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Molecular origin of cancer: catechol estrogen-3,4-quinones as endogenous tumor initiators

Molecular origin of cancer: catechol estrogen-3,4-quinones as endogenous tumor initiators

http://www.wikidata.org/entity/Q28645872

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Enzyme induction and cytotoxicity in human hepatocytes by chlorpyrifos and N,N-diethyl-m-toluamide (DEET).Major differences exist in the function and tissue-specific expression of human aflatoxin B1 aldehyde reductase and the principal human aldo-keto reductase AKR1 family members The extrathyronine actions of iodine as antioxidant, apoptotic, and differentiation factor in various tissues Endogenous hormones and the aetiology of breast cancer.Understanding the human health effects of chemical mixtures.Perspectives on the chemical etiology of breast cancer.Estradiol uptake, toxicity, metabolism, and adverse effects on cadmium-treated amphibian embryos.Genetic polymorphisms in human SULT1A1 and UGT1A1 genes associate with breast tumor characteristics: a case-series study.Linking Pesticide Exposure with Pediatric Leukemia: Potential Underlying Mechanisms Depurinating estrogen-DNA adducts, generators of cancer initiation: their minimization leads to cancer prevention Hormones in Dairy Foods and Their Impact on Public Health - A Narrative Review Article Environmental exposure to xenoestrogens and oestrogen related cancers: reproductive system, breast, lung, kidney, pancreas, and brain The molecular etiology and prevention of estrogen-initiated cancers: Ockham's Razor: Pluralitas non est ponenda sine necessitate. Plurality should not be posited without necessity Continuing to illuminate the mechanisms underlying UV-mediated melanomagenesis Crystal structure of human sex hormone-binding globulin in complex with 2-methoxyestradiol reveals the molecular basis for high affinity interactions with C-2 derivatives of estradiol Binding Modes of Aromatic Ligands to Mammalian Heme Peroxidases with Associated Functional Implications: CRYSTAL STRUCTURES OF LACTOPEROXIDASE COMPLEXES WITH ACETYLSALICYLIC ACID, SALICYLHYDROXAMIC ACID, AND BENZYLHYDROXAMIC ACID Mode of Binding of the Tuberculosis Prodrug Isoniazid to Heme Peroxidases: BINDING STUDIES AND CRYSTAL STRUCTURE OF BOVINE LACTOPEROXIDASE WITH ISONIAZID AT 2.7 A RESOLUTION Aflatoxin B1 aldehyde reductase (AFAR) genes cluster at 1p35-1p36.1 in a region frequently altered in human tumour cells High-resolution chromosome 3p allelotyping of breast carcinomas and precursor lesions demonstrates frequent loss of heterozygosity and a discontinuous pattern of allele loss Catechol-O-Methyltransferase (COMT) gene polymorphism and breast cancer risk in young women Causes of genome instability: the effect of low dose chemical exposures in modern society Urinary biomarkers suggest that estrogen-DNA adducts may play a role in the aetiology of non-Hodgkin lymphoma Metformin represses self-renewal of the human breast carcinoma stem cells via inhibition of estrogen receptor-mediated OCT4 expression Raloxifene and desmethylarzoxifene block estrogen-induced malignant transformation of human breast epithelial cells Urinary estrogen metabolites and self-reported infertility in women infected with Schistosoma haematobium Differential regulation of hepatic cytochrome P450 monooxygenases in streptozotocin-induced diabetic rats Depurinating naphthalene-DNA adducts in mouse skin related to cancer initiation The Role of Human Aldo-Keto Reductases in the Metabolic Activation and Detoxication of Polycyclic Aromatic Hydrocarbons: Interconversion of PAH Catechols and PAH o-Quinones Detoxication of benzo[a]pyrene-7,8-dione by sulfotransferases (SULTs) in human lung cells The resveratrol analogue, 2,3',4,5'-tetramethoxystilbene, does not inhibit CYP gene expression, enzyme activity and benzo[a]pyrene-DNA adduct formation in MCF-7 cells exposed to benzo[a]pyrene Estrogen metabolism Susceptibility to human cancer: From the perspective of a pathologist.Breast-related effects of selective estrogen receptor modulators and tissue-selective estrogen complexes Proton linkage for CO binding and redox properties of bovine lactoperoxidase.Ratiometric analysis of the ferrocene boronate esters of 2- and 4-hydroxyestradiol by tandem electrospray mass spectrometry.Balance of beneficial and deleterious health effects of quinones: a case study of the chemical properties of genistein and estrone quinones.Selective estrogen receptor modulator delivery of quinone warheads to DNA triggering apoptosis in breast cancer cells.Mechanism of metabolic activation and DNA adduct formation by the human carcinogen diethylstilbestrol: the defining link to natural estrogens.Benzene and dopamine catechol quinones could initiate cancer or neurogenic disease Tolcapone induces oxidative stress leading to apoptosis and inhibition of tumor growth in Neuroblastoma.

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Molecular origin of cancer: catechol estrogen-3,4-quinones as endogenous tumor initiators

http://www.wikidata.org/entity/Q28645872

description

1997 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

1997 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

article publié dans les Procee ...... f the United States of America

@fr

artículu científicu espublizáu en 1997

@ast

im September 1997 veröffentlichter wissenschaftlicher Artikel

@de

scientific journal article

@en

vedecký článok (publikovaný 1997/09/30)

@sk

vědecký článek publikovaný v roce 1997

@cs

wetenschappelijk artikel (gepubliceerd op 1997/09/30)

@nl

наукова стаття, опублікована у вересні 1997

@uk

name

Molecular origin of cancer: catechol estrogen-3,4-quinones as endogenous tumor initiators

@ast

Molecular origin of cancer: catechol estrogen-3,4-quinones as endogenous tumor initiators

@en

Molecular origin of cancer: catechol estrogen-3,4-quinones as endogenous tumor initiators

@nl

type

label

Molecular origin of cancer: catechol estrogen-3,4-quinones as endogenous tumor initiators

@ast

Molecular origin of cancer: catechol estrogen-3,4-quinones as endogenous tumor initiators

@en

Molecular origin of cancer: catechol estrogen-3,4-quinones as endogenous tumor initiators

@nl

prefLabel

Molecular origin of cancer: catechol estrogen-3,4-quinones as endogenous tumor initiators

@ast

Molecular origin of cancer: catechol estrogen-3,4-quinones as endogenous tumor initiators

@en

Molecular origin of cancer: catechol estrogen-3,4-quinones as endogenous tumor initiators

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PNAS.94.20.10937

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Proceedings of the National Academy of Sciences of the United States of America

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Molecular origin of cancer: catechol estrogen-3,4-quinones as endogenous tumor initiators

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D. E. Stack

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E. G. Rogan

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E. L. Cavalieri

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M. L. Gross

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R. Ramanathan

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Rat CYP1B1: an adrenal cytochrome P450 that exhibits sex-dependent expression in livers and kidneys of TCDD-treated animals

Relating aromatic hydrocarbon-induced DNA adducts and c-H-ras mutations in mouse skin papillomas: the role of apurinic sites

4-Hydroxylation of estradiol by human uterine myometrium and myoma microsomes: implications for the mechanism of uterine tumorigenesis.

Cytochrome P-450-mediated redox cycling of estrogens.

Adenocarcinoma of the vagina. Association of maternal stilbestrol therapy with tumor appearance in young women.

Hormones and mammary carcinogenesis in mice, rats, and humans: a unifying hypothesis.

The approach to understanding aromatic hydrocarbon carcinogenesis. The central role of radical cations in metabolic activation.

Models of DNA structure achieve almost perfect discrimination between normal prostate, benign prostatic hyperplasia (BPH), and adenocarcinoma and have a high potential for predicting BPH and prostate cancer.

Mouse cytochrome P-450EF, representative of a new 1B subfamily of cytochrome P-450s. Cloning, sequence determination, and tissue expression.

17 beta-estradiol hydroxylation catalyzed by human cytochrome P450 1B1.

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