Estrogen utilization of IGF-1-R and EGF-R to signal in breast cancer cells. - Wikidata - wikimedia - TriplyDB

Estrogen utilization of IGF-1-R and EGF-R to signal in breast cancer cells.

Estrogen utilization of IGF-1-R and EGF-R to signal in breast cancer cells.

http://www.wikidata.org/entity/Q30433113

about

Insulin-like growth factor and epidermal growth factor signaling in breast cancer cell growth: focus on endocrine resistant disease Targeting insulin and insulin-like growth factor signaling in breast cancer Interaction of JAK with steroid receptor function Met receptor acts uniquely for survival and morphogenesis of EGFR-dependent normal mammary epithelial and cancer cells Adenosine monophosphate activated protein kinase (AMPK), a mediator of estradiol-induced apoptosis in long-term estrogen deprived breast cancer cells.Cytosolic phospholipase A2 activation correlates with HER2 overexpression and mediates estrogen-dependent breast cancer cell growth.A mechanistic study of the effect of doxorubicin/adriamycin on the estrogen response in a breast cancer model ESR1 mutations affect anti-proliferative responses to tamoxifen through enhanced cross-talk with IGF signaling Defining the pathway to insulin-like growth factor system targeting in cancer.Can we unlock the potential of IGF-1R inhibition in cancer therapy?Exploiting MEK inhibitor-mediated activation of ERα for therapeutic intervention in ER-positive ovarian carcinoma.AR collaborates with ERα in aromatase inhibitor-resistant breast cancer.Elevated insulin-like growth factor 1 receptor signaling induces antiestrogen resistance through the MAPK/ERK and PI3K/Akt signaling routes.Interactions between IGF-I, estrogen receptor-α (ERα), and ERβ in regulating growth/apoptosis of MCF-7 human breast cancer cells.Quantifying insulin receptor isoform expression in FFPE breast tumors Stromal cells promote anti-estrogen resistance of breast cancer cells through an insulin-like growth factor binding protein 5 (IGFBP5)/B-cell leukemia/lymphoma 3 (Bcl-3) axis Targeted genome-wide methylation and gene expression analyses reveal signaling pathways involved in ovarian dysfunction after developmental EDC exposure in rats.Extranuclear ERα is associated with regression of T47D PKCα-overexpressing, tamoxifen-resistant breast cancer Epithelial to mesenchymal transition promotes breast cancer progression via a fibronectin-dependent STAT3 signaling pathway.Insulin-like growth factor: current concepts and new developments in cancer therapy.Extranuclear signaling by estrogen: role in breast cancer progression and metastasis.Mediator of ERBB2-driven cell motility (MEMO) promotes extranuclear estrogen receptor signaling involving the growth factor receptors IGF1R and ERBB2 Endocrine resistance in breast cancer: molecular pathways and rational development of targeted therapies.Modulation of sensitivity and resistance to multikinase inhibitors by microenvironmental platelet factors in HCC.The exposure of breast cancer cells to fulvestrant and tamoxifen modulates cell migration differently.Flaxseed cotyledon fraction reduces tumour growth and sensitises tamoxifen treatment of human breast cancer xenograft (MCF-7) in athymic mice.Metformin Attenuates Testosterone-Induced Prostatic Hyperplasia in Rats: A Pharmacological Perspective.Evaluation of the coordinated actions of estrogen receptors with epidermal growth factor receptor and insulin-like growth factor receptor in the expression of cell surface heparan sulfate proteoglycans and cell motility in breast cancer cells.Extranuclear signaling by sex steroid receptors and clinical implications in breast cancer.Divergent effects of insulin-like growth factor-1 receptor expression on prognosis of estrogen receptor positive versus triple negative invasive ductal breast carcinoma.Sex steroids and breast cancer metastasis.Endocrine Resistance in Breast Cancer

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P2860

Estrogen utilization of IGF-1-R and EGF-R to signal in breast cancer cells.

http://www.wikidata.org/entity/Q30433113

description

2009 nî lūn-bûn

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2009 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Estrogen utilization of IGF-1-R and EGF-R to signal in breast cancer cells.

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Estrogen utilization of IGF-1-R and EGF-R to signal in breast cancer cells.

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type

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Estrogen utilization of IGF-1-R and EGF-R to signal in breast cancer cells.

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Estrogen utilization of IGF-1-R and EGF-R to signal in breast cancer cells.

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prefLabel

Estrogen utilization of IGF-1-R and EGF-R to signal in breast cancer cells.

@ast

Estrogen utilization of IGF-1-R and EGF-R to signal in breast cancer cells.

@en

P1433

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J.JSBMB.2009.09.018

P1433

The Journal of Steroid Biochemistry and Molecular Biology

P1476

Estrogen utilization of IGF-1-R and EGF-R to signal in breast cancer cells.

@en

P2093

Ji-Ping Wang

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Ping Fan

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Richard J Santen

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Robert X-D Song

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Wei Yue

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Yongde Bao

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Yuchai Chen

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Zhenguo Zhang

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P2860

Estrogen receptor alpha rapidly activates the IGF-1 receptor pathway

Linkage of rapid estrogen action to MAPK activation by ERalpha-Shc association and Shc pathway activation

Estrogen receptor alpha in human breast cancer: occurrence and significance

Effect of long-term estrogen deprivation on apoptotic responses of breast cancer cells to 17beta-estradiol.

The role of Shc and insulin-like growth factor 1 receptor in mediating the translocation of estrogen receptor alpha to the plasma membrane.

Farnesylthiosalicylic acid blocks mammalian target of rapamycin signaling in breast cancer cells.

Estrogen signaling via a linear pathway involving insulin-like growth factor I receptor, matrix metalloproteinases, and epidermal growth factor receptor to activate mitogen-activated protein kinase in MCF-7 breast cancer cells.

Long-term treatment with tamoxifen facilitates translocation of estrogen receptor alpha out of the nucleus and enhances its interaction with EGFR in MCF-7 breast cancer cells.

Regulation of tyrosine kinase cascades by G-protein-coupled receptors.

Epidermal growth factor receptors: critical mediators of multiple receptor pathways.

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219-230

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10.1016/J.JSBMB.2009.09.018

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4-5

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118

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2009-10-06T00:00:00Z

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26881053

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19815064

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2826506

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