Particulate matter exposure exacerbates high glucose-induced cardiomyocyte dysfunction through ROS generation
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Redox Characterization of Functioning Skeletal MuscleMolecular Regulation of Toll-like Receptors in Asthma and COPDFine particulate matter in acute exacerbation of COPDRedox Mechanism of Reactive Oxygen Species in ExerciseRedox Roles of Reactive Oxygen Species in Cardiovascular DiseasesComparison of High-Protein, Intermittent Fasting Low-Calorie Diet and Heart Healthy Diet for Vascular Health of the ObeseHuman Peritoneal Mesothelial Cell Death Induced by High-Glucose Hypertonic Solution Involves Ca2+ and Na+ Ions and Oxidative Stress with the Participation of PKC/NOX2 and PI3K/Akt PathwaysProtein kinase RNA-like endoplasmic reticulum kinase (PERK) signaling pathway plays a major role in reactive oxygen species (ROS)-mediated endoplasmic reticulum stress-induced apoptosis in diabetic cardiomyopathy.Direct and indirect effects of particulate matter on the cardiovascular systemIn vitro particulate matter exposure causes direct and lung-mediated indirect effects on cardiomyocyte function.Glucagon-like peptide-1 protects against cardiac microvascular injury in diabetes via a cAMP/PKA/Rho-dependent mechanism.The influence of Nrf2 on cardiac responses to environmental stressors.Low Po₂ conditions induce reactive oxygen species formation during contractions in single skeletal muscle fibersCharacterization of reactive oxygen species in diaphragm.The mechanisms of air pollution and particulate matter in cardiovascular diseases.Paeonol Protects Rat Heart by Improving Regional Blood Perfusion during No-ReflowAir Pollution and Other Environmental Modulators of Cardiac Function.Recombinant human deoxyribonuclease attenuates oxidative stress in a model of eosinophilic pulmonary response in mice.Characterization of oxygen radical formation mechanism at early cardiac ischemia.Simvastatin promotes cardiac microvascular endothelial cells proliferation, migration and survival by phosphorylation of p70 S6K and FoxO3a.Selenium attenuates high glucose-induced ROS/TLR-4 involved apoptosis of rat cardiomyocyte.Po2 cycling protects diaphragm function during reoxygenation via ROS, Akt, ERK, and mitochondrial channels.Klotho Improves Cardiac Function by Suppressing Reactive Oxygen Species (ROS) Mediated Apoptosis by Modulating Mapks/Nrf2 Signaling in Doxorubicin-Induced Cardiotoxicity.Tiron ameliorates high glucose-induced cardiac myocyte apoptosis by PKCδ-dependent inhibition of osteopontin.In utero exposure to diesel exhaust particulates is associated with an altered cardiac transcriptional response to transverse aortic constriction and altered DNA methylation.Mangiferin Enhanced Autophagy via Inhibiting mTORC1 Pathway to Prevent High Glucose-Induced Cardiomyocyte Injury.Ambient fine particulate matter exposure induces reversible cardiac dysfunction and fibrosis in juvenile and older female mice.
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P2860
Particulate matter exposure exacerbates high glucose-induced cardiomyocyte dysfunction through ROS generation
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2011 թուականին հրատարակուած գիտական յօդուած
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2011 թվականին հրատարակված գիտական հոդված
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2011年の論文
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2011年論文
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2011年論文
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2011年論文
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2011年論文
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2011年論文
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2011年论文
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Particulate matter exposure ex ...... unction through ROS generation
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Particulate matter exposure ex ...... unction through ROS generation
@en
Particulate matter exposure ex ...... unction through ROS generation
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type
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Particulate matter exposure ex ...... unction through ROS generation
@ast
Particulate matter exposure ex ...... unction through ROS generation
@en
Particulate matter exposure ex ...... unction through ROS generation
@nl
prefLabel
Particulate matter exposure ex ...... unction through ROS generation
@ast
Particulate matter exposure ex ...... unction through ROS generation
@en
Particulate matter exposure ex ...... unction through ROS generation
@nl
P2093
P2860
P3181
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P1476
Particulate matter exposure ex ...... unction through ROS generation
@en
P2093
Dane J Youtz
Loren E Wold
P2860
P304
P3181
P356
10.1371/JOURNAL.PONE.0023116
P407
P577
2011-01-01T00:00:00Z