Genetic differences between adenocarcinomas arising in Barrett's esophagus and gastric mucosa.
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UbcH10 overexpression may represent a marker of anaplastic thyroid carcinomasDARPP-32: from neurotransmission to cancerEarly events during neoplastic progression in Barrett's esophagusSignaling pathways in the molecular pathogenesis of adenocarcinomas of the esophagus and gastroesophageal junctionDNA hypermethylation regulates the expression of members of the Mu-class glutathione S-transferases and glutathione peroxidases in Barrett's adenocarcinoma.Expression of tight-junction protein claudin-7 is an early event in gastric tumorigenesisExpression of calcium-binding proteins S100A2 and S100A4 in Barrett's adenocarcinomasGenome-wide gene copy number and expression analysis of primary gastric tumors and gastric cancer cell linesSingle nucleotide polymorphism-based genome-wide chromosome copy change, loss of heterozygosity, and aneuploidy in Barrett's esophagus neoplastic progression.Regions of allelic imbalance in the distal portion of chromosome 12q in gastric cancerEsophageal adenocarcinoma: treatment modalities in the era of targeted therapy.Epigenetic and genetic silencing of CHFR in esophageal adenocarcinomas.Genetic Insights in Barrett's Esophagus and Esophageal Adenocarcinoma.Differences in the histological findings, phenotypic marker expressions and genetic alterations between adenocarcinoma of the gastric cardia and distal stomach.The aurora kinase A regulates GSK-3beta in gastric cancer cellsLinkage and related analyses of Barrett's esophagus and its associated adenocarcinomas.Chromosomal instability and copy number alterations in Barrett's esophagus and esophageal adenocarcinoma.DNA copy number changes in young gastric cancer patients with special reference to chromosome 19.Integrated molecular analysis reveals complex interactions between genomic and epigenomic alterations in esophageal adenocarcinomas.CHFR: a key checkpoint component implicated in a wide range of cancers.Angiogenesis-related agents in esophageal cancer.Chromosomal gains and genomic loss of p53 and p16 genes in Barrett's esophagus detected by fluorescence in situ hybridization of cytology specimens.An individual coding polymorphism and the haplotype of the SPARC gene predict gastric cancer recurrence.Different roles for p16(INK) (4a) -Rb pathway and INK4a/ARF methylation between adenocarcinomas of gastric cardia and distal stomach.
P2860
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P2860
Genetic differences between adenocarcinomas arising in Barrett's esophagus and gastric mucosa.
description
2001 nî lūn-bûn
@nan
2001 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Genetic differences between ad ...... esophagus and gastric mucosa.
@ast
Genetic differences between ad ...... esophagus and gastric mucosa.
@en
type
label
Genetic differences between ad ...... esophagus and gastric mucosa.
@ast
Genetic differences between ad ...... esophagus and gastric mucosa.
@en
prefLabel
Genetic differences between ad ...... esophagus and gastric mucosa.
@ast
Genetic differences between ad ...... esophagus and gastric mucosa.
@en
P2093
P356
P1433
P1476
Genetic differences between ad ...... esophagus and gastric mucosa.
@en
P2093
Bissonette EA
El-Rifai W
Frierson HF Jr
Knuutila S
Moskaluk CA
Petroni GR
P304
P356
10.1053/GAST.2001.27215
P407
P577
2001-09-01T00:00:00Z