Ozone exposure of macrophages induces an alveolar epithelial chemokine response through IL-1alpha
about
Genes of innate immunity and the biological response to inhaled ozoneThe alarmin IL-1α is a master cytokine in acute lung inflammation induced by silica micro- and nanoparticlesLinezolid has unique immunomodulatory effects in post-influenza community acquired MRSA pneumoniaLasers, stem cells, and COPD.Antipollution skin protection - a new paradigm and its demonstration on two active compounds.TLR4 is necessary for hyaluronan-mediated airway hyperresponsiveness after ozone inhalation.Apoptosis induced by ozone and oxysterols in human alveolar epithelial cells.Normal CFTR inhibits epidermal growth factor receptor-dependent pro-inflammatory chemokine production in human airway epithelial cells.Interaction with epithelial cells modifies airway macrophage response to ozoneSalmonella flagellins are potent adjuvants for intranasally administered whole inactivated influenza vaccine.Pulmonary inflammation triggered by ricin toxin requires macrophages and IL-1 signalingThe IL-1 axis is associated with airway inflammation after O3 exposure in allergic asthmatic patients.Effects of Exposure to Ozone on the Ocular Surface in an Experimental Model of Allergic Conjunctivitis.Ozone exposed epithelial cells modify cocultured natural killer cellsAmbient ozone and pulmonary innate immunity.The Interleukin-1α Precursor is Biologically Active and is Likely a Key Alarmin in the IL-1 Family of Cytokines.Host-pathogen interactions during coronavirus infection of primary alveolar epithelial cells.Update in pediatric lung disease 2009.Alarmins: Feel the Stress.Exposure to ozone induces a systemic inflammatory response: possible source of the neurological alterations induced by this gas.Promotion of cardiovascular disease by exposure to the air pollutant ozone.Interleukin-1α Mediates Ozone-Induced Myeloid Differentiation Factor-88-Dependent Epithelial Tissue Injury and Inflammation.Functional and morphological differences of the lung upon acute and chronic ozone exposure in mice.TLR9 and IL-1R1 Promote Mobilization of Pulmonary Dendritic Cells during Beryllium SensitizationCardiovascular and inflammatory mechanisms in healthy humans exposed to air pollution in the vicinity of a steel mill
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P2860
Ozone exposure of macrophages induces an alveolar epithelial chemokine response through IL-1alpha
description
2008 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի մարտին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2008
@ast
im März 2008 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2008/03/01)
@sk
vědecký článek publikovaný v roce 2008
@cs
wetenschappelijk artikel (gepubliceerd op 2008/03/01)
@nl
наукова стаття, опублікована в березні 2008
@uk
مقالة علمية (نشرت في مارس 2008)
@ar
name
Ozone exposure of macrophages ...... ine response through IL-1alpha
@ast
Ozone exposure of macrophages ...... ine response through IL-1alpha
@en
Ozone exposure of macrophages ...... ine response through IL-1alpha
@nl
type
label
Ozone exposure of macrophages ...... ine response through IL-1alpha
@ast
Ozone exposure of macrophages ...... ine response through IL-1alpha
@en
Ozone exposure of macrophages ...... ine response through IL-1alpha
@nl
prefLabel
Ozone exposure of macrophages ...... ine response through IL-1alpha
@ast
Ozone exposure of macrophages ...... ine response through IL-1alpha
@en
Ozone exposure of macrophages ...... ine response through IL-1alpha
@nl
P2093
P2860
P356
P1476
Ozone exposure of macrophages ...... ine response through IL-1alpha
@en
P2093
Glen McConville
Rizwan Manzer
Robert J. Mason
P2860
P304
P356
10.1165/RCMB.2007-0250OC
P577
2008-03-01T00:00:00Z