Inhibition of Fas-mediated apoptosis through administration of soluble Fas receptor improves functional outcome and reduces posttraumatic axonal degeneration after acute spinal cord injury
about
Deficiency in complement C1q improves histological and functional locomotor outcome after spinal cord injuryDeath receptor-ligand systems in cancer, cell death, and inflammationEphB3 receptors function as dependence receptors to mediate oligodendrocyte cell death following contusive spinal cord injuryOlprinone attenuates the acute inflammatory response and apoptosis after spinal cord trauma in miceInhibition of CXCR1 and CXCR2 chemokine receptors attenuates acute inflammation, preserves gray matter and diminishes autonomic dysreflexia after spinal cord injury.Feasibility of combination allogeneic stem cell therapy for spinal cord injury: a case report.FAS-dependent cell death in α-synuclein transgenic oligodendrocyte models of multiple system atrophyTight squeeze, slow burn: inflammation and the aetiology of cervical myelopathy.Fas/FasL-mediated apoptosis and inflammation are key features of acute human spinal cord injury: implications for translational, clinical application.Signaling by death receptors in the nervous systemModulation of inflammatory response after spinal cord trauma with deferoxamine, an iron chelator.Human neuropathological and animal model evidence supporting a role for Fas-mediated apoptosis and inflammation in cervical spondylotic myelopathy.Demyelination initiated by oligodendrocyte apoptosis through enhancing endoplasmic reticulum-mitochondria interactions and Id2 expression after compressed spinal cord injury in rats.Role of Caspase-8 and Fas in Cell Death After Spinal Cord Injury.KU0063794, a Dual mTORC1 and mTORC2 Inhibitor, Reduces Neural Tissue Damage and Locomotor Impairment After Spinal Cord Injury in Mice.Evidence for the role of PI(3) -kinase-AKT-eNOS signalling pathway in secondary inflammatory process after spinal cord compression injury in mice.Effect of Apocynin, an inhibitor of NADPH oxidase, in the inflammatory process induced by an experimental model of spinal cord injury.Emerging drugs for spinal cord injury
P2860
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P2860
Inhibition of Fas-mediated apoptosis through administration of soluble Fas receptor improves functional outcome and reduces posttraumatic axonal degeneration after acute spinal cord injury
description
2006 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի մայիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2006
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im Mai 2006 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
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vedecký článok (publikovaný 2006/05/01)
@sk
vědecký článek publikovaný v roce 2006
@cs
wetenschappelijk artikel (gepubliceerd op 2006/05/01)
@nl
наукова стаття, опублікована в травні 2006
@uk
مقالة علمية (نشرت في مايو 2006)
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name
Inhibition of Fas-mediated apo ...... after acute spinal cord injury
@ast
Inhibition of Fas-mediated apo ...... after acute spinal cord injury
@en
Inhibition of Fas-mediated apo ...... after acute spinal cord injury
@nl
type
label
Inhibition of Fas-mediated apo ...... after acute spinal cord injury
@ast
Inhibition of Fas-mediated apo ...... after acute spinal cord injury
@en
Inhibition of Fas-mediated apo ...... after acute spinal cord injury
@nl
prefLabel
Inhibition of Fas-mediated apo ...... after acute spinal cord injury
@ast
Inhibition of Fas-mediated apo ...... after acute spinal cord injury
@en
Inhibition of Fas-mediated apo ...... after acute spinal cord injury
@nl
P356
P1476
Inhibition of Fas-mediated apo ...... after acute spinal cord injury
@en
P2093
Alun Ackery
Sherri Robins
P304
P356
10.1089/NEU.2006.23.604
P577
2006-05-01T00:00:00Z