Genetic inactivation of IL-1 signaling enhances atherosclerotic plaque instability and reduces outward vessel remodeling in advanced atherosclerosis in mice - Wikidata - wikimedia - TriplyDB

Genetic inactivation of IL-1 signaling enhances atherosclerotic plaque instability and reduces outward vessel remodeling in advanced atherosclerosis in mice

Genetic inactivation of IL-1 signaling enhances atherosclerotic plaque instability and reduces outward vessel remodeling in advanced atherosclerosis in mice

http://www.wikidata.org/entity/Q30429015

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Oxidative DNA Damage Mediated by Intranuclear MMP Activity Is Associated with Neuronal Apoptosis in Ischemic Stroke Anti-inflammatory therapy in chronic disease: challenges and opportunities Targeting interleukin-1 in heart disease Cholesterol crystals and inflammation CXCL5 limits macrophage foam cell formation in atherosclerosis Smooth muscle cell-specific deletion of Col15a1 unexpectedly leads to impaired development of advanced atherosclerotic lesions.Activation of the pluripotency factor OCT4 in smooth muscle cells is atheroprotective.KLF4-dependent phenotypic modulation of smooth muscle cells has a key role in atherosclerotic plaque pathogenesis.Recent insights into the cellular biology of atherosclerosis Detection of high-risk atherosclerotic plaques with ultrasound molecular imaging of glycoprotein IIb/IIIa receptor on activated platelets.Adenosine 2A receptor modulates inflammation and phenotype in experimental abdominal aortic aneurysms Cigarette smoke modulates vascular smooth muscle phenotype: implications for carotid and cerebrovascular disease Smooth muscle cell phenotypic switching in atherosclerosis The addition of vildagliptin to metformin prevents the elevation of interleukin 1ß in patients with type 2 diabetes and coronary artery disease: a prospective, randomized, open-label study.Interleukin 1 receptor 1 and interleukin 1β regulate megakaryocyte maturation, platelet activation, and transcript profile during inflammation in mice and humans.Inflammation and its resolution as determinants of acute coronary syndromes.Moderate hypoxia potentiates interleukin-1β production in activated human macrophages.Hematopoietic IKBKE limits the chronicity of inflammasome priming and metaflammation.Bone marrow deficiency of MCPIP1 results in severe multi-organ inflammation but diminishes atherogenesis in hyperlipidemic mice.Network analysis of inflammatory genes and their transcriptional regulators in coronary artery disease.Cytokines in atherosclerosis: Key players in all stages of disease and promising therapeutic targets.IL-1 and atherosclerosis: a murine twist to an evolving human story.Serum CD121a (Interleukin 1 Receptor, Type I): A Potential Novel Inflammatory Marker for Coronary Heart Disease.Inflammatory cell phenotypes in AAAs: their role and potential as targets for therapy.Generation and Characterization of Mice Expressing a Conditional Allele of the Interleukin-1 Receptor Type 1.Redundancy of IL-1 Isoform Signaling and Its Implications for Arterial Remodeling Cytokines: roles in atherosclerosis disease progression and potential therapeutic targets Cardiometabolic effects of genetic upregulation of the interleukin 1 receptor antagonist: a Mendelian randomisation analysis IL-1 Signaling Is Critically Required in Stromal Cells in Kawasaki Disease Vasculitis Mouse Model: Role of Both IL-1α and IL-1β Interleukin-1 beta, interferon-gamma, and tumor necrosis factor-alpha gene expression in peripheral blood mononuclear cells of patients with coronary artery disease.Sputum mediator profiling and relationship to airway wall geometry imaging in severe asthma.Beneficial effects of quinoline-3-carboxamide (ABR-215757) on atherosclerotic plaque morphology in S100A12 transgenic ApoE null mice A Systems Biology Approach to Characterize Biomarkers for Blood Stasis Syndrome of Unstable Angina Patients by Integrating MicroRNA and Messenger RNA Expression Profiling CD47-blocking antibodies restore phagocytosis and prevent atherosclerosis.Lipopolysaccharide stimulates platelets through an IL-1β autocrine loop.Arterial Effects of Canakinumab in Patients With Atherosclerosis and Type 2 Diabetes or Glucose Intolerance.Interleukins and atherosclerosis: a dysfunctional family grows.Rationale and study design for a phase I/IIa trial of anakinra in children with Kawasaki disease and early coronary artery abnormalities (the ANAKID trial).Cyclin-dependent kinase inhibitor 2B regulates efferocytosis and atherosclerosis Th17 cells and IL-17 are involved in the disruption of vulnerable plaques triggered by short-term combination stimulation in apolipoprotein E-knockout mice.

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Genetic inactivation of IL-1 signaling enhances atherosclerotic plaque instability and reduces outward vessel remodeling in advanced atherosclerosis in mice

http://www.wikidata.org/entity/Q30429015

description

2011 nî lūn-bûn

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2011 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Genetic inactivation of IL-1 s ...... vanced atherosclerosis in mice

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Genetic inactivation of IL-1 s ...... vanced atherosclerosis in mice

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type

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Genetic inactivation of IL-1 s ...... vanced atherosclerosis in mice

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Genetic inactivation of IL-1 s ...... vanced atherosclerosis in mice

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prefLabel

Genetic inactivation of IL-1 s ...... vanced atherosclerosis in mice

@ast

Genetic inactivation of IL-1 s ...... vanced atherosclerosis in mice

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Journal of Clinical Investigation

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Genetic inactivation of IL-1 s ...... vanced atherosclerosis in mice

@en

P2093

Christopher L Jackson

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Christopher W Moehle

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Gary K Owens

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Jae K Lee

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Matthew R Alexander

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Zhengyu Yang

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P2860

Atherosclerosis

Cellular and molecular mechanisms of fibrosis

The chemokine system in diverse forms of macrophage activation and polarization

Genomic profiling of tumor necrosis factor alpha (TNF-alpha) receptor and interleukin-1 receptor knockout mice reveals a link between TNF-alpha signaling and increased severity of 1918 pandemic influenza virus infection.

MyD88-dependent, superoxide-initiated inflammation is necessary for flow-mediated inward remodeling of conduit arteries.

Pravastatin treatment increases collagen content and decreases lipid content, inflammation, metalloproteinases, and cell death in human carotid plaques: implications for plaque stabilization.

Fibrous cap formation or destruction--the critical importance of vascular smooth muscle cell proliferation, migration and matrix formation.

Inflammation in atherosclerosis: from pathophysiology to practice

Arterial remodeling in atherosclerosis, restenosis and after alteration of blood flow: potential mechanisms and clinical implications.

Divergent effects of matrix metalloproteinases 3, 7, 9, and 12 on atherosclerotic plaque stability in mouse brachiocephalic arteries.

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10.1172/JCI43713

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1

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122

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Jason Lee Johnson

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2011-12-27T00:00:00Z

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51925851

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22201681

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atherosclerosis

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3248279

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