Mitochondrial signals initiate the activation of c-Jun N-terminal kinase (JNK) by hypoxia-reoxygenation
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Vaccinia-related kinase 2 modulates the stress response to hypoxia mediated by TAK1Mitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale.Hypothyroid phenotype is contributed by mitochondrial complex I inactivation due to translocated neuronal nitric-oxide synthaseA respiratory chain controlled signal transduction cascade in the mitochondrial intermembrane space mediates hydrogen peroxide signalingTumor cell phenotype is sustained by selective MAPK oxidation in mitochondria.Age-related molecular genetic changes of murine bone marrow mesenchymal stem cells.RAGE modulates hypoxia/reoxygenation injury in adult murine cardiomyocytes via JNK and GSK-3beta signaling pathways.Hypothermia in bleeding trauma: a friend or a foe?Crosstalk between mitogen-activated protein kinases and mitochondria in cardiac diseases: therapeutic perspectivesCell signalling by oxidized lipids and the role of reactive oxygen species in the endotheliumInhibition of JNK aggravates the recovery of rat hearts after global ischemia: the role of mitochondrial JNK.Effect of hypoxic stress-activated Polo-like kinase 3 on corneal epithelial wound healingMitochondrial c-Jun N-terminal kinase (JNK) signaling initiates physiological changes resulting in amplification of reactive oxygen species generation.Oscillatory shear stress induces mitochondrial superoxide production: implication of NADPH oxidase and c-Jun NH2-terminal kinase signalingRegulation of VEGF-induced endothelial cell migration by mitochondrial reactive oxygen speciesDynamic differences in oxidative stress and the regulation of metabolism with age in visceral versus subcutaneous adiposeMitochondrial membrane permeabilization and cell death during myocardial infarction: roles of calcium and reactive oxygen species.Inhibition of JNK mitochondrial localization and signaling is protective against ischemia/reperfusion injury in rats.Beyond anoxia: the physiology of metabolic downregulation and recovery in the anoxia-tolerant turtle.Metabolic reprogramming in dietary restriction.Energy sensing and regulation of gene expression in skeletal muscle.Ischemia-reperfusion and cardioprotection: a delicate balance between reactive oxygen species generation and redox homeostasis.Activation of Polo-like kinase 3 by hypoxic stresses.Lung alveolar integrity is compromised by telomere shortening in telomerase-null mice.Protein kinases in organ ischemia and reperfusion.Mitochondrial kinases in Parkinson's disease: converging insights from neurotoxin and genetic modelsNitrite as a mediator of ischemic preconditioning and cytoprotection.Chronic consumption of Annona muricata juice triggers and aggravates cerebral tau phosphorylation in wild-type and MAPT transgenic mice.Knockdown of augmenter of liver regeneration in HK-2 cells inhibits inflammation response via the mitogen-activated protein kinase signaling pathway.The novel indole compound SK228 induces apoptosis and FAK/Paxillin disruption in tumor cell lines and inhibits growth of tumor graft in the nude mouse.Gene expression profiling of chronic myeloid leukemia with variant t(9;22) reveals a different signature from cases with classic translocation.Effect of Hypoxia-regulated Polo-like Kinase 3 (Plk3) on Human Limbal Stem Cell Differentiation.Human adipose tissue-derived stem cells protect impaired cardiomyocytes from hypoxia/reoxygenation injury through hypoxia-induced paracrine mechanism.Cardioprotection by farnesol: role of the mevalonate pathway.Baicalein protects cardiomyocytes against mitochondrial oxidant injury associated with JNK inhibition and mitochondrial Akt activation.A peptide inhibitor of c-Jun NH2-terminal kinase reduces myocardial ischemia-reperfusion injury and infarct size in vivo.Conflicting evidence for the role of JNK as a target in breast cancer cell proliferation: Comparisons between pharmacological inhibition and selective shRNA knockdown approaches.Differential contribution of mitochondria, NADPH oxidases, and glycolysis to region-specific oxidant stress in the anoxic-reoxygenated embryonic heart.
P2860
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P2860
Mitochondrial signals initiate the activation of c-Jun N-terminal kinase (JNK) by hypoxia-reoxygenation
description
2004 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2004
@ast
im Juli 2004 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2004/07/01)
@sk
vědecký článek publikovaný v roce 2004
@cs
wetenschappelijk artikel (gepubliceerd op 2004/07/01)
@nl
наукова стаття, опублікована в липні 2004
@uk
مقالة علمية (نشرت في يوليو 2004)
@ar
name
Mitochondrial signals initiate ...... (JNK) by hypoxia-reoxygenation
@ast
Mitochondrial signals initiate ...... (JNK) by hypoxia-reoxygenation
@en
Mitochondrial signals initiate ...... (JNK) by hypoxia-reoxygenation
@nl
type
label
Mitochondrial signals initiate ...... (JNK) by hypoxia-reoxygenation
@ast
Mitochondrial signals initiate ...... (JNK) by hypoxia-reoxygenation
@en
Mitochondrial signals initiate ...... (JNK) by hypoxia-reoxygenation
@nl
prefLabel
Mitochondrial signals initiate ...... (JNK) by hypoxia-reoxygenation
@ast
Mitochondrial signals initiate ...... (JNK) by hypoxia-reoxygenation
@en
Mitochondrial signals initiate ...... (JNK) by hypoxia-reoxygenation
@nl
P2093
P356
P1433
P1476
Mitochondrial signals initiate ...... (JNK) by hypoxia-reoxygenation
@en
P2093
Christopher J. Dougherty
Donna P. Frazier
Huifang Li
Keith A. Webster
Lori A. Kubasiak
Nanette H. Bishopric
Wen-Cheng Xiong
P304
P356
10.1096/FJ.04-1505COM
P407
P577
2004-07-01T00:00:00Z