Male sterility in mice lacking retinoic acid receptor alpha involves specific abnormalities in spermiogenesis
about
Activity of retinoic acid receptor-alpha is directly regulated at its protein kinase A sites in response to follicle-stimulating hormone signalingOral administration of a retinoic Acid receptor antagonist reversibly inhibits spermatogenesis in miceAs the world grows: contraception in the 21st centuryMammalian E-type cyclins control chromosome pairing, telomere stability and CDK2 localization in male meiosisSpermiation: The process of sperm releaseCHD5 is required for spermiogenesis and chromatin condensationThe Role of Retinoic Acid (RA) in Spermatogonial DifferentiationProlonged Oral Administration of a Pan-Retinoic Acid Receptor Antagonist Inhibits Spermatogenesis in Mice With a Rapid Recovery and Changes in the Expression of Influx and Efflux Transporters.Seipin deficiency increases chromocenter fragmentation and disrupts acrosome formation leading to male infertility.Spermatogenesis: The Commitment to Meiosis.New approaches to male non-hormonal contraceptionRetinoid signaling during spermatogenesis as revealed by genetic and metabolic manipulations of retinoic acid receptor alpha.Pharmacological activity of retinoic acid receptor alpha-selective antagonists in vitro and in vivo.Expression of retinoic acid receptor alpha in the germline is essential for proper cellular association and spermiogenesis during spermatogenesis.Temporal profiling of rat transcriptomes in retinol-replenished vitamin A-deficient testis.Differentiation of spermatogonial stem cell-like cells from murine testicular tissue into haploid male germ cells in vitro.Aberrant distribution of junctional complex components in retinoic acid receptor alpha-deficient mice.The death of sertoli cells and the capacity to phagocytize elongated spermatids during testicular regression due to short photoperiod in Syrian hamster (Mesocricetus auratus).Periodic production of retinoic acid by meiotic and somatic cells coordinates four transitions in mouse spermatogenesis.BRDT is an essential epigenetic regulator for proper chromatin organization, silencing of sex chromosomes and crossover formation in male meiosis.The future of male contraception: a fertile ground.Retinoic Acid Receptor Signaling in Post-Natal Male Germ Cell Differentiation
P2860
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P2860
Male sterility in mice lacking retinoic acid receptor alpha involves specific abnormalities in spermiogenesis
description
2005 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2005
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im April 2005 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
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vedecký článok (publikovaný 2005/04/01)
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vědecký článek publikovaný v roce 2005
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wetenschappelijk artikel (gepubliceerd op 2005/04/01)
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наукова стаття, опублікована у квітні 2005
@uk
مقالة علمية (نشرت في أبريل 2005)
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name
Male sterility in mice lacking ...... bnormalities in spermiogenesis
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Male sterility in mice lacking ...... bnormalities in spermiogenesis
@en
Male sterility in mice lacking ...... bnormalities in spermiogenesis
@nl
type
label
Male sterility in mice lacking ...... bnormalities in spermiogenesis
@ast
Male sterility in mice lacking ...... bnormalities in spermiogenesis
@en
Male sterility in mice lacking ...... bnormalities in spermiogenesis
@nl
prefLabel
Male sterility in mice lacking ...... bnormalities in spermiogenesis
@ast
Male sterility in mice lacking ...... bnormalities in spermiogenesis
@en
Male sterility in mice lacking ...... bnormalities in spermiogenesis
@nl
P2093
P2860
P1433
P1476
Male sterility in mice lacking ...... bnormalities in spermiogenesis
@en
P2093
Debra J. Wolgemuth
Sanny S. W. Chung
Xiangyuan Wang
P2860
P304
P356
10.1111/J.1432-0436.2005.00018.X
P577
2005-04-01T00:00:00Z