Sexually dimorphic testosterone secretion in prenatal and neonatal mice is independent of kisspeptin-Kiss1r and GnRH signaling.
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Neuroendocrine control of the onset of pubertyThe decline in pulsatile GnRH release, as reflected by circulating LH concentrations, during the infant-juvenile transition in the agonadal male rhesus monkey (Macaca mulatta) is associated with a reduction in kisspeptin content of KNDy neurons of tDevelopment of gonadotropin-releasing hormone secretion and pituitary response.Gonadotropin and kisspeptin gene expression, but not GnRH, are impaired in cFOS deficient mice.Daily successive changes in reproductive gene expression and neuronal activation in the brains of pubertal female mice.Developmental GnRH signaling is not required for sexual differentiation of kisspeptin neurons but is needed for maximal Kiss1 gene expression in adult femalesAnalysis of multiple positive feedback paradigms demonstrates a complete absence of LH surges and GnRH activation in mice lacking kisspeptin signaling.Prenatal exposure to low levels of androgen accelerates female puberty onset and reproductive senescence in miceEndogenous kisspeptin tone is a critical excitatory component of spontaneous GnRH activity and the GnRH response to NPY and CART.Kisspeptin neurones do not directly signal to RFRP-3 neurones but RFRP-3 may directly modulate a subset of hypothalamic kisspeptin cells in mice.Impaired kisspeptin signaling decreases metabolism and promotes glucose intolerance and obesity.The development of kisspeptin circuits in the Mammalian brain.Microarray analysis of perinatal-estrogen-induced changes in gene expression related to brain sexual differentiation in mice.A critical point of male gonad development: neuroendocrine correlates of accelerated testicular growth in rats during early life.Molecular profiling of postnatal development of the hypothalamus in female and male rats.Lactation undernutrition leads to multigenerational molecular programming of hypothalamic gene networks controlling reproduction.Disrupted organization of RFamide pathways in the hypothalamus is associated with advanced puberty in female rats neonatally exposed to bisphenol A.Rbpj-κ mediated Notch signaling plays a critical role in development of hypothalamic Kisspeptin neurons.Kiss of the mutant mouse: how genetically altered mice advanced our understanding of kisspeptin's role in reproductive physiologyGonadotropin-releasing hormone receptor (Gnrhr) gene knock out: Normal growth and development of sensory, motor and spatial orientation behavior but altered metabolism in neonatal and prepubertal mice.Prenatal exposure to low doses of bisphenol A increases pituitary proliferation and gonadotroph number in female mice offspring at birth.Hypothalamic control of the male neonatal testosterone surge.Development and Aging of the Kisspeptin-GPR54 System in the Mammalian Brain: What are the Impacts on Female Reproductive Function?Icam5 Expression Exhibits Sex Differences in the Neonatal Pituitary and Is Regulated by Estradiol and Bisphenol AEmerging concepts on the epigenetic and transcriptional regulation of the Kiss1 gene.Organizational and activational effects of sex steroids on kisspeptin neuron development.Developmental profile and sexually dimorphic expression of kiss1 and kiss1r in the fetal mouse brain.Lack of functional GABAB receptors alters Kiss1 , Gnrh1 and Gad1 mRNA expression in the medial basal hypothalamus at postnatal day 4.Kisspeptin and puberty in mammals.Morphological and functional maturation of Leydig cells: from rodent models to primates.Control of puberty onset and fertility by gonadotropin-releasing hormone neurons.Metabolism and Energy Expenditure, But Not Feeding or Glucose Tolerance, Are Impaired in Young Kiss1r KO Female Mice.Effects of Exposure to the Endocrine-Disrupting Chemical Bisphenol A During Critical Windows of Murine Pituitary Development.Endogenous brain erythropoietin is a potent sex-specific respiratory stimulant in adult and newborn mice.The effects of gonadal steroid manipulation on the expression of Kiss1 mRNA in rat arcuate nucleus during postnatal development.Neonatal Kisspeptin is Steroid-Independently Required for Defeminisation and Peripubertal Kisspeptin-Induced Testosterone is Required for Masculinisation of the Brain: A Behavioural Study Using Kiss1 Knockout Rats.GnRH Neurons on LSD: A Year of Rejecting Hypotheses That May Have Made Karl Popper Proud.Reduced hypoxic ventilatory response in newborn mice knocked-out for the progesterone receptor.Haploinsufficiency of SIX3 Abolishes Male Reproductive Behavior Through Disrupted Olfactory Development, and Impairs Female Fertility Through Disrupted GnRH Neuron Migration.
P2860
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P2860
Sexually dimorphic testosterone secretion in prenatal and neonatal mice is independent of kisspeptin-Kiss1r and GnRH signaling.
description
2011 nî lūn-bûn
@nan
2011 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Sexually dimorphic testosteron ...... tin-Kiss1r and GnRH signaling.
@ast
Sexually dimorphic testosteron ...... tin-Kiss1r and GnRH signaling.
@en
type
label
Sexually dimorphic testosteron ...... tin-Kiss1r and GnRH signaling.
@ast
Sexually dimorphic testosteron ...... tin-Kiss1r and GnRH signaling.
@en
prefLabel
Sexually dimorphic testosteron ...... tin-Kiss1r and GnRH signaling.
@ast
Sexually dimorphic testosteron ...... tin-Kiss1r and GnRH signaling.
@en
P2860
P356
P1433
P1476
Sexually dimorphic testosteron ...... tin-Kiss1r and GnRH signaling.
@en
P2093
Alexander S Kauffman
Matthew C Poling
P2860
P304
P356
10.1210/EN.2011-1838
P407
P577
2011-12-27T00:00:00Z