Hindbrain ghrelin receptor signaling is sufficient to maintain fasting glucose. - Wikidata - wikimedia - TriplyDB

Hindbrain ghrelin receptor signaling is sufficient to maintain fasting glucose.

Hindbrain ghrelin receptor signaling is sufficient to maintain fasting glucose.

http://www.wikidata.org/entity/Q30425047

about

Ghrelin.The central nervous system sites mediating the orexigenic actions of ghrelin.Ghrelin: much more than a hunger hormone.Divergent neuronal circuitries underlying acute orexigenic effects of peripheral or central ghrelin: critical role of brain accessibility.Neuroanatomical characterization of a growth hormone secretagogue receptor-green fluorescent protein reporter mouse.Gender differences in ghrelin association with cardiometabolic risk factors in arab population.Gene-environment interactions controlling energy and glucose homeostasis and the developmental origins of obesity Genetic determination of the cellular basis of the ghrelin-dependent bone remodeling.The β-cell GHSR and downstream cAMP/TRPM2 signaling account for insulinostatic and glycemic effects of ghrelin Central ghrelin increases food foraging/hoarding that is blocked by GHSR antagonism and attenuates hypothalamic paraventricular nucleus neuronal activation.Ghrelin and eating behavior: evidence and insights from genetically-modified mouse models.Neuroendocrine Regulation of Metabolism Analysis of brain nuclei accessible to ghrelin present in the cerebrospinal fluid.Arcuate AgRP neurons mediate orexigenic and glucoregulatory actions of ghrelin.The role of ghrelin-responsive mediobasal hypothalamic neurons in mediating feeding responses to fasting.Ghrelin as a Survival Hormone.Fourth ventricle injection of ghrelin decreases angiotensin II-induced fluid intake and neuronal activation in the paraventricular nucleus of the hypothalamus.The Ghrelin/GOAT System Regulates Obesity-Induced Inflammation in Male Mice.Profiling of G protein-coupled receptors in vagal afferents reveals novel gut-to-brain sensing mechanisms.

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P2860

Hindbrain ghrelin receptor signaling is sufficient to maintain fasting glucose.

http://www.wikidata.org/entity/Q30425047

description

2012 nî lūn-bûn

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2012 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի օգոստոսին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Hindbrain ghrelin receptor signaling is sufficient to maintain fasting glucose.

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Hindbrain ghrelin receptor signaling is sufficient to maintain fasting glucose.

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type

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Hindbrain ghrelin receptor signaling is sufficient to maintain fasting glucose.

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Hindbrain ghrelin receptor signaling is sufficient to maintain fasting glucose.

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prefLabel

Hindbrain ghrelin receptor signaling is sufficient to maintain fasting glucose.

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Hindbrain ghrelin receptor signaling is sufficient to maintain fasting glucose.

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JOURNAL.PONE.0044089

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Hindbrain ghrelin receptor signaling is sufficient to maintain fasting glucose.

@en

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Charlotte E Lee

http://www.w3.org/2001/XMLSchema#string

Danielle Lauzon

http://www.w3.org/2001/XMLSchema#string

Ichiro Sakata

http://www.w3.org/2001/XMLSchema#string

Jeffrey M Zigman

http://www.w3.org/2001/XMLSchema#string

Jen-Chieh Chuang

http://www.w3.org/2001/XMLSchema#string

Joel K Elmquist

http://www.w3.org/2001/XMLSchema#string

Laurent Gautron

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Michael M Scott

http://www.w3.org/2001/XMLSchema#string

P2860

Ghrelin is a growth-hormone-releasing acylated peptide from stomach

Mice lacking ghrelin receptors resist the development of diet-induced obesity

Ghrelin modulates the activity and synaptic input organization of midbrain dopamine neurons while promoting appetite

Identification of the acyltransferase that octanoylates ghrelin, an appetite-stimulating peptide hormone

A role for ghrelin in the central regulation of feeding

The GOAT-ghrelin system is not essential for hypoglycemia prevention during prolonged calorie restriction.

Ghrelin acts in the central nervous system to stimulate gastric acid secretion.

Ghrelin O-acyltransferase (GOAT) is essential for growth hormone-mediated survival of calorie-restricted mice.

A preprandial rise in plasma ghrelin levels suggests a role in meal initiation in humans.

Hyperphagic effects of brainstem ghrelin administration.

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10.1371/JOURNAL.PONE.0044089

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