The role of GABAB receptor activation in absence seizures of lethargic (lh/lh) mice
about
Coding and noncoding variation of the human calcium-channel beta4-subunit gene CACNB4 in patients with idiopathic generalized epilepsy and episodic ataxia.Human gamma-aminobutyric acid type B receptors are differentially expressed and regulate inwardly rectifying K+ channelsA brief history on the oscillating roles of thalamus and cortex in absence seizuresIncreased excitability and inward rectification in layer V cortical pyramidal neurons in the epileptic mutant mouse StargazerExcitatory but not inhibitory synaptic transmission is reduced in lethargic (Cacnb4(lh)) and tottering (Cacna1atg) mouse thalamiMutations of calcium channel beta subunit genes in miceThe ß subunit of voltage-gated Ca2+ channelsAbsence seizures in C3H/HeJ and knockout mice caused by mutation of the AMPA receptor subunit Gria4Endogenous positive allosteric modulation of GABA(A) receptors by diazepam binding inhibitorReduction of thalamic and cortical Ih by deletion of TRIP8b produces a mouse model of human absence epilepsy.Ion channels and the genetic contribution to epilepsy.Eliminating Absence Seizures through the Deep Brain Stimulation to Thalamus Reticular Nucleus.The histone fold: a ubiquitous architectural motif utilized in DNA compaction and protein dimerization.Association analysis of exonic variants of the gene encoding the GABAB receptor and idiopathic generalized epilepsy.Genetic epilepsy model derived from common inbred mouse strains.An integrative view of mechanisms underlying generalized spike-and-wave epileptic seizures and its implication on optimal therapeutic treatmentsInteractions between membrane conductances underlying thalamocortical slow-wave oscillationsGeneralized epileptic disorders: an update.Paired-pulse depression of the N-methyl-D-aspartate receptor-mediated synaptic potentials in the amygdala.GABAB receptors in maintenance of neocortical circuit function.Bidirectional control of absence seizures by the basal ganglia: a computational evidence.Multimodal, multidimensional models of mouse brainDisinhibition-induced transitions between absence and tonic-clonic epileptic seizuresVoltage-gated calcium channels and idiopathic generalized epilepsies.Defective gamma-aminobutyric acid type B receptor-activated inwardly rectifying K+ currents in cerebellar granule cells isolated from weaver and Girk2 null mutant mice.Neurochemical and behavioral features in genetic absence epilepsy and in acutely induced absence seizures.Choosing the correct antiepileptic drugs: from animal studies to the clinic.GABA(B) autoreceptor-mediated cell type-specific reduction of inhibition in epileptic mice.Maternal epileptic seizure induced by pentylenetetrazol: apoptotic neurodegeneration and decreased GABAB1 receptor expression in prenatal rat brain.Relationship between aberrant brain connectivity and clinical features in Angelman Syndrome: a new method using tract based spatial statistics of DTI color-coded orientation maps.Stimulus-induced Epileptic Spike-Wave Discharges in Thalamocortical Model with Disinhibition.Targeting Neural Synchrony Deficits is Sufficient to Improve Cognition in a Schizophrenia-Related Neurodevelopmental Model.T-type Ca2+ channels in normal and abnormal brain functions.Cav3 T-type channels: regulators for gating, membrane expression, and cation selectivity.T-type Ca2+ channels in absence epilepsy.Alternative splicing in the C-terminal tail of Cav2.1 is essential for preventing a neurological disease in mice.Microbial Proteins as Novel Industrial Biotechnology Hosts to Treat Epilepsy.Emerging insights into mechanisms of epilepsy: implications for new antiepileptic drug development.Conditionally-immortalized astrocytic cell line expresses GAD and secretes GABA under tetracycline regulation.Identification and preclinical testing of novel antiepileptic compounds.
P2860
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P2860
The role of GABAB receptor activation in absence seizures of lethargic (lh/lh) mice
description
1992 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
1992 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
article publié dans la revue scientifique Science
@fr
artículu científicu espublizáu en 1992
@ast
im Juli 1992 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 1992/07/17)
@sk
vědecký článek publikovaný v roce 1992
@cs
wetenschappelijk artikel (gepubliceerd op 1992/07/17)
@nl
наукова стаття, опублікована в липні 1992
@uk
name
The role of GABAB receptor activation in absence seizures of lethargic (lh/lh) mice
@ast
The role of GABAB receptor activation in absence seizures of lethargic (lh/lh) mice
@en
The role of GABAB receptor activation in absence seizures of lethargic (lh/lh) mice
@nl
type
label
The role of GABAB receptor activation in absence seizures of lethargic (lh/lh) mice
@ast
The role of GABAB receptor activation in absence seizures of lethargic (lh/lh) mice
@en
The role of GABAB receptor activation in absence seizures of lethargic (lh/lh) mice
@nl
prefLabel
The role of GABAB receptor activation in absence seizures of lethargic (lh/lh) mice
@ast
The role of GABAB receptor activation in absence seizures of lethargic (lh/lh) mice
@en
The role of GABAB receptor activation in absence seizures of lethargic (lh/lh) mice
@nl
P2093
P356
P1433
P1476
The role of GABAB receptor activation in absence seizures of lethargic (lh/lh) mice
@en
P2093
D. A. Hosford
R. A. Morrisett
W. A. Wilson
P304
P356
10.1126/SCIENCE.1321503
P407
P577
1992-07-17T00:00:00Z