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Human Ventricular Unloading Induces Cardiomyocyte Proliferation

Human Ventricular Unloading Induces Cardiomyocyte Proliferation

http://www.wikidata.org/entity/Q29300198

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Pharmacological Therapy in the Heart as an Alternative to Cellular Therapy: A Place for the Brain Natriuretic Peptide?MicroRNAs regulating meis1 expression and inducing cardiomyocyte proliferation Cardiac resident macrophages are involved in hypoxia‑induced postnatal cardiomyocyte proliferation MicroRNA-1825 induces proliferation of adult cardiomyocytes and promotes cardiac regeneration post ischemic injury.Age-Dependent Oxidative DNA Damage Does Not Correlate with Reduced Proliferation of Cardiomyocytes in Humans.Regulation of cell proliferation by hypoxia-inducible factors Enhanced engraftment, proliferation, and therapeutic potential in heart using optimized human iPSC-derived cardiomyocytes "Young at heart": Regenerative potential linked to immature cardiac phenotypes.Heart regeneration and repair after myocardial infarction: translational opportunities for novel therapeutics.Myocardial recovery with mechanical circulatory support.Biomaterial-Enhanced Cell and Drug Delivery: Lessons Learned in the Cardiac Field and Future Perspectives.Innate heart regeneration: endogenous cellular sources and exogenous therapeutic amplification.Acute exercise activates p38 MAPK and increases the expression of telomere-protective genes in cardiac muscle.Redirecting cardiac growth mechanisms for therapeutic regeneration.Predicting cell viability within tissue scaffolds under equiaxial strain: multi-scale finite element model of collagen-cardiomyocytes constructs.Cardiac regeneration: All work and no repair?Molecular mechanisms of cardiovascular benefits of exercise: Running for cover from heart disease.Cardiomyocyte Regeneration: A Consensus Statement.Stem Cells in Regenerative Cardiology.Dedifferentiation, Proliferation, and Redifferentiation of Adult Mammalian Cardiomyocytes After Ischemic Injury.Evolution, comparative biology and ontogeny of vertebrate heart regeneration.New Myocyte Formation in the Adult Heart: Endogenous Sources and Therapeutic Implications Cardiomyocyte gene programs encoding morphological and functional signatures in cardiac hypertrophy and failure Therapeutic approaches for cardiac regeneration and repair

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Human Ventricular Unloading Induces Cardiomyocyte Proliferation

http://www.wikidata.org/entity/Q29300198

description

2015 nî lūn-bûn

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2015 թուականի Մարտին հրատարակուած գիտական յօդուած

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2015 թվականի մարտին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

Human Ventricular Unloading Induces Cardiomyocyte Proliferation

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Human Ventricular Unloading Induces Cardiomyocyte Proliferation

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Human Ventricular Unloading Induces Cardiomyocyte Proliferation

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Human Ventricular Unloading Induces Cardiomyocyte Proliferation

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Human Ventricular Unloading Induces Cardiomyocyte Proliferation

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Human Ventricular Unloading Induces Cardiomyocyte Proliferation

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J.JACC.2014.12.027

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Journal of the American College of Cardiology

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Human ventricular unloading induces cardiomyocyte proliferation

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Aroumougame Asaithamby

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Diana C Canseco

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Hesham A Sadek

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Pradeep P A Mammen

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Salim Abdisalaam

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Sandeep Das

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Shibani Mukherjee

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Sonia Garg

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Wataru Kimura

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P2860

Evidence for cardiomyocyte renewal in humans

The DNA-damage response in human biology and disease

Aurora-B regulates the cleavage furrow-specific vimentin phosphorylation in the cytokinetic process

Use of a continuous-flow device in patients awaiting heart transplantation

Neuregulin1/ErbB4 signaling induces cardiomyocyte proliferation and repair of heart injury

mir-17-92 cluster is required for and sufficient to induce cardiomyocyte proliferation in postnatal and adult hearts

Assessment of myocardial viability and left ventricular function in patients supported by a left ventricular assist device

The oxygen-rich postnatal environment induces cardiomyocyte cell-cycle arrest through DNA damage response

A novel strategy to maximize the efficacy of left ventricular assist devices as a bridge to recovery.

Meis1 regulates postnatal cardiomyocyte cell cycle arrest.

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892-900

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10.1016/J.JACC.2014.12.027

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9

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Souparno Bhattacharya

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2015-01-21T00:00:00Z

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