vanilloid type 1 (TRPV1) receptors by noradrenaline in nociceptive neurons.The potentiating effect of calcitonin gene-related peptide on transient receptor potential vanilloid-1 activity and the electrophysiological responses of rat trigeminal neurons to nociceptive stimuli.Phosphorylation regulates TRPV1 association with β-arrestin-2.Ca++/CaMKII switches nociceptor-sensitizing stimuli into desensitizing stimuli.Dopamine modulation of transient receptor potential vanilloid type 1 (TRPV1) receptor in dorsal root ganglia neurons.
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P2860
Pharmacological interactions between calcium/calmodulin-dependent kinase II alpha and TRPV1 receptors in rat trigeminal sensory neurons
description
2005 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2005
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im Dezember 2005 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
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vedecký článok (publikovaný 2005/12/02)
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vědecký článek publikovaný v roce 2005
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wetenschappelijk artikel (gepubliceerd op 2005/12/02)
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наукова стаття, опублікована в грудні 2005
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مقالة علمية (نشرت في 2-12-2005)
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name
Pharmacological interactions b ...... rat trigeminal sensory neurons
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Pharmacological interactions b ...... rat trigeminal sensory neurons
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Pharmacological interactions b ...... rat trigeminal sensory neurons
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type
label
Pharmacological interactions b ...... rat trigeminal sensory neurons
@ast
Pharmacological interactions b ...... rat trigeminal sensory neurons
@en
Pharmacological interactions b ...... rat trigeminal sensory neurons
@nl
prefLabel
Pharmacological interactions b ...... rat trigeminal sensory neurons
@ast
Pharmacological interactions b ...... rat trigeminal sensory neurons
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Pharmacological interactions b ...... rat trigeminal sensory neurons
@nl
P2093
P2860
P3181
P1433
P1476
Pharmacological interactions b ...... rat trigeminal sensory neurons
@en
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Christopher M. Flores
Kenneth M. Hargreaves
Nathanial A. Jeske
Theodore J. Price
P2860
P3181
P356
10.1016/J.NEULET.2005.07.029
P407
P577
2005-12-02T00:00:00Z