Gene therapy with extracellular superoxide dismutase protects conscious rabbits against myocardial infarction.
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Gene therapy for ischemic heart diseaseExtracellular Superoxide Dismutase: Growth Promoter or Tumor Suppressor?Antioxidant-based therapies for angiotensin II-associated cardiovascular diseasesPotential implication of the chemical properties and bioactivity of nitrone spin traps for therapeuticsExtracellular superoxide dismutase ameliorates skeletal muscle abnormalities, cachexia, and exercise intolerance in mice with congestive heart failure.Cardiac-selective expression of extracellular superoxide dismutase after systemic injection of adeno-associated virus 9 protects the heart against post-myocardial infarction left ventricular remodeling.A single direct injection into the left ventricular wall of an adeno-associated virus 9 (AAV9) vector expressing extracellular superoxide dismutase from the cardiac troponin-T promoter protects mice against myocardial infarction.Delivering regenerative cues to the heart: cardiac drug delivery by microspheres and peptide nanofibers.The delivery of superoxide dismutase encapsulated in polyketal microparticles to rat myocardium and protection from myocardial ischemia-reperfusion injury.Selective inhibition of class I but not class IIb histone deacetylases exerts cardiac protection from ischemia reperfusion.Transcriptional responses in the adaptation to ischaemia-reperfusion injury: a study of the effect of ischaemic preconditioning in total knee arthroplasty patients.Extracellular superoxide dismutase (ecSOD) in vascular biology: an update on exogenous gene transfer and endogenous regulators of ecSOD.Toll-like receptors 2 and 4: initiators of non-septic inflammation in critical care medicine?Protective effects of M40403, a selective superoxide dismutase mimetic, in myocardial ischaemia and reperfusion injury in vivoPlasma extracellular superoxide dismutase concentration, allelic variations in the SOD3 gene and risk of myocardial infarction and all-cause mortality in people with type 1 and type 2 diabetesOxidative stress and heart disease: cardiac dysfunction, nutrition, and gene therapy.The COX-2/PGI2 receptor axis plays an obligatory role in mediating the cardioprotection conferred by the late phase of ischemic preconditioning.Molecular Signature of Nitroso-Redox Balance in Idiopathic Dilated CardiomyopathiesReperfusion injury and reactive oxygen species: The evolution of a concept.Oxidative stress in myocardial ischaemia reperfusion injury: a renewed focus on a long-standing area of heart research.Cardiomyocyte-restricted overexpression of extracellular superoxide dismutase increases nitric oxide bioavailability and reduces infarct size after ischemia/reperfusion.Molecular aspects of ischemic heart disease: ischemia/reperfusion-induced genetic changes and potential applications of gene and RNA interference therapy.Extracellular superoxide dismutase deficiency exacerbates pressure overload-induced left ventricular hypertrophy and dysfunctionCarbon monoxide induces a late preconditioning-mimetic cardioprotective and antiapoptotic milieu in the myocardium.Loss of Mrp1 Potentiates Doxorubicin-Induced Cytotoxicity in Neonatal Mouse Cardiomyocytes and Cardiac FibroblastsDelivery of Nox2-NADPH oxidase siRNA with polyketal nanoparticles for improving cardiac function following myocardial infarctionDesigning heart performance by gene transfer.Antioxidant enzyme gene transfer for ischemic diseases.Role of nitroso radicals as drug targets in circulatory shock.Extracellular Superoxide Dismutase Expression in Papillary Thyroid Cancer Mesenchymal Stem/Stromal Cells Modulates Cancer Cell Growth and Migration.Myocardial protective effect of extracellular superoxide dismutase gene modified bone marrow mesenchymal stromal cells on infarcted mice hearts.The second window of preconditioning (SWOP) where are we now?Genetics and genomics of ischemic tolerance: focus on cardiac and cerebral ischemic preconditioning.TPA induces the expression of EC-SOD in human monocytic THP-1 cells: involvement of PKC, MEK/ERK and NOX-derived ROS.Gene transfer as a strategy to achieve permanent cardioprotection I: rAAV-mediated gene therapy with inducible nitric oxide synthase limits infarct size 1 year later without adverse functional consequences.Liposome-mediated extracellular superoxide dismutase gene delivery protects against acute liver injury in mice.Histone Deacetylase 1 Depletion Activates Human Cardiac Mesenchymal Stromal Cell Proangiogenic Paracrine Signaling Through a Mechanism Requiring Enhanced Basic Fibroblast Growth Factor Synthesis and Secretion.Cardiac myocyte-specific transgenic ecSOD targets mitochondria to protect against Ca(2+) induced permeability transitionAdenovirus-mediated overexpression of extracellular superoxide dismutase improves endothelial dysfunction in a rat model of hypertension.Role of intracellular antioxidant enzymes after in vivo myocardial ischemia and reperfusion.
P2860
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P2860
Gene therapy with extracellular superoxide dismutase protects conscious rabbits against myocardial infarction.
description
2001 nî lūn-bûn
@nan
2001 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Gene therapy with extracellula ...... against myocardial infarction.
@ast
Gene therapy with extracellula ...... against myocardial infarction.
@en
type
label
Gene therapy with extracellula ...... against myocardial infarction.
@ast
Gene therapy with extracellula ...... against myocardial infarction.
@en
prefLabel
Gene therapy with extracellula ...... against myocardial infarction.
@ast
Gene therapy with extracellula ...... against myocardial infarction.
@en
P2093
P2860
P1433
P1476
Gene therapy with extracellula ...... against myocardial infarction.
@en
P2093
P2860
P304
P356
10.1161/01.CIR.103.14.1893
P407
P577
2001-04-01T00:00:00Z