A homozygous nonsense mutation in the gene for Tmem79, a component for the lamellar granule secretory system, produces spontaneous eczema in an experimental model of atopic dermatitis
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Molecular Mechanisms of Cutaneous Inflammatory Disorder: Atopic DermatitisMechanisms of abnormal lamellar body secretion and the dysfunctional skin barrier in patients with atopic dermatitisDeciphering the complexities of atopic dermatitis: shifting paradigms in treatment approachesEpidermal permeability barrier defects and barrier repair therapy in atopic dermatitisGenetic and epigenetic studies of atopic dermatitisDefects of corneocyte structural proteins and epidermal barrier in atopic dermatitisEpidermal barrier defects link atopic dermatitis with altered skin cancer susceptibilityThe Role of Skin Barrier in the Pathogenesis of Food AllergySkin Diseases in Laboratory Mice: Approaches to Drug Target Identification and Efficacy Screening.Lipid abnormalities and lipid-based repair strategies in atopic dermatitis.Epidermal barriersDissecting the formation, structure and barrier function of the stratum corneum.The Genetics and Epigenetics of Atopic Dermatitis-Filaggrin and Other Polymorphisms.Bacterial and Viral Infections in Atopic Dermatitis: a Comprehensive Review.The interplay between genetic and environmental factors in the pathogenesis of atopic dermatitis.IL-17 Receptor A Maintains and Protects the Skin Barrier To Prevent Allergic Skin Inflammation.IL-17A induces heterogeneous macrophages, and it does not alter the effects of lipopolysaccharides on macrophage activation in the skin of mice.Increased Th2 activity and diminished skin barrier function cooperate in allergic skin inflammation.High TMEM45A expression is correlated to epidermal keratinization.The etiopathogenesis of atopic dermatitis: barrier disruption, immunological derangement, and pruritus.Acidification of stratum corneum prevents the progression from atopic dermatitis to respiratory allergy.Atopic Eczema: Genetic Associations and Potential Links to Developmental Exposures.Translational Animal Models of Atopic Dermatitis for Preclinical Studies.Explant cultures of atopic dermatitis biopsies maintain their epidermal characteristics in vitro.Epidermal filaggrin deficiency mediates increased systemic T-helper 17 immune response
P2860
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P2860
A homozygous nonsense mutation in the gene for Tmem79, a component for the lamellar granule secretory system, produces spontaneous eczema in an experimental model of atopic dermatitis
description
2013 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2013
@ast
im November 2013 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2013/11/01)
@sk
vědecký článek publikovaný v roce 2013
@cs
wetenschappelijk artikel (gepubliceerd op 2013/11/01)
@nl
наукова стаття, опублікована в листопаді 2013
@uk
مقالة علمية (نشرت في نوفمبر 2013)
@ar
name
A homozygous nonsense mutation ...... tal model of atopic dermatitis
@ast
A homozygous nonsense mutation ...... tal model of atopic dermatitis
@en
A homozygous nonsense mutation ...... tal model of atopic dermatitis
@nl
type
label
A homozygous nonsense mutation ...... tal model of atopic dermatitis
@ast
A homozygous nonsense mutation ...... tal model of atopic dermatitis
@en
A homozygous nonsense mutation ...... tal model of atopic dermatitis
@nl
prefLabel
A homozygous nonsense mutation ...... tal model of atopic dermatitis
@ast
A homozygous nonsense mutation ...... tal model of atopic dermatitis
@en
A homozygous nonsense mutation ...... tal model of atopic dermatitis
@nl
P2093
P50
P3181
P1476
A homozygous nonsense mutation ...... tal model of atopic dermatitis
@en
P2093
Aiko Shiohama
Atsushi Shimizu
Hiroshi Kawasaki
Takashi Sasaki
Takayuki Hachiya
Taketo Yamada
P304
1111–1120.e4
P3181
P356
10.1016/J.JACI.2013.08.027
P407
P577
2013-11-01T00:00:00Z