Neointimal formation in two apolipoprotein E-deficient mouse strains with different atherosclerosis susceptibility. - Wikidata - wikimedia - TriplyDB

Neointimal formation in two apolipoprotein E-deficient mouse strains with different atherosclerosis susceptibility.

Neointimal formation in two apolipoprotein E-deficient mouse strains with different atherosclerosis susceptibility.

http://www.wikidata.org/entity/Q30434230

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Antiretrovirals induce endothelial dysfunction via an oxidant-dependent pathway and promote neointimal hyperplasia Genes within the MHC region have a dramatic influence on radiation-enhanced atherosclerosis in mice Quantitative trait locus analysis of neointimal formation in an intercross between C57BL/6 and C3H/HeJ apolipoprotein E-deficient mice.Intimal hyperplasia in murine models Influence of genetic background on genetically engineered mouse phenotypes.Essential role of ER-alpha-dependent NO production in resveratrol-mediated inhibition of restenosis.Cell adhesion molecules and eNOS expression in aorta of normocholesterolemic mice with different predispositions to atherosclerosis.Accelerated atherogenesis in completely ligated common carotid artery of apolipoprotein E-deficient mice.

P2860

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P2860

Neointimal formation in two apolipoprotein E-deficient mouse strains with different atherosclerosis susceptibility.

http://www.wikidata.org/entity/Q30434230

description

2004 nî lūn-bûn

@nan

2004 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2004 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2004年の論文

@ja

2004年論文

@yue

2004年論文

@zh-hant

2004年論文

@zh-hk

2004年論文

@zh-mo

2004年論文

@zh-tw

2004年论文

@wuu

name

Neointimal formation in two ap ...... therosclerosis susceptibility.

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Neointimal formation in two ap ...... therosclerosis susceptibility.

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type

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Neointimal formation in two ap ...... therosclerosis susceptibility.

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Neointimal formation in two ap ...... therosclerosis susceptibility.

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Neointimal formation in two ap ...... therosclerosis susceptibility.

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Neointimal formation in two ap ...... therosclerosis susceptibility.

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JLR.M400254-JLR200

P1433

Journal of Lipid Research

P1476

Neointimal formation in two ap ...... therosclerosis susceptibility.

@en

P2093

Gregory A Helm

http://www.w3.org/2001/XMLSchema#string

Hong Pei

http://www.w3.org/2001/XMLSchema#string

Ian J Sarembock

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Jessica C James

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John F Angle

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Joshua J Fischer

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P2860

Atherosclerosis — An Inflammatory Disease

Atherogenesis in perspective: hypercholesterolemia and inflammation as partners in crime

Atherosclerosis: basic mechanisms. Oxidation, inflammation, and genetics

Absence of p-selectin, but not intercellular adhesion molecule-1, attenuates neointimal growth after arterial injury in apolipoprotein e-deficient mice.

Critical role of platelet P-selectin in the response to arterial injury in apolipoprotein-E-deficient mice.

Lipid retention in the arterial wall of two mouse strains with different atherosclerosis susceptibility.

Minimally modified low density lipoprotein induces monocyte chemotactic protein 1 in human endothelial cells and smooth muscle cells.

Adenovirus-mediated gene transfer of human platelet-activating factor-acetylhydrolase prevents injury-induced neointima formation and reduces spontaneous atherosclerosis in apolipoprotein E-deficient mice.

Mechanisms of angioplasty and stent restenosis: implications for design of rational therapy.

Modified low density lipoprotein and its constituents augment cytokine-activated vascular cell adhesion molecule-1 gene expression in human vascular endothelial cells.

P304

2008-2014

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scholarly article

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10.1194/JLR.M400254-JLR200

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11

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45

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Alan H Matsumoto

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2004-08-16T00:00:00Z

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15314103

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P921

atherosclerosis