Gli3-deficient mice exhibit cleft palate associated with abnormal tongue development
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Molecular basis of cleft palates in miceFuz mutant mice reveal shared mechanisms between ciliopathies and FGF-related syndromesPrdm16 is required for normal palatogenesis in mice.Tmem26 is dynamically expressed during palate and limb development but is not required for embryonic survival.Gsk3β is required in the epithelium for palatal elevation in mice.A Shh-Foxf-Fgf18-Shh Molecular Circuit Regulating Palate Development.Identification and functional characterization of novel transcriptional enhancers involved in regulating human GLI3 expression during early development.Disruption of the ERK/MAPK pathway in neural crest cells as a potential cause of Pierre Robin sequence.Molecular signaling along the anterior-posterior axis of early palate development.Mice with Tak1 deficiency in neural crest lineage exhibit cleft palate associated with abnormal tongue development.Type 1 fibroblast growth factor receptor in cranial neural crest cell-derived mesenchyme is required for palatogenesis.Disrupting hedgehog and WNT signaling interactions promotes cleft lip pathogenesis.Roles of retinoic acid signaling in normal and abnormal development of the palate and tongue.Ablation of the Sox11 Gene Results in Clefting of the Secondary Palate Resembling the Pierre Robin Sequence.Tbx1 is necessary for palatal elongation and elevationAssociation between polymorphisms at the GREM1 locus and the risk of nonsyndromic cleft lip with or without cleft palate in the Polish population.Loss-of-Function of Gli3 in Mice Causes Abnormal Frontal Bone Morphology and Premature Synostosis of the Interfrontal Suture.The association study of nonsyndromic cleft lip with or without cleft palate identified risk variants of the GLI3 gene in a Chinese population.The regulation of endogenous retinoic acid level through CYP26B1 is required for elevation of palatal shelves.Association Between Genes Involved in Craniofacial Development and Nonsyndromic Cleft Lip and/or Palate in the Brazilian Population.Cleft Palate and Aglossia Result From Perturbations in Wnt and Hedgehog Signaling.
P2860
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P2860
Gli3-deficient mice exhibit cleft palate associated with abnormal tongue development
description
2008 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2008
@ast
im Oktober 2008 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2008/10/01)
@sk
vědecký článek publikovaný v roce 2008
@cs
wetenschappelijk artikel (gepubliceerd op 2008/10/01)
@nl
наукова стаття, опублікована в жовтні 2008
@uk
مقالة علمية (نشرت في أكتوبر 2008)
@ar
name
Gli3-deficient mice exhibit cleft palate associated with abnormal tongue development
@ast
Gli3-deficient mice exhibit cleft palate associated with abnormal tongue development
@en
Gli3-deficient mice exhibit cleft palate associated with abnormal tongue development
@nl
type
label
Gli3-deficient mice exhibit cleft palate associated with abnormal tongue development
@ast
Gli3-deficient mice exhibit cleft palate associated with abnormal tongue development
@en
Gli3-deficient mice exhibit cleft palate associated with abnormal tongue development
@nl
prefLabel
Gli3-deficient mice exhibit cleft palate associated with abnormal tongue development
@ast
Gli3-deficient mice exhibit cleft palate associated with abnormal tongue development
@en
Gli3-deficient mice exhibit cleft palate associated with abnormal tongue development
@nl
P2093
P356
P1476
Gli3-deficient mice exhibit cleft palate associated with abnormal tongue development
@en
P2093
Chin Chiang
Steven L. Goudy
Tatiana Ketova
Ying Litingtung
P304
P356
10.1002/DVDY.21714
P577
2008-10-01T00:00:00Z