Loss of Usp9x disrupts cortical architecture, hippocampal development and TGFβ-mediated axonogenesis
about
Mutations in USP9X are associated with X-linked intellectual disability and disrupt neuronal cell migration and growthNeuronal migration and its disorders affecting the CA3 regionLa FAM fatale: USP9X in development and diseaseSeizures are regulated by ubiquitin-specific peptidase 9 X-linked (USP9X), a de-ubiquitinaseUSP9X deubiquitylating enzyme maintains RAPTOR protein levels, mTORC1 signalling and proliferation in neural progenitors.Ubiquitin-specific proteases as therapeutic targets for the treatment of breast cancerCombined inhibition of Bcl-2/Bcl-xL and Usp9X/Bag3 overcomes apoptotic resistance in glioblastoma in vitro and in vivo.Sequential Elution Interactome Analysis of the Mind Bomb 1 Ubiquitin Ligase Reveals a Novel Role in Dendritic Spine Outgrowth.Role of Angiomotin-like 2 mono-ubiquitination on YAP inhibition.De Novo Loss-of-Function Mutations in USP9X Cause a Female-Specific Recognizable Syndrome with Developmental Delay and Congenital MalformationsDeubiquitylating enzyme USP9x regulates radiosensitivity in glioblastoma cells by Mcl-1-dependent and -independent mechanisms.Usp9x-deficiency disrupts the morphological development of the postnatal hippocampal dentate gyrus.The emerging roles of deubiquitylating enzymes (DUBs) in the TGFβ and BMP pathways.Loss of Usp9x disrupts cell adhesion, and components of the Wnt and Notch signaling pathways in neural progenitorsNeuroanatomy of pain-deficiency and cross-modal activation in calcium channel subunit (CACN) α2δ3 knockout mice.Regulation of pluripotency and differentiation by deubiquitinating enzymes.Targeting Deubiquitinating Enzymes in Glioblastoma Multiforme: Expectations and Challenges.Organization of the connections between claustrum and cortex in the mouse.USP9X deletion elevates the density of oligodendrocytes within the postnatal dentate gyrus.Homozygous mutation of STXBP5L explains an autosomal recessive infantile-onset neurodegenerative disorder.Grb2-Mediated Recruitment of USP9X to LAT Enhances Themis Stability following Thymic Selection.Deubiquitinating Enzyme USP9X Suppresses Tumor Growth via LATS Kinase and Core Components of the Hippo Pathway.Stabilization of the methyl-CpG binding protein ZBTB38 by the deubiquitinase USP9X limits the occurrence and toxicity of oxidative stress in human cells.A Fat-Facets-Dscam1-JNK Pathway Enhances Axonal Growth in Development and after Injury.
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Loss of Usp9x disrupts cortical architecture, hippocampal development and TGFβ-mediated axonogenesis
description
2013 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի հունվարին հրատարակված գիտական հոդված
@hy
article publié dans la revue scientifique PLoS ONE
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artículu científicu espublizáu en 2013
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im Januar 2013 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
@en
vedecký článok (publikovaný 2013/01/01)
@sk
vědecký článek publikovaný v roce 2013
@cs
wetenschappelijk artikel (gepubliceerd op 2013/01/01)
@nl
наукова стаття, опублікована в січні 2013
@uk
name
Loss of Usp9x disrupts cortica ...... and TGFβ-mediated axonogenesis
@ast
Loss of Usp9x disrupts cortica ...... and TGFβ-mediated axonogenesis
@en
Loss of Usp9x disrupts cortica ...... and TGFβ-mediated axonogenesis
@nl
type
label
Loss of Usp9x disrupts cortica ...... and TGFβ-mediated axonogenesis
@ast
Loss of Usp9x disrupts cortica ...... and TGFβ-mediated axonogenesis
@en
Loss of Usp9x disrupts cortica ...... and TGFβ-mediated axonogenesis
@nl
prefLabel
Loss of Usp9x disrupts cortica ...... and TGFβ-mediated axonogenesis
@ast
Loss of Usp9x disrupts cortica ...... and TGFβ-mediated axonogenesis
@en
Loss of Usp9x disrupts cortica ...... and TGFβ-mediated axonogenesis
@nl
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Loss of Usp9x disrupts cortica ...... and TGFβ-mediated axonogenesis
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Shane Stegeman
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P3181
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10.1371/JOURNAL.PONE.0068287
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2013-07-05T00:00:00Z