Effects of CD2-associated protein deficiency on amyloid-β in neuroblastoma cells and in an APP transgenic mouse model. - Wikidata - wikimedia - TriplyDB

Effects of CD2-associated protein deficiency on amyloid-β in neuroblastoma cells and in an APP transgenic mouse model.

Effects of CD2-associated protein deficiency on amyloid-β in neuroblastoma cells and in an APP transgenic mouse model.

http://www.wikidata.org/entity/Q30009187

about

Genes associated with Alzheimer's disease: an overview and current status The Alzheimer's disease risk factor CD2AP maintains blood-brain barrier integrity.Genetic Influences on Plasma Homocysteine Levels in African Americans and Yoruba Nigerians The Adaptor Protein CD2AP Is a Coordinator of Neurotrophin Signaling-Mediated Axon Arbor Plasticity.The effect of the top 20 Alzheimer disease risk genes on gray-matter density and FDG PET brain metabolism.Clearance of cerebral Aβ in Alzheimer's disease: reassessing the role of microglia and monocytes.Bin1 and CD2AP polarise the endocytic generation of beta-amyloid.Targeting of nonlipidated, aggregated apoE with antibodies inhibits amyloid accumulation.

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P2860

Effects of CD2-associated protein deficiency on amyloid-β in neuroblastoma cells and in an APP transgenic mouse model.

http://www.wikidata.org/entity/Q30009187

description

2015 nî lūn-bûn

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2015 թուականի Մարտին հրատարակուած գիտական յօդուած

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2015 թվականի մարտին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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Molecular Neurodegeneration

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Effects of CD2-associated prot ...... an APP transgenic mouse model.

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Andrey S Shaw

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Hong Jiang

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Jin-Moo Lee

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Katheryn B Lefton

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Qingli Xiao

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Subhashini Srivatsan

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P2860

Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease

Cloning of a gene bearing missense mutations in early-onset familial Alzheimer's disease

Decreased CALM expression reduces Aβ42 to total Aβ ratio through clathrin-mediated endocytosis of γ-secretase

The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

Mutant presenilins specifically elevate the levels of the 42 residue beta-amyloid peptide in vivo: evidence for augmentation of a 42-specific gamma secretase

CD2-associated protein haploinsufficiency is linked to glomerular disease susceptibility

Evidence that production and release of amyloid beta-protein involves the endocytic pathway

Trafficking and proteolytic processing of APP

A novel adaptor protein orchestrates receptor patterning and cytoskeletal polarity in T-cell contacts

Candidate gene for the chromosome 1 familial Alzheimer's disease locus

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