The Mos/MAP kinase pathway stabilizes c-Fos by phosphorylation and augments its transforming activity in NIH 3T3 cells
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Down-regulation of c-Fos/c-Jun AP-1 dimer activity by sumoylation.A role for the immediate early gene product c-fos in imprinting T cells with short-term memory for signal summationNeoplastic transformation of rat thyroid cells requires the junB and fra-1 gene induction which is dependent on the HMGI-C gene productERK and p38 MAPK-activated protein kinases: a family of protein kinases with diverse biological functionsRegulation of sprouty stability by Mnk1-dependent phosphorylation.Resilience to audiogenic seizures is associated with p-ERK1/2 dephosphorylation in the subiculum of Fmr1 knockout mice.Multipolar mitosis of tetraploid cells: inhibition by p53 and dependency on Mos.Tissue-specific target analysis of disease-associated microRNAs in human signaling pathways.Heterodimerization with different Jun proteins controls c-Fos intranuclear dynamics and distributionEpidermal Growth Factor Receptor Cell Proliferation Signaling PathwaysIntracellular signaling by the chemokine receptor US28 during human cytomegalovirus infectionEssential role of RSK2 in c-Fos-dependent osteosarcoma development.An extracellular signal-regulated kinase 1- and 2-dependent program of chromatin trafficking of c-Fos and Fra-1 is required for cyclin D1 expression during cell cycle reentry.Ischemic preconditioning affects long-term cell fate through DNA damage-related molecular signaling and altered proliferation.Computational approaches for modeling regulatory cellular networks.Quantitative analysis of ERK2 interactions with substrate proteins: roles for kinase docking domains and activity in determining binding affinity.A network of immediate early gene products propagates subtle differences in mitogen-activated protein kinase signal amplitude and duration.Cytokeratin19 induced by HER2/ERK binds and stabilizes HER2 on cell membranes.Phosphorylation of the carboxyl-terminal transactivation domain of c-Fos by extracellular signal-regulated kinase mediates the transcriptional activation of AP-1 and cellular transformation induced by platelet-derived growth factor.Signaling in cell differentiation and morphogenesisLaguerre Filter Analysis with Partial Least Square Regression Reveals a Priming Effect of ERK and CREB on c-FOS Induction.Role of Erk1/2 signaling in the regulation of neutrophil versus monocyte development in response to G-CSF and M-CSF.Mineralocorticoid receptors are present in skeletal muscle and represent a potential therapeutic target.Keratins turn over by ubiquitination in a phosphorylation-modulated fashionControl of FLIP(L) expression and TRAIL resistance by the extracellular signal-regulated kinase1/2 pathway in breast epithelial cells.Crystallization and preliminary X-ray diffraction analysis of rat protein tyrosine phosphatase etac-Fos Protects Neurons Through a Noncanonical Mechanism Involving HDAC3 Interaction: Identification of a 21-Amino Acid Fragment with Neuroprotective Activity.c-Fos proto-oncoprotein is degraded by the proteasome independently of its own ubiquitinylation in vivo.The extracellular signal-regulated kinase pathway phosphorylates AML1, an acute myeloid leukemia gene product, and potentially regulates its transactivation ability.Mitogen-activated protein kinase activation is not necessary for, but antagonizes, 3T3-L1 adipocytic differentiation.Mitogen-activated protein kinase contributes to lipopolysaccharide-induced activation of corticotropin-releasing hormone synthesizing neurons in the hypothalamic paraventricular nucleus.Melanoma cell adhesion molecule stimulates yes-associated protein transcription by enhancing CREB activity via c-Jun/c-Fos in hepatocellular carcinoma cellsTumor-derived variants of Epstein-Barr virus latent membrane protein 1 induce sustained Erk activation and c-FosRIPK1 and RIPK3 Kinases Promote Cell-Death-Independent Inflammation by Toll-like Receptor 4.M-CSF elevates c-Fos and phospho-C/EBPalpha(S21) via ERK whereas G-CSF stimulates SHP2 phosphorylation in marrow progenitors to contribute to myeloid lineage specification.Lead effects on development and function of bone marrow-derived dendritic cells promote Th2 immune responses.Temporal production of the signaling lipid phosphatidic acid by phospholipase D2 determines the output of extracellular signal-regulated kinase signaling in cancer cellsRat protein tyrosine phosphatase eta suppresses the neoplastic phenotype of retrovirally transformed thyroid cells through the stabilization of p27(Kip1).The C-terminal domain of c-fos is required for activation of an AP-1 site specific for jun-fos heterodimers.Ras-GAP binding and phosphorylation by herpes simplex virus type 2 RR1 PK (ICP10) and activation of the Ras/MEK/MAPK mitogenic pathway are required for timely onset of virus growth.
P2860
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P2860
The Mos/MAP kinase pathway stabilizes c-Fos by phosphorylation and augments its transforming activity in NIH 3T3 cells
description
1995 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 1995
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im Oktober 1995 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
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vedecký článok (publikovaný 1995/10/16)
@sk
vědecký článek publikovaný v roce 1995
@cs
wetenschappelijk artikel (gepubliceerd op 1995/10/16)
@nl
наукова стаття, опублікована в жовтні 1995
@uk
مقالة علمية (نشرت في 16-10-1995)
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name
The Mos/MAP kinase pathway sta ...... ming activity in NIH 3T3 cells
@ast
The Mos/MAP kinase pathway sta ...... ming activity in NIH 3T3 cells
@en
The Mos/MAP kinase pathway sta ...... ming activity in NIH 3T3 cells
@nl
type
label
The Mos/MAP kinase pathway sta ...... ming activity in NIH 3T3 cells
@ast
The Mos/MAP kinase pathway sta ...... ming activity in NIH 3T3 cells
@en
The Mos/MAP kinase pathway sta ...... ming activity in NIH 3T3 cells
@nl
prefLabel
The Mos/MAP kinase pathway sta ...... ming activity in NIH 3T3 cells
@ast
The Mos/MAP kinase pathway sta ...... ming activity in NIH 3T3 cells
@en
The Mos/MAP kinase pathway sta ...... ming activity in NIH 3T3 cells
@nl
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P3181
P1433
P1476
The Mos/MAP kinase pathway sta ...... ming activity in NIH 3T3 cells
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P2093
P2860
P304
P3181
P407
P577
1995-10-16T00:00:00Z