Carbon black and titanium dioxide nanoparticles elicit distinct apoptotic pathways in bronchial epithelial cells. - Wikidata - wikimedia - TriplyDB

Carbon black and titanium dioxide nanoparticles elicit distinct apoptotic pathways in bronchial epithelial cells.

Carbon black and titanium dioxide nanoparticles elicit distinct apoptotic pathways in bronchial epithelial cells.

http://www.wikidata.org/entity/Q30396943

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Dispersion method for safety research on manufactured nanomaterials Toxicity evaluations of nanoclays and thermally degraded byproducts through spectroscopical and microscopical approaches Health and cellular impacts of air pollutants: from cytoprotection to cytotoxicity Carbon black and titanium dioxide nanoparticles induce distinct molecular mechanisms of toxicity Pulmonary exposure to carbon black nanoparticles and vascular effects Nanoparticles: Is Toxicity a Concern?Cytotoxicity and oxidative stress induced by different metallic nanoparticles on human kidney cells Causes of genome instability: the effect of low dose chemical exposures in modern society Inflammasome activation in airway epithelial cells after multi-walled carbon nanotube exposure mediates a profibrotic response in lung fibroblasts Autophagy and lysosomal dysfunction as emerging mechanisms of nanomaterial toxicity Intracellular signal modulation by nanomaterials Cellular targets and mechanisms in the cytotoxic action of non-biodegradable engineered nanoparticles Intratracheally administered titanium dioxide or carbon black nanoparticles do not aggravate elastase-induced pulmonary emphysema in rats Nanomaterials toxicity and cell death modalities Systematic Review of Screening and Surveillance Programs to Protect Workers from Nanomaterials Oxidative Stress and Nano-Toxicity Induced by TiO2 and ZnO on WAG Cell Line Radical-containing ultrafine particulate matter initiates epithelial-to-mesenchymal transitions in airway epithelial cells Autophagy as a Possible Underlying Mechanism of Nanomaterial Toxicity Decreased Uptake and Enhanced Mitochondrial Protection Underlie Reduced Toxicity of Nanoceria in Human Monocyte-Derived Macrophages The Toxicological Mechanisms of Environmental Soot (Black Carbon) and Carbon Black: Focus on Oxidative Stress and Inflammatory Pathways Silver wire amplifies the signaling mechanism for IL-1beta production more than silver submicroparticles in human monocytic THP-1 cells Induction of inflammasome-dependent pyroptosis by carbon black nanoparticles.Distinct biological effects of different nanoparticles commonly used in cosmetics and medicine coatings.Carbon black nanoparticles promote endothelial activation and lipid accumulation in macrophages independently of intracellular ROS production.Effect of carbon black nanomaterial on biological membranes revealed by shape of human erythrocytes, platelets and phospholipid vesicles.A mixture of anatase and rutile TiO₂ nanoparticles induces histamine secretion in mast cells Cerium dioxide nanoparticles induce apoptosis and autophagy in human peripheral blood monocytes Carbon nanoparticles induce ceramide- and lipid raft-dependent signalling in lung epithelial cells: a target for a preventive strategy against environmentally-induced lung inflammation Health hazards associated with nanomaterials.Towards an alternative testing strategy for nanomaterials used in nanomedicine: lessons from NanoTEST.Genome wide identification and expression profile in epithelial cells exposed to TiO₂ particles.The potential health challenges of TiO2 nanomaterials.Nanomedicine and nanotoxicology: the pros and cons for neurodegeneration and brain cancer.Nanomaterial-induced cell death in pulmonary and hepatic cells following exposure to three different metallic materials: The role of autophagy and apoptosis.Ozonized carbon black induces mitochondrial dysfunction and DNA damage.The effects of nanoparticles on the renal system.Induction of eosinophil apoptosis by hydrogen peroxide promotes the resolution of allergic inflammation Effects of titanium dioxide nanoparticles isolated from confectionery products on the metabolic stress pathway in human lung fibroblast cells.Cytotoxicity, DNA damage, and apoptosis induced by titanium dioxide nanoparticles in human non-small cell lung cancer A549 cells.Amorphous silica nanoparticles trigger nitric oxide/peroxynitrite imbalance in human endothelial cells: inflammatory and cytotoxic effects.

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P2860

Carbon black and titanium dioxide nanoparticles elicit distinct apoptotic pathways in bronchial epithelial cells.

http://www.wikidata.org/entity/Q30396943

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2010 nî lūn-bûn

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2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2010 թվականի ապրիլին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Carbon black and titanium diox ...... in bronchial epithelial cells.

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Carbon black and titanium diox ...... in bronchial epithelial cells.

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Particle and Fibre Toxicology

P1476

Carbon black and titanium diox ...... in bronchial epithelial cells.

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P2093

Armelle Baeza-Squiban

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Francelyne Marano

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Ioana Ferecatu

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Jocelyne Fleury

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Leen C J Thomassen

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Marie-Caroline Borot

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Salik Hussain

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Sonja Boland

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P2860

Engineered nanoparticle respiratory exposure and potential risks for cardiovascular toxicity: predictive tests and biomarkers

Apoptosis: a basic biological phenomenon with wide-ranging implications in tissue kinetics

Mechanisms of caspase activation and inhibition during apoptosis.

Toxic potential of materials at the nanolevel

Caspases: enemies within

The BCL-2 protein family: opposing activities that mediate cell death

Apoptosis: controlled demolition at the cellular level

In vitro effects of nanoparticles on renal cells

Redox regulation of apoptotic cell death in the immune system.

Pulmonary effects of inhaled ultrafine particles.

P2888

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10.1186/1743-8977-7-10

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7

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Johan A. Martens

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20398356

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P859

Development of methodology for alternative testing strategies for the assessment of the toxicological profile of nanoparticles used in medical diagnostics

P921

apoptotic process

Béésh doo Ńdiniichxíihii

P932

2873464

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