Dissociation and dispersion of claudin-3 from the tight junction could be one of the most sensitive indicators of reflux esophagitis in a rat model of the disease
about
Role of tight junction proteins in gastroesophageal reflux diseaseGastrointestinal mucosal barrier function and diseases.Lipopolysaccharide disrupts the milk-blood barrier by modulating claudins in mammary alveolar tight junctions.Mast cell activation is involved in stress-induced epithelial barrier dysfunction in the esophagus.Apical acidity decreases inhibitory effect of omeprazole on Mg(2+) absorption and claudin-7 and -12 expression in Caco-2 monolayers.Biomarkers of Reflux Disease.Esophageal intraluminal baseline impedance is associated with severity of acid reflux and epithelial structural abnormalities in patients with gastroesophageal reflux diseaseGastroesophageal reflux activates the NF-κB pathway and impairs esophageal barrier function in mice.The degradation of airway tight junction protein under acidic conditions is probably mediated by transient receptor potential vanilloid 1 receptor.A randomized prospective study comparing the efficacy of on-demand therapy versus continuous therapy for 6 months for long-term maintenance with omeprazole 20 mg in patients with gastroesophageal reflux disease in Japan.Contribution of immunomodulators to gastroesophageal reflux disease and its complications: stromal cells, interleukin 4, and adiponectin.Esophageal baseline impedance levels in patients with pathophysiological characteristics of functional heartburn.Epithelial-derived nuclear IL-33 aggravates inflammation in the pathogenesis of reflux esophagitis.In vivo evaluation of acid-induced changes in oesophageal mucosa integrity and sensitivity in non-erosive reflux disease.Additive Effects of Rebamipide Plus Proton Pump Inhibitors on the Expression of Tight Junction Proteins in a Rat Model of Gastro-Esophageal Reflux Disease.Estrogen Enhances Esophageal Barrier Function by Potentiating Occludin Expression.Effect of rikkunshito on the expression of substance P and CGRP in dorsal root ganglion neurons and voluntary movement in rats with experimental reflux esophagitis.Comparison of Tight Junction Protein-Related Gene mRNA Expression Levels between Male and Female Gastroesophageal Reflux Disease Patients.Bile salts disrupt human esophageal squamous epithelial barrier function by modulating tight junction proteins.Increased Immunoreactivity of Cathepsins in the Rat Esophagus under Chronic Acid Reflux Esophagitis
P2860
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P2860
Dissociation and dispersion of claudin-3 from the tight junction could be one of the most sensitive indicators of reflux esophagitis in a rat model of the disease
description
2011 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մայիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2011
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scientific journal article
@en
vedecký článok (publikovaný 2011/05/01)
@sk
vědecký článek publikovaný v roce 2011
@cs
wetenschappelijk artikel (gepubliceerd op 2011/05/01)
@nl
наукова стаття, опублікована в травні 2011
@uk
مقالة علمية (نشرت في مايو 2011)
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name
Dissociation and dispersion of ...... in a rat model of the disease
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Dissociation and dispersion of ...... in a rat model of the disease
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Dissociation and dispersion of ...... in a rat model of the disease
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type
label
Dissociation and dispersion of ...... in a rat model of the disease
@ast
Dissociation and dispersion of ...... in a rat model of the disease
@en
Dissociation and dispersion of ...... in a rat model of the disease
@nl
prefLabel
Dissociation and dispersion of ...... in a rat model of the disease
@ast
Dissociation and dispersion of ...... in a rat model of the disease
@en
Dissociation and dispersion of ...... in a rat model of the disease
@nl
P2093
P2860
P1476
Dissociation and dispersion of ...... in a rat model of the disease
@en
P2093
Akihito Nagahara
Daisuke Asaoka
Hiroki Mori
Masako Oguro
Masato Koike
Sumio Watanabe
Takashi Ueno
Yasuo Uchiyama
P2860
P304
P356
10.1007/S00535-011-0390-1
P577
2011-05-01T00:00:00Z